Cardioprotection--time to take into account clinical complexity: the case of antiplatelet agents : editorial to "two classes of anti-platelet drugs reduce anatomical infarct size in monkey hearts" by Xi-Ming Yang et al.

scientific article published in April 2013

Cardioprotection--time to take into account clinical complexity: the case of antiplatelet agents : editorial to "two classes of anti-platelet drugs reduce anatomical infarct size in monkey hearts" by Xi-Ming Yang et al. is …
instance of (P31):
scholarly articleQ13442814
editorialQ871232

External links are
P356DOI10.1007/S10557-013-6443-3
P698PubMed publication ID23354515
P5875ResearchGate publication ID235376250

P50authorJean-Claude TardifQ16244673
P2093author name stringFrançois Roubille
P2860cites workInhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioningQ28187266
Myocardial reperfusion injury.Q34687124
Loss of DPP4 activity is related to a prothrombogenic status of endothelial cells: implications for the coronary microvasculature of myocardial infarction patientsQ35654597
Platelet P2Y₁₂ blockers confer direct postconditioning-like protection in reperfused rabbit heartsQ36763104
Two classes of anti-platelet drugs reduce anatomical infarct size in monkey heartsQ37277256
Circulating microparticles: new insights into the biochemical basis of microparticle release and activity.Q37892456
Clopidogrel affects leukocyte dependent platelet aggregation by P2Y12 expressing leukocytesQ43234743
Activated platelets contribute importantly to myocardial reperfusion injuryQ44914023
Postconditioning inhibits mPTP opening independent of oxidative phosphorylation and membrane potential.Q46103192
Effect of cyclosporine on reperfusion injury in acute myocardial infarctionQ46453592
Postconditioning the human heartQ46722557
Long-term benefit of postconditioningQ46758924
Myocardial expression of a dominant-negative form of Daxx decreases infarct size and attenuates apoptosis in an in vivo mouse model of ischemia/reperfusion injury.Q46891649
Post-conditioning reduces infarct size and edema in patients with ST-segment elevation myocardial infarctionQ47305177
The dimeric platelet collagen receptor GPVI-Fc reduces platelet adhesion to activated endothelium and preserves myocardial function after transient ischemia in mice.Q51757699
Down-regulation of the transcription factor ZAC1 upon pre- and postconditioning protects against I/R injury in the mouse myocardium.Q53203960
ACC/AHA Guidelines for the Management of Patients With ST-Elevation Myocardial Infarction—Executive SummaryQ56986933
P433issue2
P304page(s)105-107
P577publication date2013-04-01
P1433published inCardiovascular Drugs and TherapyQ2473050
P1476titleCardioprotection--time to take into account clinical complexity: the case of antiplatelet agents : editorial to "two classes of anti-platelet drugs reduce anatomical infarct size in monkey hearts" by Xi-Ming Yang et al.
P478volume27

Reverse relations

cites work (P2860)
Q51106338No post-conditioning in the human heart with thrombolysis in myocardial infarction flow 2-3 on admission.
Q54296892Reduction of no-reflow and reperfusion injury with the synthetic 17β-aminoestrogen compound Prolame is associated with PI3K/Akt/eNOS signaling cascade.
Q38108537What is the role of erythropoietin in acute myocardial infarct? Bridging the gap between experimental models and clinical trials

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