scholarly article | Q13442814 |
P356 | DOI | 10.1515/BC.2010.133 |
P8608 | Fatcat ID | release_xumonedwsvddbe4yvtadjztbta |
P698 | PubMed publication ID | 20868226 |
P50 | author | Lars-Oliver Klotz | Q40367538 |
P2093 | author name string | Niloofar Ale-Agha | |
Catrin Albrecht | |||
P2860 | cites work | Tyrosine phosphorylation and dissociation of occludin-ZO-1 and E-cadherin-beta-catenin complexes from the cytoskeleton by oxidative stress | Q24534909 |
Nanotoxicology: an emerging discipline evolving from studies of ultrafine particles | Q24536032 | ||
Life cycle of connexins in health and disease | Q24541428 | ||
The effects of connexin phosphorylation on gap junctional communication | Q24603834 | ||
Oops they did it again! Carbon nanotubes hoax scientists in viability assays | Q29615573 | ||
Microtubule plus-end-tracking proteins target gap junctions directly from the cell interior to adherens junctions | Q30479975 | ||
Oxidative-dependent integration of signal transduction with intercellular gap junctional communication in the control of gene expression | Q34103121 | ||
Cross-talk between pulmonary injury, oxidant stress, and gap junctional communication. | Q34103139 | ||
Structural and functional diversity of connexin genes in the mouse and human genome | Q34137753 | ||
Regulation of connexin-43 gap junctional intercellular communication by mitogen-activated protein kinase | Q34463403 | ||
Oxidant-induced signaling: effects of peroxynitrite and singlet oxygen | Q34658898 | ||
Signal transduction pathways relevant for neoplastic effects of fibrous and non-fibrous particles | Q35853217 | ||
Phosphoinositide 3-kinase signaling in the cellular response to oxidative stress | Q36103291 | ||
Phosphorylation of connexin43 on serine368 by protein kinase C regulates gap junctional communication | Q36328306 | ||
HuR regulates gap junctional intercellular communication by controlling beta-catenin levels and adherens junction integrity | Q37442157 | ||
Supplemental role of the Ames mutation assay and gap junction intercellular communication in studies of possible carcinogenic compounds from diesel exhaust particles | Q39654643 | ||
Impact of the FcgammaII-receptor on quartz uptake and inflammatory response by alveolar macrophages | Q39994857 | ||
Epidermal growth factor- and stress-induced loss of gap junctional communication is mediated by ERK-1/ERK-2 but not ERK-5 in rat liver epithelial cells | Q40065966 | ||
Expression and role of EGFR ligands induced in airway cells by PM2.5 and its components | Q40084505 | ||
Inflammatory effects of respirable quartz collected in workplaces versus standard DQ12 quartz: particle surface correlates | Q40785435 | ||
Disruption of gap junctional communication by the platelet-derived growth factor is mediated via multiple signaling pathways | Q40963041 | ||
Reactive oxygen species as mediators of membrane-dependent signaling induced by ultrafine particles | Q43008719 | ||
Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to quartz particles | Q43274176 | ||
Inhibition of connexin43 gap junctional intercellular communication by TPA requires ERK activation | Q43704495 | ||
Silica-induced generation of extracellular factor(s) increases reactive oxygen species in human bronchial epithelial cells | Q43994096 | ||
Epidermal growth factor receptor is a common mediator of quinone-induced signaling leading to phosphorylation of connexin-43: role of glutathione and tyrosine phosphatases | Q44521284 | ||
Inflammatory time course after quartz instillation: role of tumor necrosis factor-alpha and particle surface | Q44932426 | ||
Activation of endothelial cells after exposure to ambient ultrafine particles: the role of NADPH oxidase | Q46048540 | ||
Doxorubicin induces EGF receptor-dependent downregulation of gap junctional intercellular communication in rat liver epithelial cells | Q46447814 | ||
P433 | issue | 11 | |
P921 | main subject | nanoparticle | Q61231 |
P304 | page(s) | 1333-1339 | |
P577 | publication date | 2010-11-01 | |
P1433 | published in | Biological Chemistry | Q4914948 |
P1476 | title | Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to carbon or silica-based nanoparticles | |
P478 | volume | 391 |
Q38251996 | 1,4-naphthoquinones: from oxidative damage to cellular and inter-cellular signaling |
Q28389467 | Biocompatibility assessment of Si-based nano- and micro-particles |
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Q28395335 | Effects of carbon nanotubes on intercellular communication and involvement of IL-1 genes |
Q38852801 | Excess titanium dioxide nanoparticles on the cell surface induce cytotoxicity by hindering ion exchange and disrupting exocytosis processes. |
Q28396268 | Force dependent internalization of magnetic nanoparticles results in highly loaded endothelial cells for use as potential therapy delivery vectors |
Q38648995 | Free radicals and related reactive species as mediators of tissue injury and disease: implications for Health. |
Q57377095 | Genome-wide transcriptional analysis of silica nanoparticle-induced toxicity in zebrafish embryos |
Q37722729 | Inhibition of gap junction intercellular communication is involved in silica nanoparticles-induced H9c2 cardiomyocytes apoptosis via the mitochondrial pathway |
Q33981469 | Mitigation of quantum dot cytotoxicity by microencapsulation |
Q46242351 | Silver nanoparticles increase connexin43-mediated gap junctional intercellular communication in HaCaT cells through activation of reactive oxygen species and mitogen-activated protein kinase signal pathway. |
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