scholarly article | Q13442814 |
P356 | DOI | 10.1080/21541248.2017.1333188 |
P8608 | Fatcat ID | release_kbql34f3nfhoxbsszlmf53qkg4 |
P932 | PMC publication ID | 6748372 |
P698 | PubMed publication ID | 28692342 |
P50 | author | John P O'Bryan | Q84582721 |
Russell Spencer-Smith | Q88483734 | ||
P2093 | author name string | Akiko Koide | |
Shohei Koide | |||
Lie Li | |||
Sheela Prasad | |||
P2860 | cites work | Ras nanoclusters: Versatile lipid-based signaling platforms | Q38252413 |
The Ras-Membrane Interface: Isoform-specific Differences in The Catalytic Domain | Q38310579 | ||
Affinity profiling of the cellular kinome for the nucleotide cofactors ATP, ADP, and GTP. | Q39231834 | ||
The Ras G Domain Lacks the Intrinsic Propensity to Form Dimers | Q40583259 | ||
GTP-dependent segregation of H-ras from lipid rafts is required for biological activity | Q40815505 | ||
A polybasic domain or palmitoylation is required in addition to the CAAX motif to localize p21ras to the plasma membrane | Q41718569 | ||
N-Ras forms dimers at POPC membranes | Q42425935 | ||
Functional specificity of ras isoforms: so similar but so different | Q42784309 | ||
Dimerization opens new avenues into Ras signaling research | Q43490714 | ||
Nonradioactive determination of Ras-GTP levels using activated ras interaction assay | Q43635841 | ||
Interaction of GTP derivatives with cellular and oncogenic ras-p21 proteins | Q44953937 | ||
NMR characterization of full-length farnesylated and non-farnesylated H-Ras and its implications for Raf activation | Q45112645 | ||
US National Cancer Institute's new Ras project targets an old foe. | Q46929162 | ||
Ras isoforms vary in their ability to activate Raf-1 and phosphoinositide 3-kinase | Q77195536 | ||
A novel switch region regulates H-ras membrane orientation and signal output | Q24313439 | ||
Small-molecule ligands bind to a distinct pocket in Ras and inhibit SOS-mediated nucleotide exchange activity | Q27678097 | ||
Discovery of Small Molecules that Bind to K-Ras and Inhibit Sos-Mediated Activation | Q27678948 | ||
K-Ras(G12C) inhibitors allosterically control GTP affinity and effector interactions | Q27680654 | ||
Approach for targeting Ras with small molecules that activate SOS-mediated nucleotide exchange | Q27681830 | ||
Targeting RAS signalling pathways in cancer therapy | Q28201363 | ||
The Ras superfamily at a glance | Q29617344 | ||
A comprehensive survey of Ras mutations in cancer | Q30416821 | ||
H-Ras forms dimers on membrane surfaces via a protein-protein interface. | Q30573159 | ||
Ras membrane orientation and nanodomain localization generate isoform diversity | Q33667116 | ||
Inhibition of purified p21ras farnesyl:protein transferase by Cys-AAX tetrapeptides | Q34219356 | ||
K- and N-Ras are geranylgeranylated in cells treated with farnesyl protein transferase inhibitors | Q34427029 | ||
Inhibition of Ras-Effector Interaction by Cyclic Peptides | Q34668051 | ||
Direct inhibition of oncogenic KRAS by hydrocarbon-stapled SOS1 helices | Q35089991 | ||
Drugging the undruggable RAS: Mission possible? | Q35163434 | ||
Three separable domains regulate GTP-dependent association of H-ras with the plasma membrane | Q35663831 | ||
Oncogenic and RASopathy-associated K-RAS mutations relieve membrane-dependent occlusion of the effector-binding site | Q35669207 | ||
Direct Inhibitors of Ras-Effector Protein Interactions | Q35794271 | ||
Ras-GTP dimers activate the Mitogen-Activated Protein Kinase (MAPK) pathway | Q35818835 | ||
Specific cancer-associated mutations in the switch III region of Ras increase tumorigenicity by nanocluster augmentation. | Q36038664 | ||
Allele-specific inhibitors inactivate mutant KRAS G12C by a trapping mechanism | Q37113394 | ||
Structural basis of recognition of farnesylated and methylated KRAS4b by PDEδ. | Q37398069 | ||
Inhibition of RAS function through targeting an allosteric regulatory site. | Q37540897 | ||
Dragging ras back in the ring | Q38197825 | ||
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 5 | |
P304 | page(s) | 378-387 | |
P577 | publication date | 2017-12-31 | |
P1433 | published in | Small GTPases (journal) | Q15709529 |
P1476 | title | Targeting the α4-α5 interface of RAS results in multiple levels of inhibition | |
P478 | volume | 10 |
Q49721819 | Direct inhibition of RAS: Quest for the Holy Grail? |
Q64084217 | Drugging the Small GTPase Pathways in Cancer Treatment: Promises and Challenges |
Q47256433 | Oncogenic Ras Isoforms Signaling Specificity at the Membrane |
Q90681757 | Targeting the α4-α5 dimerization interface of K-RAS inhibits tumor formation in vivo |
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