Mariarita Galbiati

researcher

Mariarita Galbiati is …
instance of (P31):
humanQ5

External links are
P10057IRIS UNIMI author ID10364
P496ORCID iD0000-0003-3250-5591
P3829Publons author ID2307817
P1053ResearcherIDC-1033-2016
P1153Scopus author ID6701815972

P108employerUniversity of MilanQ46210
P734family nameGalbiatiQ56538419
GalbiatiQ56538419
GalbiatiQ56538419
P735given nameMariaritaQ108929274
MariaritaQ108929274
P106occupationresearcherQ1650915
P21sex or genderfemaleQ6581072

Reverse relations

author (P50)
Q38275911ALS-related misfolded protein management in motor neurons and muscle cells
Q36189002Aberrant Autophagic Response in The Muscle of A Knock-in Mouse Model of Spinal and Bulbar Muscular Atrophy.
Q38986023Altered expression of 3-betahydroxysterol delta-24-reductase/selective Alzheimer's disease indicator-1 gene in Huntington's disease models.
Q30489907Androgen regulation of axon growth and neurite extension in motoneurons
Q57650052Androgens affect muscle, motor neuron, and survival in a mouse model of SOD1-related amyotrophic lateral sclerosis
Q41137209Astrocyte-neuron interactions in vitro: role of growth factors and steroids on LHRH dynamics.
Q54330074CAG repeat length in androgen receptor gene is not associated with amyotrophic lateral sclerosis.
Q37342284Differential autophagy power in the spinal cord and muscle of transgenic ALS mice.
Q44773170Effects of progesterone derivatives, dihydroprogesterone and tetrahydroprogesterone, on the subependymal layer of the adult rat.
Q62111179Excitatory amino acids as modulators of gonadotropin secretion
Q34361255Glial cells: a target for steroid hormones.
Q34973852Growth factors and steroid hormones: a complex interplay in the hypothalamic control of reproductive functions.
Q43644915Hypothalamic transforming growth factor beta1 and basic fibroblast growth factor mRNA expression is modified during the rat oestrous cycle.
Q38840491Inhibition of retrograde transport modulates misfolded protein accumulation and clearance in motoneuron diseases.
Q62111163Interactions between growth factors and steroids in the control of LHRH-secreting neurons
Q38164394Motoneuronal and muscle-selective removal of ALS-related misfolded proteins.
Q96604034Multiple Roles of Transforming Growth Factor Beta in Amyotrophic Lateral Sclerosis
Q38906989Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis
Q50694919Neuritin (cpg15) enhances the differentiating effect of NGF on neuronal PC12 cells.
Q45382418Neuritin 1 promotes neuronal migration.
Q56591723Neuroactive steroids and peripheral myelin proteins
Q37771693Proteasomal and autophagic degradative activities in spinal and bulbar muscular atrophy.
Q35214823Role of glial cells, growth factors and steroid hormones in the control of LHRH-secreting neurons
Q34355088Steroid effects on the gene expression of peripheral myelin proteins.
Q34673417Synergic prodegradative activity of Bicalutamide and trehalose on the mutant androgen receptor responsible for spinal and bulbar muscular atrophy
Q58771388Tdp-25 Routing to Autophagy and Proteasome Ameliorates its Aggregation in Amyotrophic Lateral Sclerosis Target Cells
Q62111182The Anterior Pituitary Gland as a Possible Site of Action of Kainic Acid
Q38634366The Role of the Protein Quality Control System in SBMA.
Q34292720The action of steroid hormones on peripheral myelin proteins: a possible new tool for the rebuilding of myelin?
Q35737086The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis
Q39155257The role of dynein mediated transport in the clearance of misfolded proteins responsible for motoneuron diseases
Q39689378The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).
Q36670771Transcriptional induction of the heat shock protein B8 mediates the clearance of misfolded proteins responsible for motor neuron diseases
Q40933380Transforming growth factor beta2 is able to modify mRNA levels and release of luteinizing hormone-releasing hormone in a immortalized hypothalamic cell line (GT1-1).
Q41149525Transforming growth factor-beta and astrocytic conditioned medium influence luteinizing hormone-releasing hormone gene expression in the hypothalamic cell line GT1.
Q58592816Trehalose induces autophagy via lysosomal-mediated TFEB activation in models of motoneuron degeneration

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