| scholarly article | Q13442814 |
| P50 | author | Simona Capsoni | Q42873546 |
| Luciano Domenici | Q47224571 | ||
| Antonino Cattaneo | Q56589787 | ||
| Ottavio Arancio | Q57733929 | ||
| Ann Marie Schmidt | Q89261684 | ||
| P2093 | author name string | Fang Fang | |
| Massimo Righi | |||
| John Xi Chen | |||
| David M Stern | |||
| Shi Du Yan | |||
| Nicola Origlia | |||
| P2860 | cites work | Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways | Q22010208 |
| RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides | Q22010236 | ||
| RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease | Q24314906 | ||
| Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo | Q28131779 | ||
| SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase | Q28364720 | ||
| Regulation of NMDA receptor trafficking by amyloid-beta | Q28506085 | ||
| Role of receptor for advanced glycation end-product (RAGE) and the JAK/STAT-signaling pathway in AGE-induced collagen production in NRK-49F cells | Q28570142 | ||
| N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression | Q28645594 | ||
| A specific amyloid-beta protein assembly in the brain impairs memory | Q28854597 | ||
| Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins | Q29547593 | ||
| Alzheimer's disease is a synaptic failure | Q29547670 | ||
| Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction | Q29547778 | ||
| AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss | Q29614850 | ||
| Natural oligomers of the amyloid-beta protein specifically disrupt cognitive function | Q29614851 | ||
| High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation | Q29616564 | ||
| Learning induces long-term potentiation in the hippocampus | Q29619482 | ||
| The LTP Program: a data acquisition program for on-line analysis of long-term potentiation and other synaptic events | Q30656626 | ||
| The p38 signal transduction pathway: activation and function | Q33838955 | ||
| Postsynaptic Hebbian and non-Hebbian long-term potentiation of synaptic efficacy in the entorhinal cortex in slices and in the isolated adult guinea pig brain | Q33913732 | ||
| The Alzheimer's A beta -peptide is deposited at sites of complement activation in pathologic deposits associated with aging and age-related macular degeneration | Q34154466 | ||
| Amyloid beta -peptide inhibition of the PKA/CREB pathway and long-term potentiation: reversibility by drugs that enhance cAMP signaling | Q34192226 | ||
| The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses | Q34387881 | ||
| AMPA receptor downscaling at the onset of Alzheimer's disease pathology in double knockin mice. | Q34480004 | ||
| Central role of RAGE-dependent neointimal expansion in arterial restenosis | Q34890522 | ||
| Plaque-independent disruption of neural circuits in Alzheimer's disease mouse models | Q35070000 | ||
| Functional neuroanatomy of the medial temporal lobe memory system | Q35740386 | ||
| Preventing activation of receptor for advanced glycation endproducts in Alzheimer's disease | Q36173060 | ||
| Evaluation of rage isoforms, ligands, and signaling in the brain. | Q36281326 | ||
| Functional organization of the extrinsic and intrinsic circuitry of the parahippocampal region | Q38717138 | ||
| RAGE potentiates Abeta-induced perturbation of neuronal function in transgenic mice | Q40251711 | ||
| P38 activation mediates amyloid-beta cytotoxicity | Q40368411 | ||
| Certain inhibitors of synthetic amyloid beta-peptide (Abeta) fibrillogenesis block oligomerization of natural Abeta and thereby rescue long-term potentiation. | Q40448359 | ||
| RAGE-Abeta interactions in the pathophysiology of Alzheimer's disease | Q40659139 | ||
| Entorhinal-hippocampal interaction in mnestic disorders. | Q40803845 | ||
| The pyridinyl imidazole inhibitor SB203580 blocks phosphoinositide-dependent protein kinase activity, protein kinase B phosphorylation, and retinoblastoma hyperphosphorylation in interleukin-2-stimulated T cells independently of p38 mitogen-activate | Q40897138 | ||
| Alterations in synaptic transmission and long-term potentiation in hippocampal slices from young and aged PDAPP mice | Q41695737 | ||
| PDGF B-chain in neurons of the central nervous system, posterior pituitary, and in a transgenic model | Q41699855 | ||
| RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy | Q42263637 | ||
| Non-fibrillar beta-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptors. | Q42493702 | ||
| Acute cholinergic rescue of synaptic plasticity in the neurodegenerating cortex of anti-nerve-growth-factor mice. | Q42519359 | ||
| Alzheimer amyloid beta-peptide inhibits the late phase of long-term potentiation through calcineurin-dependent mechanisms in the hippocampal dentate gyrus | Q43980897 | ||
| Activation of p38 plays a pivotal role in the inhibitory effect of lipopolysaccharide and interleukin-1 beta on long term potentiation in rat dentate gyrus | Q44333324 | ||
| NMDA receptor-dependent long-term synaptic depression in the entorhinal cortex in vitro | Q44336835 | ||
| NMDA receptor regulation by amyloid-beta does not account for its inhibition of LTP in rat hippocampus | Q44380914 | ||
| A role for c-Jun N-terminal kinase in the inhibition of long-term potentiation by interleukin-1beta and long-term depression in the rat dentate gyrus in vitro | Q44406446 | ||
| Oxidative neurotoxicity in rat cerebral cortex neurons: Synergistic effects of H2O2 and NO on apoptosis involving activation of p38 mitogen‐activated protein kinase and caspase‐3 | Q44409701 | ||
| RAGE mediates amyloid-beta peptide transport across the blood-brain barrier and accumulation in brain | Q44479225 | ||
| Early Abeta accumulation and progressive synaptic loss, gliosis, and tangle formation in AD brain | Q44810166 | ||
| beta-Amyloid peptides impair PKC-dependent functions of metabotropic glutamate receptors in prefrontal cortical neurons | Q45230577 | ||
| ABAD enhances Abeta-induced cell stress via mitochondrial dysfunction | Q45234437 | ||
| Entorhinal cortex of the rat: organization of intrinsic connections | Q46244767 | ||
| Amyloid-beta peptide inhibits activation of the nitric oxide/cGMP/cAMP-responsive element-binding protein pathway during hippocampal synaptic plasticity. | Q46610687 | ||
| Amyloid beta prevents activation of calcium/calmodulin-dependent protein kinase II and AMPA receptor phosphorylation during hippocampal long-term potentiation | Q46641415 | ||
| Toxicity of various amyloid beta peptide species in cultured human blood-brain barrier endothelial cells: increased toxicity of dutch-type mutant | Q46881655 | ||
| Progressive age-related development of Alzheimer-like pathology in APP/PS1 mice | Q47403984 | ||
| ABAD directly links Abeta to mitochondrial toxicity in Alzheimer's disease | Q47754186 | ||
| Intracellular accumulation of detergent-soluble amyloidogenic A beta fragment of Alzheimer's disease precursor protein in the hippocampus of aged transgenic mice. | Q48198168 | ||
| p38 kinase is activated in the Alzheimer's disease brain | Q48227440 | ||
| Amyloid beta-peptide Abeta(1-42) but not Abeta(1-40) attenuates synaptic AMPA receptor function | Q48234804 | ||
| Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice | Q48242315 | ||
| Localization of active forms of C-jun kinase (JNK) and p38 kinase in Alzheimer's disease brains at different stages of neurofibrillary degeneration. | Q48492166 | ||
| Variation in effective stimulus patterns for induction of long-term potentiation across different layers of rat entorhinal cortex. | Q48668581 | ||
| Demonstration of Amyloid Deposits and Neurofibrillary Changes in Whole Brain Sections | Q48764154 | ||
| Intraneuronal Abeta accumulation precedes plaque formation in beta-amyloid precursor protein and presenilin-1 double-transgenic mice | Q48846848 | ||
| beta-Amyloid-induced neuronal apoptosis requires c-Jun N-terminal kinase activation | Q48932916 | ||
| Activation of the Receptor for Advanced Glycation End Products Triggers a p21 -dependent Mitogen-activated Protein Kinase Pathway Regulated by Oxidant Stress | Q50180621 | ||
| Spatial learning deficit in mice expressing human 751-amino acid beta-amyloid precursor protein. | Q52198981 | ||
| Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide. | Q53200549 | ||
| Soluble oligomers of beta amyloid (1-42) inhibit long-term potentiation but not long-term depression in rat dentate gyrus. | Q53245545 | ||
| Involvement of microglial receptor for advanced glycation endproducts (RAGE) in Alzheimer's disease: identification of a cellular activation mechanism. | Q53348339 | ||
| A redox-triggered ras-effector interaction. Recruitment of phosphatidylinositol 3'-kinase to Ras by redox stress. | Q54582355 | ||
| Surface plasmon resonance and nuclear magnetic resonance studies of ABAD-Abeta interaction | Q57187128 | ||
| Patterns of synaptic activity in forward and feedback pathways within rat visual cortex | Q71365389 | ||
| Secondary structure of amyloid beta peptide correlates with neurotoxic activity in vitro | Q72334677 | ||
| Selective enhancement of non-NMDA receptor-mediated responses following induction of long-term potentiation in entorhinal cortex | Q73223397 | ||
| In vivo synaptic transmission in young and aged amyloid precursor protein transgenic mice | Q74269225 | ||
| Block of long-term potentiation by naturally secreted and synthetic amyloid beta-peptide in hippocampal slices is mediated via activation of the kinases c-Jun N-terminal kinase, cyclin-dependent kinase 5, and p38 mitogen-activated protein kinase as | Q79851539 | ||
| P433 | issue | 13 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Advanced glycosylation end product-specific receptor | Q21496784 |
| response to amyloid-beta | Q22273351 | ||
| advanced glycosylation end-product specific receptor | Q22683568 | ||
| P304 | page(s) | 3521-3530 | |
| P577 | publication date | 2008-03-01 | |
| P1433 | published in | Journal of Neuroscience | Q1709864 |
| P1476 | title | Receptor for advanced glycation end product-dependent activation of p38 mitogen-activated protein kinase contributes to amyloid-beta-mediated cortical synaptic dysfunction | |
| P478 | volume | 28 |
| Q41829626 | Abeta-dependent Inhibition of LTP in different intracortical circuits of the visual cortex: the role of RAGE |
| Q91710120 | Acute Pannexin 1 Blockade Mitigates Early Synaptic Plasticity Defects in a Mouse Model of Alzheimer's Disease |
| Q35014590 | Adenosine A2A receptor blockade prevents synaptotoxicity and memory dysfunction caused by beta-amyloid peptides via p38 mitogen-activated protein kinase pathway |
| Q41682122 | Advanced glycation end products (AGEs) activate mast cells |
| Q43648217 | Advanced glycation end products reduce the calcium transient in cardiomyocytes by increasing production of reactive oxygen species and nitric oxide |
| Q30426453 | Advanced glycation end-products and their receptor-mediated roles: inflammation and oxidative stress |
| Q37598118 | Alzheimer's disease amyloid beta-protein and synaptic function |
| Q48726810 | Alzheimer's disease: A prion protein connection |
| Q49256155 | Amyloid-Beta and Phosphorylated Tau Accumulations Cause Abnormalities at Synapses of Alzheimer's disease Neurons |
| Q30493710 | Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38alpha MAPK pathway. |
| Q58202794 | An isoform-selective p38α mitogen-activated protein kinase inhibitor rescues early entorhinal cortex dysfunctions in a mouse model of Alzheimer's disease |
| Q84875066 | Association between the RAGE G82S polymorphism and Alzheimer's disease |
| Q33956505 | Association of the RAGE G82S polymorphism with Alzheimer's disease |
| Q42198316 | Atorvastatin prevents amyloid-β peptide oligomer-induced synaptotoxicity and memory dysfunction in rats through a p38 MAPK-dependent pathway |
| Q37673274 | Basic mechanisms of neurodegeneration: a critical update |
| Q42255091 | Brain amyloid β protein and memory disruption in Alzheimer’s disease |
| Q36943048 | Brain-derived neurotrophic factor-dependent synaptic plasticity is suppressed by interleukin-1β via p38 mitogen-activated protein kinase |
| Q30279162 | Cdk5 is a New Rapid Synaptic Homeostasis Regulator Capable of Initiating the Early Alzheimer-Like Pathology |
| Q37399983 | Centaurin-α1-Ras-Elk-1 signaling at mitochondria mediates β-amyloid-induced synaptic dysfunction |
| Q49159731 | Comparison of the Effect of Exercise on Late-Phase LTP of the Dentate Gyrus and CA1 of Alzheimer's Disease Model |
| Q37602130 | Crosstalk between calcium, amyloid beta and the receptor for advanced glycation endproducts in Alzheimer's disease |
| Q37463481 | Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease |
| Q37404781 | Cyclophilin D deficiency rescues Aβ-impaired PKA/CREB signaling and alleviates synaptic degeneration |
| Q30484598 | Deletion of RAGE causes hyperactivity and increased sensitivity to auditory stimuli in mice |
| Q42872730 | Derailed intraneuronal signalling drives pathogenesis in sporadic and familial Alzheimer's disease. |
| Q64257598 | Development of a Multicomponent Intervention to Prevent Alzheimer's Disease |
| Q34166256 | Drug development for Alzheimer's disease: where are we now and where are we headed? |
| Q47938284 | ERM protein moesin is phosphorylated by advanced glycation end products and modulates endothelial permeability |
| Q58549216 | Early Life Stress and Epigenetics in Late-onset Alzheimer's Dementia: A Systematic Review |
| Q38874855 | Effect of Paullinia cupana Mart. Commercial Extract During the Aging of Middle Age Wistar Rats: Differential Effects on the Hippocampus and Striatum |
| Q36580926 | Effects of Heart Bypass Surgery on Plasma Aβ40 and Aβ42 Levels in Infants and Young Children |
| Q89944288 | Endogenous 3-Iodothyronamine (T1AM) and Synthetic Thyronamine-like Analog SG-2 Act as Novel Pleiotropic Neuroprotective Agents Through the Modulation of SIRT6 |
| Q37638090 | Entorhinal Cortex dysfunction can be rescued by inhibition of microglial RAGE in an Alzheimer's disease mouse model |
| Q48833407 | Erythropoietin improves memory function with reducing endothelial dysfunction and amyloid-beta burden in Alzheimer's disease models |
| Q36179583 | Genetic deficiency of neuronal RAGE protects against AGE-induced synaptic injury |
| Q36973739 | Glycation exacerbates the neuronal toxicity of β-amyloid. |
| Q45893258 | Impaired slow axonal transport in diabetic peripheral nerve is independent of RAGE. |
| Q37541359 | Increased expression of the receptor for advanced glycation end products in neurons and astrocytes in a triple transgenic mouse model of Alzheimer's disease |
| Q38120744 | Innate immunity and neuroinflammation |
| Q48535760 | Insulin induces phosphorylation of the AMPA receptor subunit GluR1, reversed by ZIP, and over-expression of Protein Kinase M zeta, reversed by amyloid beta |
| Q27001177 | Interaction between therapeutic interventions for Alzheimer's disease and physiological Aβ clearance mechanisms |
| Q48658737 | Intraneuronally injected amyloid β inhibits long-term potentiation in rat hippocampal slices |
| Q38260264 | Investigation of phosphoproteome in RAGE signaling |
| Q37630543 | MAPK, beta-amyloid and synaptic dysfunction: the role of RAGE. |
| Q38195641 | Microglia receptors and their implications in the response to amyloid β for Alzheimer's disease pathogenesis |
| Q35124699 | Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ) |
| Q30090185 | Microglial receptor for advanced glycation end product-dependent signal pathway drives beta-amyloid-induced synaptic depression and long-term depression impairment in entorhinal cortex |
| Q28730589 | Mitochondria-specific accumulation of amyloid β induces mitochondrial dysfunction leading to apoptotic cell death |
| Q48284335 | Mitochondrial Perturbation in Alzheimer's Disease and Diabetes |
| Q39355183 | Molecular Pathogenesis of Alzheimer's Disease: An Update |
| Q38045223 | Neurodegeneration, β-amyloid and mood disorders: state of the art and future perspectives |
| Q30843406 | Neuronal p38α mediates synaptic and cognitive dysfunction in an Alzheimer's mouse model by controlling β-amyloid production. |
| Q37977251 | Neuronal receptors as targets for the action of amyloid-beta protein (Aβ) in the brain |
| Q41665177 | Neurovascular Interaction Promotes the Morphological and Functional Maturation of Cortical Neurons |
| Q34368970 | New therapeutic approaches for Alzheimer's disease and cerebral amyloid angiopathy |
| Q89498915 | Nitroxidized-Albumin Advanced Glycation End Product and Rheumatoid Arthritis |
| Q61135270 | Overexpression of endophilin A1 exacerbates synaptic alterations in a mouse model of Alzheimer's disease |
| Q45873646 | PINK1 signalling rescues amyloid pathology and mitochondrial dysfunction in Alzheimer's disease |
| Q47775496 | Persistent Increase in Microglial RAGE Contributes to Chronic Stress-Induced Priming of Depressive-like Behavior. |
| Q34417983 | Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis |
| Q34054508 | Pinocembrin protects human brain microvascular endothelial cells against fibrillar amyloid-β(1-40) injury by suppressing the MAPK/NF-κB inflammatory pathways |
| Q91703077 | Presynaptic long-term potentiation requires extracellular signal-regulated kinases in the anterior cingulate cortex |
| Q43288312 | Quantitative reverse-transcription polymerase chain reaction analysis of Alzheimer's-associated genes in mouse scrapie |
| Q26744630 | RAGE Expression and ROS Generation in Neurons: Differentiation versus Damage |
| Q37455857 | RAGE and Alzheimer's disease: a progression factor for amyloid-beta-induced cellular perturbation? |
| Q33801587 | RAGE inhibition in microglia prevents ischemia-dependent synaptic dysfunction in an amyloid-enriched environment |
| Q36942363 | RAGE is a key cellular target for Abeta-induced perturbation in Alzheimer's disease |
| Q47867340 | RAGE mediates Aβ accumulation in a mouse model of Alzheimer's disease via modulation of β- and γ-secretase activity |
| Q38094691 | RAGE regulation and signaling in inflammation and beyond. |
| Q39638245 | RAGE-dependent signaling in microglia contributes to neuroinflammation, Abeta accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease |
| Q24315809 | RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction |
| Q37599419 | RAGE: the beneficial and deleterious effects by diverse mechanisms of actions |
| Q34999535 | Recent advances in our understanding of neurodegeneration |
| Q21285070 | Receptor for advanced glycation end products and its involvement in inflammatory diseases |
| Q47414857 | Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment. |
| Q37259863 | Receptor for advanced glycation end products: its role in Alzheimer's disease and other neurological diseases |
| Q35663419 | Soluble Aβ oligomer production and toxicity |
| Q42738430 | Soluble Aβ oligomers inhibit long-term potentiation through a mechanism involving excessive activation of extrasynaptic NR2B-containing NMDA receptors |
| Q24656613 | Soluble oligomers of amyloid Beta protein facilitate hippocampal long-term depression by disrupting neuronal glutamate uptake |
| Q40984233 | Somatostatin binds to the human amyloid β peptide and favors the formation of distinct oligomers |
| Q38000563 | Stimulation-dependent intraspinal microtubules and synaptic failure in Alzheimer's disease: a review |
| Q48965555 | Suppression of a neocortical potassium channel activity by intracellular amyloid-β and its rescue with Homer1a. |
| Q37667218 | Synaptic Dysfunction in Alzheimer's Disease and Glaucoma: From Common Degenerative Mechanisms Toward Neuroprotection |
| Q35869170 | Synaptic depression and aberrant excitatory network activity in Alzheimer's disease: two faces of the same coin? |
| Q47865490 | Synthetic Fragment of Receptor for Advanced Glycation End Products Prevents Memory Loss and Protects Brain Neurons in Olfactory Bulbectomized Mice. |
| Q34792043 | Targets for AD treatment: conflicting messages from γ-secretase inhibitors |
| Q37424029 | Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation |
| Q41980641 | The Complexity of Sporadic Alzheimer's Disease Pathogenesis: The Role of RAGE as Therapeutic Target to Promote Neuroprotection by Inhibiting Neurovascular Dysfunction |
| Q33639414 | The Influence of Fluorine on the Disturbances of Homeostasis in the Central Nervous System |
| Q37736982 | The chemokine CXCL12 mediates the anti-amyloidogenic action of painless human nerve growth factor |
| Q28744600 | The molecular assembly of amyloid aβ controls its neurotoxicity and binding to cellular proteins |
| Q36454454 | The novel RAGE interactor PRAK is associated with autophagy signaling in Alzheimer's disease pathogenesis |
| Q36040645 | The p38 MAP Kinase Family as Regulators of Proinflammatory Cytokine Production in Degenerative Diseases of the CNS. |
| Q40991050 | Time-dependent reversal of synaptic plasticity induced by physiological concentrations of oligomeric Aβ42: an early index of Alzheimer's disease. |
| Q50716120 | β-Asarone Mitigates Amyloidosis and Downregulates RAGE in a Transgenic Mouse Model of Alzheimer's Disease. |
Search more.