Tranilast prevents the progression of experimental diabetic nephropathy through suppression of enhanced extracellular matrix gene expression.

scientific article published on 27 April 2005

Tranilast prevents the progression of experimental diabetic nephropathy through suppression of enhanced extracellular matrix gene expression. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1124/JPET.105.084772
P698PubMed publication ID15857946
P5875ResearchGate publication ID36437654

P50authorHitoshi AndoQ60313852
P2093author name stringTsuguhito Ota
Shuichi Kaneko
Toshinari Takamura
Hiroshi Akahori
Muneyoshi Torita
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectdiabetic nephropathyQ1129105
P304page(s)514-521
P577publication date2005-04-27
P1433published inJournal of Pharmacology and Experimental TherapeuticsQ1500272
P1476titleTranilast prevents the progression of experimental diabetic nephropathy through suppression of enhanced extracellular matrix gene expression
P478volume314

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cites work (P2860)
Q37887401Antifibrotic treatment and other new strategies for improving renal outcomes
Q37964276New options and perspectives for proteinuria management after kidney transplantation.
Q36921222Pathobiology of transforming growth factor beta in cancer, fibrosis and immunologic disease, and therapeutic considerations
Q26864133Present and future in the treatment of diabetic kidney disease
Q35763771Proregenerative Microenvironment Triggered by Donor Mesenchymal Stem Cells Preserves Renal Function and Structure in Mice with Severe Diabetes Mellitus.
Q38764267TXNIP in Metabolic Regulation: Physiological Role and Therapeutic Outlook.
Q46470767Tranilast ameliorates impaired hepatic functions in Schistosoma mansoni-infected mice
Q45559795Tranilast modulates fibrosis, epithelial-mesenchymal transition and peritubular capillary injury in unilateral ureteral obstruction rats
Q39969437Tranilast, an antifibrogenic agent, ameliorates a dietary rat model of nonalcoholic steatohepatitis

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