Preconditioning with diazoxide prevents reoxygenation-induced rigor-type hypercontracture.

scientific article published on 4 May 2009

Preconditioning with diazoxide prevents reoxygenation-induced rigor-type hypercontracture. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.YJMCC.2009.04.013
P698PubMed publication ID19406125

P2093author name stringC Wolf
H M Piper
H P Reusch
Y Ladilov
Y Abdallah
K Meuter
P2860cites workBioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondriaQ31833702
Potassium channel openers depolarize hippocampal mitochondriaQ31923521
Preconditioning protects by inhibiting the mitochondrial permeability transitionQ33201290
Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.Q34380288
Evidence for mitochondrial K+ channels and their role in cardioprotectionQ35682888
Downmodulation of mitochondrial F0F1 ATP synthase by diazoxide in cardiac myoblasts: a dual effect of the drugQ40173370
Selective inhibition of the contractile apparatus. A new approach to modification of infarct size, infarct composition, and infarct geometry during coronary artery occlusion and reperfusionQ41112644
Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injuryQ41601375
Cytosolic free Ca2+ in single rat heart cells during anoxia and reoxygenationQ41955683
Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channelsQ43572789
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K(ATP) channelQ43572811
Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotectionQ43794479
Diazoxide opens the mitochondrial permeability transition pore and alters Ca2+ transients in rat ventricular myocytesQ44015023
Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heartQ44027010
K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heartQ44087161
Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotectionQ44383873
The role of ATP in energy-deprivation contractures in unloaded rat ventricular myocytesQ44533613
Reoxygenation-induced rigor-type contractureQ44679153
K+-dependent regulation of matrix volume improves mitochondrial function under conditions mimicking ischemia-reperfusionQ46160602
Culturing of calcium stable adult cardiac myocytesQ70606821
Pretreatment with PKC activator protects cardiomyocytes against reoxygenation-induced hypercontracture independently of Ca2+ overloadQ73119938
Activation of mitochondrial K(ATP) channel elicits late preconditioning against myocardial infarction via protein kinase C signaling pathwayQ73254502
Protection of rat cardiomyocytes against simulated ischemia and reoxygenation by treatment with protein kinase C activatorQ74324525
ATP-sensitive K+ channel openers prevent Ca2+ overload in rat cardiac mitochondriaQ78157357
MitoK(ATP)-dependent changes in mitochondrial volume and in complex II activity during ischemic and pharmacological preconditioning of Langendorff-perfused rat heartQ79217801
The mechanism by which the mitochondrial ATP-sensitive K+ channel opening and H2O2 inhibit the mitochondrial permeability transitionQ83832665
Myocardial ischemic contracture. Metabolites affect rigor tension development and stiffnessQ93874555
P433issue1
P304page(s)270-276
P577publication date2009-05-04
P1433published inJournal of Molecular and Cellular CardiologyQ2061932
P1476titlePreconditioning with diazoxide prevents reoxygenation-induced rigor-type hypercontracture.
P478volume48

Reverse relations

cites work (P2860)
Q37777273Mitochondrial reprogramming through cardiac oxygen sensors in ischaemic heart disease
Q34631989Remote ischemic preconditioning of cardiomyocytes inhibits the mitochondrial permeability transition pore independently of reduced calcium-loading or sarcKATP channel activation
Q51318851Role of mitochondrial permeability transition pore and mitochondrial ATP-sensitive potassium channels in the protective effects of ischemic preconditioning in isolated hearts from fed and fasted rats.
Q87424378Skeletal muscle post-conditioning by diazoxide, anti-oxidative and anti-apoptotic mechanisms
Q37895248Use of nicorandil in cardiovascular disease and its optimization

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