Functional consequences of the SHP-1 defect in motheaten viable mice: role of NF-kappa B.

scientific article published in April 1998

Functional consequences of the SHP-1 defect in motheaten viable mice: role of NF-kappa B. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1006/CIMM.1998.1272
P698PubMed publication ID9636682

P2093author name stringKhaled AR
Sobel ES
Schiffenbauer J
Butfiloski EJ
P433issue1
P921main subjectNF-κBQ411114
P304page(s)49-58
P577publication date1998-04-01
P1433published inCellular ImmunologyQ2597167
P1476titleFunctional consequences of the SHP-1 defect in motheaten viable mice: role of NF-kappa B.
P478volume185

Reverse relations

cites work (P2860)
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Q46881381Autophosphorylation docking site Tyr-867 in Mer receptor tyrosine kinase allows for dissociation of multiple signaling pathways for phagocytosis of apoptotic cells and down-modulation of lipopolysaccharide-inducible NF-kappaB transcriptional activat
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Q33905233Central neuroinvasion and demyelination by inflammatory macrophages after peripheral virus infection is controlled by SHP-1.
Q92301856Enterohemorrhagic Escherichia coli Tir inhibits TAK1 activation and mediates immune evasion
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Q33781265Leishmania interferes with host cell signaling to devise a survival strategy
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