human | Q5 |
P496 | ORCID iD | 0000-0001-6848-7407 |
P108 | employer | University College London | Q193196 |
University of Barcelona | Q219615 | ||
Leonard M. Miller School of Medicine | Q6525526 | ||
Royal Free Hospital | Q7374196 | ||
Institute for Research and Technology in Food and Agriculture | Q9006472 | ||
Institute of Marine Sciences | Q11926742 | ||
Instituto de Productos Naturales y Agrobiología | Q30263829 | ||
Fundació Clínic per a la Recerca Biomèdica | Q30287378 | ||
Centre for Biomedical Network Research on Rare Diseases | Q30296152 | ||
Foundation for Research and Prevention of AIDS in Spain | Q45135795 | ||
University College London Medical School - Royal Free Campus | Q86028187 | ||
P106 | occupation | researcher | Q1650915 |
P21 | sex or gender | female | Q6581072 |
Q124979860 | A Mitocentric View of the Main Bacterial and Parasitic Infectious Diseases in the Pediatric Population |
Q112288356 | A longitudinal study of gene expression in first-episode schizophrenia; exploring relapse mechanisms by co-expression analysis in peripheral blood |
Q47120743 | Addendum: Morén, C.; Hernández, S.; Guitart-Mampel, M.; Garrabou, G. Mitochondrial Toxicity in Human Pregnancy: An Update on Clinical and Experimental Approaches in the Last 10 Years. Int. J. Environ. Res. Public Health 2014, 11, 9897–9918. |
Q39800044 | BACE-1, PS-1 and sAPPβ levels are increased in plasma from sporadic inclusion body myositis patients: surrogate biomarkers among inflammatory myopathies. |
Q90263483 | Bioenergetics and Autophagic Imbalance in Patients-Derived Cell Models of Parkinson Disease Supports Systemic Dysfunction in Neurodegeneration |
Q64040134 | Cardiac and mitochondrial function in HIV-uninfected fetuses exposed to antiretroviral treatment |
Q101125479 | Decreased Glycolysis as Metabolic Fingerprint of Endothelial Cells in Chronic Thromboembolic Pulmonary Hypertension |
Q40998695 | Decreased Mitochondrial Function Among Healthy Infants Exposed to Antiretrovirals During Gestation, Delivery and the Neonatal Period. |
Q101215006 | Disrupted Mitochondrial and Metabolic Plasticity Underlie Comorbidity between Age-Related and Degenerative Disorders as Parkinson Disease and Type 2 Diabetes Mellitus |
Q42938219 | Evolution of mitochondrial DNA content after planned interruption of HAART in HIV-infected pediatric patients |
Q54976661 | Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson's disease. |
Q91373313 | GBA mutation promotes early mitochondrial dysfunction in 3D neurosphere models |
Q43271718 | Genetic and functional mitochondrial assessment of HIV-infected patients developing HAART-related hyperlactatemia |
Q37604361 | HIV-1 promonocytic and lymphoid cell lines: an in vitro model of in vivo mitochondrial and apoptotic lesion. |
Q57631516 | Hyperbaric oxygen therapy for carbon monoxide poisoning |
Q43296433 | Improvement of mitochondrial toxicity in patients receiving a nucleoside reverse-transcriptase inhibitor-sparing strategy: results from the Multicenter Study with Nevirapine and Kaletra (MULTINEKA). |
Q64039240 | Metabolic, mitochondrial, renal and hepatic safety of enfuvirtide and raltegravir antiretroviral administration: Randomized crossover clinical trial in healthy volunteers |
Q51703326 | Mitochondrial DNA (mtDNA) variants in the European haplogroups HV, JT, and U do not have a major role in schizophrenia. |
Q44128587 | Mitochondrial DNA depletion in oocytes of HIV-infected antiretroviral-treated infertile women |
Q51490832 | Mitochondrial DNA disturbances and deregulated expression of oxidative phosphorylation and mitochondrial fusion proteins in sporadic inclusion body myositis. |
Q57631525 | Mitochondrial Injury in Human Acute Carbon Monoxide Poisoning: The Effect of Oxygen Treatment |
Q96303722 | Mitochondrial Toxicogenomics for Antiretroviral Management: HIV Post-exposure Prophylaxis in Uninfected Patients |
Q41276695 | Mitochondrial and apoptotic in vitro modelling of differential HIV-1 progression and antiretroviral toxicity |
Q64764170 | Mitochondrial and autophagic alterations in skin fibroblasts from Parkinson disease patients with Parkin mutations |
Q57631521 | Mitochondrial assessment in asymptomatic HIV-infected paediatric patients on HAART |
Q50551965 | Mitochondrial damage in adipose tissue of untreated HIV-infected patients. |
Q41721533 | Mitochondrial disturbances in HIV pregnancies |
Q59217093 | Mitochondrial effects of 3 years of CD4-guided HIV treatment interruption |
Q54322317 | Mitochondrial evolution in HIV-infected children receiving first- or second-generation nucleoside analogues. |
Q45365531 | Mitochondrial impact of human immunodeficiency virus and antiretrovirals on infected pediatric patients with or without lipodystrophy |
Q34348392 | Mitochondrial toxicity in human pregnancy: an update on clinical and experimental approaches in the last 10 years |
Q47438814 | Molecular basis of reduced birth weight in smoking pregnant women: mitochondrial dysfunction and apoptosis |
Q112366884 | Neuronal induction and bioenergetics characterization of human forearm adipose stem cells from Parkinson's disease patients and healthy controls |
Q44108125 | Partial immunological and mitochondrial recovery after reducing didanosine doses in patients on didanosine and tenofovir-based regimens. |
Q57316869 | Perinatal outcomes, mitochondrial toxicity and apoptosis in HIV-treated pregnant women and in-utero-exposed newborn |
Q40318981 | Placental Mitochondrial Toxicity, Oxidative Stress, Apoptosis, and Adverse Perinatal Outcomes in HIV Pregnancies Under Antiretroviral Treatment Containing Zidovudine |
Q55059717 | Severe TK2 enzyme activity deficiency in patients with mild forms of myopathy. |
Q43496856 | Study of oxidative, enzymatic mitochondrial respiratory chain function and apoptosis in perinatally HIV-infected pediatric patients |
Q38783524 | The Role of Therapeutic Drugs on Acquired Mitochondrial Toxicity. |
Q44817068 | The effects of sepsis on mitochondria |
Q51765978 | Transcriptional alterations in skin fibroblasts from Parkinson's disease patients with parkin mutations. |
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