scholarly article | Q13442814 |
P2093 | author name string | Richard T Lee | |
Jan Lammerding | |||
P2860 | cites work | Myoferlin, a candidate gene and potential modifier of muscular dystrophy | Q22010963 |
Loss of A-type lamin expression compromises nuclear envelope integrity leading to muscular dystrophy | Q24681029 | ||
Defective membrane repair in dysferlin-deficient muscular dystrophy | Q28203095 | ||
Abnormal nuclear shape and impaired mechanotransduction in emerin-deficient cells | Q28268495 | ||
Dysferlin, a novel skeletal muscle gene, is mutated in Miyoshi myopathy and limb girdle muscular dystrophy | Q28281738 | ||
A gene related to Caenorhabditis elegans spermatogenesis factor fer-1 is mutated in limb-girdle muscular dystrophy type 2B | Q28281749 | ||
In vivo and in vitro examination of the functional significances of novel lamin gene mutations in heart failure patients | Q30478576 | ||
Cardiac involvement in Emery-Dreifuss muscular dystrophy | Q33152322 | ||
Dysferlin and the plasma membrane repair in muscular dystrophy | Q33201061 | ||
Plasma Membrane Disruption: Repair, Prevention, Adaptation | Q34271469 | ||
A clamping mechanism involved in SNARE-dependent exocytosis | Q34541145 | ||
The heart in human dystrophinopathies | Q35067344 | ||
Nuclear envelope proteins and neuromuscular diseases | Q35093315 | ||
Lamin A/C deficiency causes defective nuclear mechanics and mechanotransduction. | Q35632054 | ||
Disruptions of muscle fiber plasma membranes. Role in exercise-induced damage | Q35831693 | ||
Dysferlin-mediated membrane repair protects the heart from stress-induced left ventricular injury | Q35865366 | ||
Laminopathies | Q35923554 | ||
An emergency response team for membrane repair | Q36146942 | ||
Desmin myopathy, a skeletal myopathy with cardiomyopathy caused by mutations in the desmin gene | Q40893835 | ||
Dystrophic heart failure blocked by membrane sealant poloxamer | Q46604988 | ||
Ultrastructural abnormality of sarcolemmal nuclei in Emery-Dreifuss muscular dystrophy (EDMD). | Q47699129 | ||
Poloxamer 188 failed to prevent exercise-induced membrane breakdown in mdx skeletal muscle fibers. | Q50473991 | ||
Decreased mechanical stiffness in LMNA-/- cells is caused by defective nucleo-cytoskeletal integrity: implications for the development of laminopathies. | Q51616068 | ||
Loss of cytoplasmic basic fibroblast growth factor from physiologically wounded myofibers of normal and dystrophic muscle. | Q52515753 | ||
Lamins A and C but not lamin B1 regulate nuclear mechanics. | Q53613002 | ||
Architectural abnormalities in muscle nuclei. Ultrastructural differences between X-linked and autosomal dominant forms of EDMD | Q73366593 | ||
Pharmacokinetics of RheothRx injection in healthy male volunteers | Q73533574 | ||
Nuclear sequestration of delta-sarcoglycan disrupts the nuclear localization of lamin A/C and emerin in cardiomyocytes | Q79428627 | ||
P433 | issue | 7 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 1749-1752 | |
P577 | publication date | 2007-07-01 | |
P1433 | published in | Journal of Clinical Investigation | Q3186904 |
P1476 | title | Torn apart: membrane rupture in muscular dystrophies and associated cardiomyopathies | |
P478 | volume | 117 |
Q34118633 | Biochemical markers of muscular damage |
Q93384433 | Cardiac MRI biomarkers for Duchenne muscular dystrophy |
Q34497771 | Caveolae protect endothelial cells from membrane rupture during increased cardiac output |
Q56981953 | Dysferlin, dystrophy, and dilatative cardiomyopathy |
Q90634574 | Heart disease in a mutant mouse model of spontaneous eosinophilic myocarditis maps to three loci |
Q42209023 | Muscle membrane repair and inflammatory attack in dysferlinopathy. |
Q49704946 | Nuclear envelope proteins Nesprin2 and LaminA regulate proliferation and apoptosis of vascular endothelial cells in response to shear stress |
Q46801990 | Qualitative electron microscopic analysis of cultured chick embryonic cardiac and skeletal muscle cells: the cellular effect of coenzyme q10 after exposure to triton x-100. |
Q48695970 | The susceptibility to experimental autoimmune encephalomyelitis is not related to dysferlin-deficiency |
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