| scholarly article | Q13442814 |
| P50 | author | Heiko Witt | Q90796906 |
| P2093 | author name string | András Szabó | |
| Jiayi Zhou | |||
| Miklós Sahin-Tóth | |||
| Giriraj Ratan Chandak | |||
| Steven Keiles | |||
| Sebastian Beer | |||
| P2860 | cites work | Comprehensive screening of chymotrypsin C (CTRC) gene in tropical calcific pancreatitis identifies novel variants | Q39334642 |
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| Chymotrypsin C (CTRC) variants that diminish activity or secretion are associated with chronic pancreatitis | Q24645261 | ||
| Hereditary pancreatitis caused by mutation-induced misfolding of human cationic trypsinogen: a novel disease mechanism | Q24650338 | ||
| Chymotrypsin C (caldecrin) promotes degradation of human cationic trypsin: identity with Rinderknecht's enzyme Y | Q24675338 | ||
| Chymotrypsin C mutations in chronic pancreatitis | Q27693296 | ||
| Pancreatitis-associated chymotrypsinogen C (CTRC) mutant elicits endoplasmic reticulum stress in pancreatic acinar cells | Q33764920 | ||
| High affinity small protein inhibitors of human chymotrypsin C (CTRC) selected by phage display reveal unusual preference for P4' acidic residues | Q33880616 | ||
| Association of rare chymotrypsinogen C (CTRC) gene variations in patients with idiopathic chronic pancreatitis | Q34732835 | ||
| Tropical calcific pancreatitis and its association with CTRC and SPINK1 (p.N34S) variants | Q34978483 | ||
| Association of novel chymotrypsin C gene variations and haplotypes in patients with chronic pancreatitis in Chinese in Taiwan. | Q34978689 | ||
| Increased activation of hereditary pancreatitis-associated human cationic trypsinogen mutants in presence of chymotrypsin C | Q36016998 | ||
| Glucocorticoids increase amylase mRNA levels, secretory organelles, and secretion in pancreatic acinar AR42J cells | Q36212907 | ||
| Chronic pancreatitis: genetics and pathogenesis | Q37488219 | ||
| Genetic aspects of pancreatitis | Q37670869 | ||
| Genetics of pancreatitis: a guide for clinicians | Q37869759 | ||
| Asparagine-linked glycosylation of human chymotrypsin C is required for folding and secretion but not for enzyme activity | Q38963298 | ||
| P433 | issue | 11 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 1616-1624 | |
| P577 | publication date | 2012-09-01 | |
| P1433 | published in | Gut | Q5621669 |
| P1476 | title | Comprehensive functional analysis of chymotrypsin C (CTRC) variants reveals distinct loss-of-function mechanisms associated with pancreatitis risk | |
| P478 | volume | 62 |
| Q42725781 | A Carboxyl Ester Lipase (CEL) Mutant Causes Chronic Pancreatitis by Forming Intracellular Aggregates That Activate Apoptosis |
| Q30000783 | A conservative assessment of the major genetic causes of idiopathic chronic pancreatitis: data from a comprehensive analysis of PRSS1, SPINK1, CTRC and CFTR genes in 253 young French patients |
| Q37725572 | Association of CTRC and SPINK1 gene variants with recurrent hospitalizations for pancreatitis or acute abdominal pain in lipoprotein lipase deficiency |
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| Q86874923 | Characterization of two deletions of the CTRC locus |
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| Q35319462 | Endoplasmic reticulum stress promotes amyloid-beta peptides production in RGC-5 cells |
| Q36927073 | Functional effects of 13 rare PRSS1 variants presumed to cause chronic pancreatitis. |
| Q48247460 | Genetic Polymorphisms: A Novel Perspective on Acute Pancreatitis. |
| Q33843764 | Genetic Risk in Chronic Pancreatitis: The Trypsin-Dependent Pathway |
| Q34667122 | Genetic and phenotypic heterogeneity in tropical calcific pancreatitis |
| Q36933359 | Genetic risk factors for pancreatic disorders |
| Q39398523 | Genetic risk in chronic pancreatitis: the misfolding-dependent pathway |
| Q26739600 | Hereditary pancreatitis: current perspectives |
| Q57456156 | INternational Study Group of Pediatric Pancreatitis: In Search for a CuRE Cohort Study: Design and Rationale for INSPPIRE 2 From the Consortium for the Study of Chronic Pancreatitis, Diabetes, and Pancreatic Cancer |
| Q89517074 | Inactivation of mesotrypsin by chymotrypsin C prevents trypsin inhibitor degradation |
| Q37638790 | Insights into the genetic risk factors for the development of pancreatic disease |
| Q27676497 | Long-range Electrostatic Complementarity Governs Substrate Recognition by Human Chymotrypsin C, a Key Regulator of Digestive Enzyme Activation |
| Q97548636 | Loss of chymotrypsin-like protease (CTRL) alters intrapancreatic protease activation but not pancreatitis severity in mice |
| Q35837145 | Mesotrypsin Signature Mutation in a Chymotrypsin C (CTRC) Variant Associated with Chronic Pancreatitis. |
| Q52579674 | Mutations in the pancreatic secretory enzymes CPA1 and CPB1 are associated with pancreatic cancer. |
| Q38900762 | New insights into the pathways initiating and driving pancreatitis. |
| Q48153726 | No significant enrichment of rare functionally defective CPA1 variants in a large Chinese idiopathic chronic pancreatitis cohort |
| Q37231509 | PRSS1, SPINK1, CFTR, and CTRC Pathogenic Variants in Korean Patients With Idiopathic Pancreatitis |
| Q38548458 | Paediatric pancreatitis |
| Q36775826 | Pathogenic cellular role of the p.L104P human cationic trypsinogen variant in chronic pancreatitis |
| Q57154534 | Recurrent Acute Pancreatitis: Current Concepts in the Diagnosis and Management |
| Q34400723 | Risk factors for pancreatic cancer: underlying mechanisms and potential targets |
| Q42145402 | Robust autoactivation, chymotrypsin C independence and diminished secretion define a subset of hereditary pancreatitis-associated cationic trypsinogen mutants. |
| Q35565390 | The Common Chymotrypsinogen C (CTRC) Variant G60G (C.180T) Increases Risk of Chronic Pancreatitis But Not Recurrent Acute Pancreatitis in a North American Population. |
| Q38090903 | The role of protein synthesis and digestive enzymes in acinar cell injury |
| Q37540036 | Variants in CPA1 are strongly associated with early onset chronic pancreatitis |
| Q36174524 | Variants in pancreatic carboxypeptidase genes CPA2 and CPB1 are not associated with chronic pancreatitis |
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