| P50 | author | David N. Cooper | Q30503192 |
| | Jian-Min Chen | Q53266161 |
| P2093 | author name string | Zhao-Shen Li | |
| | Claude Férec | |
| | Zhuan Liao | |
| | Arnaud Boulling | |
| | Wen-Bin Zou | |
| | Emmanuelle Masson | |
| P2860 | cites work | Functional effects of 13 rare PRSS1 variants presumed to cause chronic pancreatitis. | Q36927073 |
| | Minigene analysis of intronic variants in common SPINK1 haplotypes associated with chronic pancreatitis. | Q37181595 |
| | Comprehensive functional analysis of chymotrypsin C (CTRC) variants reveals distinct loss-of-function mechanisms associated with pancreatitis risk | Q40906883 |
| | Exonic variants affecting pre-mRNA splicing add to genetic burden in chronic pancreatitis. | Q42405322 |
| | [-215G>A; IVS3+2T>C] mutation in the SPINK1 gene causes exon 3 skipping and loss of the trypsin binding site | Q42844578 |
| | Identification of a functional PRSS1 promoter variant in linkage disequilibrium with the chronic pancreatitis-protecting rs10273639. | Q48149884 |
| P433 | issue | 5 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 884-886 | |
| P577 | publication date | 2015-12-30 | |
| P1433 | published in | Gut | Q5621669 |
| P1476 | title | Clarifying the clinical relevance of SPINK1 intronic variants in chronic pancreatitis | |
| P478 | volume | 65 | |