| scholarly article | Q13442814 |
| P356 | DOI | 10.1152/JN.00482.2009 |
| P698 | PubMed publication ID | 19828728 |
| P2093 | author name string | Arnaud Pambo-Pambo | |
| Jacques Durand | |||
| Jean-Patrick Gueritaud | |||
| P2860 | cites work | Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1 | Q22299419 |
| Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis | Q28131805 | ||
| From Charcot to Lou Gehrig: deciphering selective motor neuron death in ALS | Q28206361 | ||
| Overloading of stable and exclusion of unstable human superoxide dismutase-1 variants in mitochondria of murine amyotrophic lateral sclerosis models | Q28235353 | ||
| Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation | Q29547561 | ||
| ALS: a disease of motor neurons and their nonneuronal neighbors | Q29618000 | ||
| Unraveling the mechanisms involved in motor neuron degeneration in ALS | Q29619073 | ||
| Changes in the spinal cord proteome of an amyotrophic lateral sclerosis murine model determined by differential in-gel electrophoresis | Q33428448 | ||
| Perinatal development of lumbar motoneurons and their inputs in the rat. | Q34134116 | ||
| Familial amyotrophic lateral sclerosis | Q34547620 | ||
| Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis | Q34718757 | ||
| Update on the glutamatergic neurotransmitter system and the role of excitotoxicity in amyotrophic lateral sclerosis | Q34936454 | ||
| Early alterations in the electrophysiological properties of rat spinal motoneurones following neonatal axotomy | Q36137570 | ||
| Variation in aggregation propensities among ALS-associated variants of SOD1: correlation to human disease. | Q37295060 | ||
| Bistability of alpha-motoneurones in the decerebrate cat and in the acute spinal cat after intravenous 5-hydroxytryptophan. | Q41425822 | ||
| Slowing of axonal transport is a very early event in the toxicity of ALS-linked SOD1 mutants to motor neurons | Q41644415 | ||
| Plateau potentials in sacrocaudal motoneurons of chronic spinal rats, recorded in vitro | Q43766624 | ||
| Developmental expression of voltage-dependent calcium currents in identified mouse motoneurons | Q43798162 | ||
| Ionic mechanisms underlying the firing properties of rat neonatal motoneurons studied in vitro. | Q44224732 | ||
| A-, T-, and H-type currents shape intrinsic firing of developing rat abducens motoneurons | Q44372226 | ||
| Persistent sodium and calcium currents cause plateau potentials in motoneurons of chronic spinal rats | Q44425187 | ||
| Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice. | Q44605286 | ||
| Role of persistent sodium and calcium currents in motoneuron firing and spasticity in chronic spinal rats | Q44755367 | ||
| Altered sensorimotor development in a transgenic mouse model of amyotrophic lateral sclerosis | Q45153262 | ||
| Increased persistent Na(+) current and its effect on excitability in motoneurones cultured from mutant SOD1 mice. | Q45223589 | ||
| ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions. | Q46135324 | ||
| Mechanisms underlying the early phase of spike frequency adaptation in mouse spinal motoneurones | Q46477248 | ||
| Persistent sodium currents and repetitive firing in motoneurons of the sacrocaudal spinal cord of adult rats | Q46799657 | ||
| Bistability in spinal motoneurons in vivo: systematic variations in persistent inward currents | Q47681439 | ||
| Postnatal changes in the inactivation properties of voltage-gated sodium channels contribute to the mature firing pattern of spinal motoneurons. | Q47830281 | ||
| Altered excitability of motor neurons in a transgenic mouse model of familial amyotrophic lateral sclerosis. | Q47874345 | ||
| Differential maturation of motoneurons innervating ankle flexor and extensor muscles in the neonatal rat. | Q47896275 | ||
| Riluzole interacts with voltage-activated sodium and potassium currents in cultured rat cortical neurons | Q47930570 | ||
| Factors influencing motoneuron rhythmic firing: results from a voltage-clamp study | Q47957233 | ||
| Facilitation of somatic calcium channels can evoke prolonged tail currents in rat hypoglossal motoneurons | Q48158181 | ||
| Minute quantities of misfolded mutant superoxide dismutase-1 cause amyotrophic lateral sclerosis | Q48177170 | ||
| Enhancement of bistability in spinal motoneurons in vivo by the noradrenergic alpha1 agonist methoxamine | Q48214058 | ||
| Effect of precocious locomotor activity on the development of motoneurones and motor units of slow and fast muscles in rat. | Q48332089 | ||
| Voltage-dependent sodium channels in spinal cord motor neurons display rapid recovery from fast inactivation in a mouse model of amyotrophic lateral sclerosis | Q48450897 | ||
| Postinhibitory rebound during locomotor-like activity in neonatal rat motoneurons in vitro | Q48531115 | ||
| Temporal sequence of changes in electrophysiological properties of oculomotor motoneurons during postnatal development. | Q48572517 | ||
| Frequency-current relationships of rat hindlimb alpha-motoneurones | Q48708355 | ||
| Synaptic activation of plateaus in hindlimb motoneurons of decerebrate cats. | Q51497750 | ||
| Electrical properties of motoneurons in the spinal cord of rat embryos. | Q51770896 | ||
| Morphological differences between wild-type and transgenic superoxide dismutase 1 lumbar motoneurons in postnatal mice. | Q51949007 | ||
| Development of L-type calcium channels and a nifedipine-sensitive motor activity in the postnatal mouse spinal cord. | Q52173220 | ||
| Development of ionic currents underlying changes in action potential waveforms in rat spinal motoneurons. | Q52181242 | ||
| Postnatal changes in rat hypoglossal motoneuron membrane properties. | Q52217808 | ||
| Repetitive firing properties of developing rat brainstem motoneurones. | Q54167443 | ||
| Electrophysiological and morphological properties of rat abducens motoneurones. | Q54363378 | ||
| An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria | Q54965159 | ||
| P433 | issue | 6 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 3627-3642 | |
| P577 | publication date | 2009-10-14 | |
| P1433 | published in | Journal of Neurophysiology | Q1709863 |
| P1476 | title | Early excitability changes in lumbar motoneurons of transgenic SOD1G85R and SOD1G(93A-Low) mice | |
| P478 | volume | 102 |
| Q36479616 | ALS biomarkers for therapy development: State of the field and future directions |
| Q41087321 | Aberrant association of misfolded SOD1 with Na(+)/K(+)ATPase-α3 impairs its activity and contributes to motor neuron vulnerability in ALS. |
| Q87132998 | Adult spinal motoneurones are not hyperexcitable in a mouse model of inherited amyotrophic lateral sclerosis |
| Q43222578 | Alterations in the motor neuron-renshaw cell circuit in the Sod1(G93A) mouse model |
| Q51871496 | Altered postnatal maturation of electrical properties in spinal motoneurons in a mouse model of amyotrophic lateral sclerosis. |
| Q37564322 | Astrocytes expressing mutant SOD1 and TDP43 trigger motoneuron death that is mediated via sodium channels and nitroxidative stress |
| Q28476121 | Characterization of a novel SOD-1(G93A) transgenic mouse line with very decelerated disease development |
| Q37407302 | Characterization of early pathogenesis in the SOD1(G93A) mouse model of ALS: part II, results and discussion |
| Q48099230 | Chronic electromyograms in treadmill running SOD1 mice reveal early changes in muscle activation |
| Q35501852 | Clinical Measures of Disease Progression in Amyotrophic Lateral Sclerosis |
| Q35357235 | Comparison of dendritic calcium transients in juvenile wild type and SOD1(G93A) mouse lumbar motoneurons |
| Q36018256 | Developing electrical properties of postnatal mouse lumbar motoneurons |
| Q36718274 | Early interneuron dysfunction in ALS: insights from a mutant sod1 zebrafish model |
| Q41957945 | Early intrinsic hyperexcitability does not contribute to motoneuron degeneration in amyotrophic lateral sclerosis. |
| Q41862808 | Early pathogenesis in the adult-onset neurodegenerative disease amyotrophic lateral sclerosis |
| Q33900692 | Effect of fluoxetine on disease progression in a mouse model of ALS. |
| Q35951394 | Effect of prolonged riluzole exposure on cultured motoneurons in a mouse model of ALS. |
| Q28533705 | Electrophysiologic biomarkers for assessing disease progression and the effect of riluzole in SOD1 G93A ALS mice |
| Q36351201 | Evidence from computer simulations for alterations in the membrane biophysical properties and dendritic processing of synaptic inputs in mutant superoxide dismutase-1 motoneurons |
| Q97066870 | Exciting Complexity: The Role of Motor Circuit Elements in ALS Pathophysiology |
| Q37534101 | Gamma motor neurons survive and exacerbate alpha motor neuron degeneration in ALS. |
| Q55215170 | Hypoexcitability precedes denervation in the large fast-contracting motor units in two unrelated mouse models of ALS. |
| Q38653220 | Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities |
| Q27687333 | Inhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis |
| Q36821693 | Intrinsic and synaptic homeostatic plasticity in motoneurons from mice with glycine receptor mutations. |
| Q54970000 | Kv1.2 Channels Promote Nonlinear Spiking Motoneurons for Powering Up Locomotion. |
| Q36880259 | Lack of sigma-1 receptor exacerbates ALS progression in mice |
| Q37150806 | Marked changes in dendritic structure and spine density precede significant neuronal death in vulnerable cortical pyramidal neuron populations in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis |
| Q33845902 | Monoaminergic control of spinal locomotor networks in SOD1G93A newborn mice |
| Q47118168 | Motor Areas Show Altered Dendritic Structure in an Amyotrophic Lateral Sclerosis Mouse Model. |
| Q91970420 | Motor Neuron Susceptibility in ALS/FTD |
| Q38110501 | Motor unit |
| Q36924065 | Mutant SOD1-expressing astrocytes release toxic factors that trigger motoneuron death by inducing hyperexcitability |
| Q47933159 | Opposite Synaptic Alterations at the Neuromuscular Junction in an ALS Mouse Model: When Motor Units Matter. |
| Q34399203 | Persistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis |
| Q88349389 | Physiology of the fasciculation potentials in amyotrophic lateral sclerosis: which motor units fasciculate? |
| Q36084199 | Potassium currents dynamically set the recruitment and firing properties of F-type motoneurons in neonatal mice |
| Q44652546 | Pre- and postsynaptic mechanisms underlying inhibition of hypoglossal motor neuron excitability by riluzole |
| Q35701794 | Reactive oxygen species trigger motoneuron death in non-cell-autonomous models of ALS through activation of c-Abl signaling |
| Q92200994 | Relaxation of synaptic inhibitory events as a compensatory mechanism in fetal SOD spinal motor networks |
| Q46313436 | SOD1 Mutations Causing Familial Amyotrophic Lateral Sclerosis Induce Toxicity in Astrocytes: Evidence for Bystander Effects in a Continuum of Astrogliosis |
| Q37894600 | SOD1 and TDP-43 animal models of amyotrophic lateral sclerosis: recent advances in understanding disease toward the development of clinical treatments |
| Q34601264 | Selective degeneration of a physiological subtype of spinal motor neuron in mice with SOD1-linked ALS |
| Q39468775 | Selective mitochondrial Ca2+ uptake deficit in disease endstage vulnerable motoneurons of the SOD1G93A mouse model of amyotrophic lateral sclerosis |
| Q42369773 | Shortcomings in the Current Amyotrophic Lateral Sclerosis Trials and Potential Solutions for Improvement |
| Q39169781 | Sigma-1 (σ1) Receptor in Memory and Neurodegenerative Diseases |
| Q48258033 | Sigma-1 Receptor in Motoneuron Disease |
| Q58671282 | Sodium-Mediated Plateau Potentials in Lumbar Motoneurons of Neonatal Rats |
| Q92797054 | Sp1-regulated expression of p11 contributes to motor neuron degeneration by membrane insertion of TASK1 |
| Q36494315 | State of the field: An informatics-based systematic review of the SOD1-G93A amyotrophic lateral sclerosis transgenic mouse model |
| Q24607879 | Stronger is not always better: could a bodybuilding dietary supplement lead to ALS? |
| Q58256286 | THEME 11 IMAGING, ELECTROPHYSIOLOGY AND MARKERS OF DISEASE PROGRESSION |
| Q88602881 | The Preparation of Oblique Spinal Cord Slices for Ventral Root Stimulation |
| Q41842390 | Voltage-gated calcium channels are abnormal in cultured spinal motoneurons in the G93A-SOD1 transgenic mouse model of ALS. |
Search more.