| scholarly article | Q13442814 |
| P356 | DOI | 10.1152/AJPENDO.00555.2002 |
| P8608 | Fatcat ID | release_34wib7zfvfe5rc5cmglwj35h4a |
| P698 | PubMed publication ID | 12644450 |
| P5875 | ResearchGate publication ID | 10850455 |
| P2093 | author name string | C Randell Brown | |
| Charles H Lang | |||
| Ly Q Hong-Brown | |||
| Robert A Frost | |||
| Robert N Cooney | |||
| P2860 | cites work | Mechanism of signaling by growth hormone receptor | Q34403059 |
| Role of growth hormone, insulin-like growth factor-I, and insulin-like growth factor binding proteins in the catabolic response to injury and infection | Q34601336 | ||
| Activation of acute phase response factor (APRF)/Stat3 transcription factor by growth hormone. | Q38298544 | ||
| Tumor necrosis factor mediates hepatic growth hormone resistance during sepsis | Q40711804 | ||
| GH regulation of IGF-I and suppressor of cytokine signaling gene expression in C2C12 skeletal muscle cells | Q40784679 | ||
| Critically ill patients have high basal growth hormone levels with attenuated oscillatory activity associated with low levels of insulin-like growth factor-I. | Q41164502 | ||
| Insulinlike growth factor 1 production is inhibited in human sepsis | Q41399625 | ||
| Inhibition by interleukin-1 beta and tumor necrosis factor-alpha of the insulin-like growth factor I messenger ribonucleic acid response to growth hormone in rat hepatocyte primary culture | Q42435114 | ||
| Acute alterations in growth hormone-insulin-like growth factor axis in humans injected with endotoxin | Q42442408 | ||
| Growth hormone preferentially induces the rapid, transient expression of SOCS-3, a novel inhibitor of cytokine receptor signaling | Q42449457 | ||
| Growth hormone stimulates tyrosine phosphorylation of JAK2 and STAT5, but not insulin receptor substrate-1 or SHC proteins in liver and skeletal muscle of normal rats in vivo | Q42522023 | ||
| Growth Hormone and Prolactin Stimulate Tyrosine Phosphorylation of Insulin Receptor Substrate-1, -2, and -3, Their Association with p85 Phosphatidylinositol 3-Kinase (PI3-kinase), and Concomitantly PI3-kinase Activation via JAK2 Kinase | Q42829023 | ||
| Liver-specific igf-1 gene deletion leads to muscle insulin insensitivity | Q43596024 | ||
| Vasodilator mechanisms in the coronary circulation of endothelial nitric oxide synthase-deficient mice | Q50509182 | ||
| Systemic and splanchnic metabolic response to exogenous human growth hormone | Q51569906 | ||
| Growth Hormone-promoted Tyrosyl Phosphorylation of SHC Proteins and SHC Association with Grb2 | Q56016875 | ||
| IMPAIRED STAT3 ACTIVATION FOLLOWING LOCALIZED INFLAMMATORY STIMULUS IN IL-6-DEFICIENT MICE | Q57272891 | ||
| A family of cytokine-inducible inhibitors of signalling | Q24313168 | ||
| A new protein containing an SH2 domain that inhibits JAK kinases | Q24313242 | ||
| Growth enhancement in suppressor of cytokine signaling 2 (SOCS-2)-deficient mice is dependent on signal transducer and activator of transcription 5b (STAT5b) | Q28201918 | ||
| Burn-induced changes in IGF-I and IGF-binding proteins are partially glucocorticoid dependent | Q28564447 | ||
| Differential expression of suppressors of cytokine signalling genes in response to nutrition and growth hormone in the septic rat | Q28566970 | ||
| Endotoxin-induced inhibition of growth hormone receptor signaling in rat liver in vivo | Q28571622 | ||
| Structure and function of a new STAT-induced STAT inhibitor | Q28584775 | ||
| Stat5a and Stat5b proteins have essential and nonessential, or redundant, roles in cytokine responses | Q28592023 | ||
| Potentiation of growth hormone-induced liver suppressors of cytokine signaling messenger ribonucleic acid by cytokines | Q30615185 | ||
| Negative regulators of cytokine signal transduction | Q33754091 | ||
| Growth factors in critical illness: regulation and therapeutic aspects | Q33775248 | ||
| The role of STAT proteins in growth hormone signaling | Q33941394 | ||
| Signal transduction via the growth hormone receptor | Q34331449 | ||
| P433 | issue | 1 | |
| P921 | main subject | phosphorylation | Q242736 |
| sepsis | Q183134 | ||
| P304 | page(s) | E63-72 | |
| P577 | publication date | 2003-03-18 | |
| P1433 | published in | American Journal of Physiology - Endocrinology and Metabolism | Q15765671 |
| P1476 | title | Sepsis-induced muscle growth hormone resistance occurs independently of STAT5 phosphorylation | |
| P478 | volume | 285 |
| Q28305674 | Alcohol-induced disruption of endocrine signaling |
| Q53876696 | Alterations in growth hormone receptor abundance regulate growth hormone signaling in murine obstructive cholestasis |
| Q34368943 | Cytokine inhibition of JAK-STAT signaling: a new mechanism of growth hormone resistance |
| Q33778791 | Disruption of genes encoding eIF4E binding proteins-1 and -2 does not alter basal or sepsis-induced changes in skeletal muscle protein synthesis in male or female mice |
| Q28579041 | Endotoxin attenuates growth hormone-induced hepatic insulin-like growth factor I expression by inhibiting JAK2/STAT5 signal transduction and STAT5b DNA binding |
| Q37281129 | Endotoxin-induced growth hormone resistance in skeletal muscle |
| Q28271735 | Endotoxin-induced proteolytic reduction in hepatic growth hormone (GH) receptor: a novel mechanism for GH insensitivity |
| Q36699521 | Hepatic Growth Hormone Resistance After Acute Injury |
| Q36617109 | The inhibitory effects of interleukin-1 on growth hormone action during catabolic illness |
| Q46069078 | Uremia attenuates growth hormone-stimulated insulin-like growth factor-1 expression, a process worsened by inflammation |
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