scholarly article | Q13442814 |
P356 | DOI | 10.1016/J.YJMCC.2009.04.013 |
P698 | PubMed publication ID | 19406125 |
P2093 | author name string | C Wolf | |
H M Piper | |||
H P Reusch | |||
Y Ladilov | |||
Y Abdallah | |||
K Meuter | |||
P2860 | cites work | Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria | Q31833702 |
Potassium channel openers depolarize hippocampal mitochondria | Q31923521 | ||
Preconditioning protects by inhibiting the mitochondrial permeability transition | Q33201290 | ||
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Evidence for mitochondrial K+ channels and their role in cardioprotection | Q35682888 | ||
Downmodulation of mitochondrial F0F1 ATP synthase by diazoxide in cardiac myoblasts: a dual effect of the drug | Q40173370 | ||
Selective inhibition of the contractile apparatus. A new approach to modification of infarct size, infarct composition, and infarct geometry during coronary artery occlusion and reperfusion | Q41112644 | ||
Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury | Q41601375 | ||
Cytosolic free Ca2+ in single rat heart cells during anoxia and reoxygenation | Q41955683 | ||
Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channels | Q43572789 | ||
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K(ATP) channel | Q43572811 | ||
Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotection | Q43794479 | ||
Diazoxide opens the mitochondrial permeability transition pore and alters Ca2+ transients in rat ventricular myocytes | Q44015023 | ||
Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heart | Q44027010 | ||
K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart | Q44087161 | ||
Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotection | Q44383873 | ||
The role of ATP in energy-deprivation contractures in unloaded rat ventricular myocytes | Q44533613 | ||
Reoxygenation-induced rigor-type contracture | Q44679153 | ||
K+-dependent regulation of matrix volume improves mitochondrial function under conditions mimicking ischemia-reperfusion | Q46160602 | ||
Culturing of calcium stable adult cardiac myocytes | Q70606821 | ||
Pretreatment with PKC activator protects cardiomyocytes against reoxygenation-induced hypercontracture independently of Ca2+ overload | Q73119938 | ||
Activation of mitochondrial K(ATP) channel elicits late preconditioning against myocardial infarction via protein kinase C signaling pathway | Q73254502 | ||
Protection of rat cardiomyocytes against simulated ischemia and reoxygenation by treatment with protein kinase C activator | Q74324525 | ||
ATP-sensitive K+ channel openers prevent Ca2+ overload in rat cardiac mitochondria | Q78157357 | ||
MitoK(ATP)-dependent changes in mitochondrial volume and in complex II activity during ischemic and pharmacological preconditioning of Langendorff-perfused rat heart | Q79217801 | ||
The mechanism by which the mitochondrial ATP-sensitive K+ channel opening and H2O2 inhibit the mitochondrial permeability transition | Q83832665 | ||
Myocardial ischemic contracture. Metabolites affect rigor tension development and stiffness | Q93874555 | ||
P433 | issue | 1 | |
P304 | page(s) | 270-276 | |
P577 | publication date | 2009-05-04 | |
P1433 | published in | Journal of Molecular and Cellular Cardiology | Q2061932 |
P1476 | title | Preconditioning with diazoxide prevents reoxygenation-induced rigor-type hypercontracture. | |
P478 | volume | 48 |
Q37777273 | Mitochondrial reprogramming through cardiac oxygen sensors in ischaemic heart disease |
Q34631989 | Remote ischemic preconditioning of cardiomyocytes inhibits the mitochondrial permeability transition pore independently of reduced calcium-loading or sarcKATP channel activation |
Q51318851 | Role of mitochondrial permeability transition pore and mitochondrial ATP-sensitive potassium channels in the protective effects of ischemic preconditioning in isolated hearts from fed and fasted rats. |
Q87424378 | Skeletal muscle post-conditioning by diazoxide, anti-oxidative and anti-apoptotic mechanisms |
Q37895248 | Use of nicorandil in cardiovascular disease and its optimization |
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