Exercise pre-conditioning reduces brain inflammation in stroke via tumor necrosis factor-alpha, extracellular signal-regulated kinase 1/2 and matrix metalloproteinase-9 activity

scientific article published on 13 August 2009

Exercise pre-conditioning reduces brain inflammation in stroke via tumor necrosis factor-alpha, extracellular signal-regulated kinase 1/2 and matrix metalloproteinase-9 activity is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1179/174313209X459101
P698PubMed publication ID19682410

P2093author name stringRohit Patel
Nathan Zwagerman
Shane Sprague
Yuchuan Ding
Miao Guo
Qin Lai
Alecia Curry
Brandon Liebelt
David Jimenez
Frank X Cao
P2860cites workReversible middle cerebral artery occlusion without craniectomy in ratsQ28268048
Inhibition of matrix metalloproteinase-9 prevents neutrophilic inflammation in ventilator-induced lung injuryQ28565915
Peripheral thermal injury causes blood-brain barrier dysfunction and matrix metalloproteinase (MMP) expression in ratQ28582311
Alteration of MAP kinase pathways after transient forebrain ischemiaQ30887275
Matrix metalloproteinases in neuroinflammationQ34803809
The many faces of tumor necrosis factor in strokeQ35011702
Role of extracellular signal regulated kinases 1 and 2 in neuronal survivalQ35778135
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Role of matrix metalloproteinases in intestinal inflammationQ36462632
Requirement for nitric oxide activation of p21(ras)/extracellular regulated kinase in neuronal ischemic preconditioningQ37156312
The involvement of the apoptosis-modulating proteins ERK 1/2, Bcl-xL and Bax in the resolution of acute inflammation in vivoQ38404632
Inhibition of extracellular signal regulated kinase (ERK) leads to apoptosis inducing factor (AIF) mediated apoptosis in epithelial breast cancer cells: the lack of effect of ERK in p53 mediated copper induced apoptosisQ40425110
Activation of extracellular signal-regulated kinases ERK1 and ERK2 induces Bcl-xL up-regulation via inhibition of caspase activities in erythropoietin signalingQ40661974
Leukocyte-derived matrix metalloproteinase-9 mediates blood-brain barrier breakdown and is proinflammatory after transient focal cerebral ischemiaQ42475527
Identification of a novel role for sphingolipid signaling in TNF alpha and ischemic preconditioning mediated cardioprotectionQ44022447
Pharmacological preconditioning with tumor necrosis factor-alpha activates signal transducer and activator of transcription-3 at reperfusion without involving classic prosurvival kinases (Akt and extracellular signal-regulated kinase).Q46847925
Hypoxia-induced ischemic tolerance in neonatal rat brain involves enhanced ERK1/2 signalingQ47927699
Mechanisms of stroke protection by physical activityQ48155955
Changes in protein tyrosine phosphorylation in the rat brain after cerebral ischemia in a model of ischemic toleranceQ48278407
Synaptic plasticity in thalamic nuclei enhanced by motor skill training in rat with transient middle cerebral artery occlusionQ48365428
Functional improvement after motor training is correlated with synaptic plasticity in rat thalamusQ48419579
Ischemic but not mechanical preconditioning attenuates ischemia/reperfusion induced myocardial apoptosis in anaesthetized rabbits: the role of Bcl-2 family proteins and ERK1/2.Q48651631
Effects of ischemic preconditioning on blood-brain barrier permeability and MMP-9 expression of ischemic brainQ48658222
Protective effects of treadmill training on infarction in ratsQ48698487
TNF-alpha pretreatment induces protective effects against focal cerebral ischemia in miceQ48714135
Exercise pre-conditioning reduces brain damage in ischemic rats that may be associated with regional angiogenesis and cellular overexpression of neurotrophin.Q51022529
P433issue7
P921main subjectencephalitisQ199615
P304page(s)756-762
P577publication date2009-08-13
P1433published inNeurological ResearchQ15765622
P1476titleExercise pre-conditioning reduces brain inflammation in stroke via tumor necrosis factor-alpha, extracellular signal-regulated kinase 1/2 and matrix metalloproteinase-9 activity
P478volume32

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