| P50 | author | Menno C van Zelm | Q37834763 |
| P2093 | author name string | King H Lam | |
| | P Martin van Hagen | |
| | Jasper H Kappen | |
| | Sophinus J W Bartol | |
| | Jan A M van Laar | |
| | Tim B van der Houwen | |
| | Wilhemina M C Timmermans | |
| P2860 | cites work | Anti-TNF treatment blocks the induction of T cell-dependent humoral responses. | Q50938596 |
| | A new CD21low B cell population in the peripheral blood of patients with SLE. | Q51997566 |
| | Problems in using statistical analysis of replacement and silent mutations in antibody genes for determining antigen-driven affinity selection. | Q52992155 |
| | Anticardiolipin antibodies in Adamantiades-Behçet's disease. | Q53023052 |
| | Human immunoglobulin M memory B cells controlling Streptococcus pneumoniae infections are generated in the spleen | Q24673510 |
| | IMGT, the international ImMunoGeneTics information system. | Q33382007 |
| | Anti-TNF agents for Behçet's disease: analysis of published data on 369 patients | Q33774951 |
| | Quantifying selection in high-throughput Immunoglobulin sequencing data sets | Q34284964 |
| | Marginal zone B cells: virtues of innate-like antibody-producing lymphocytes | Q34324243 |
| | Detecting selection in immunoglobulin sequences | Q35075665 |
| | Serum levels of TNF-alpha, sIL-2R, IL-6, and IL-8 are increased and associated with elevated lipid peroxidation in patients with Behçet's disease | Q35609440 |
| | Complement receptor 2/CD21- human naive B cells contain mostly autoreactive unresponsive clones | Q36026609 |
| | A Reassessment of IgM Memory Subsets in Humans | Q36125626 |
| | Current concepts in the etiology and treatment of Behçet disease | Q36168752 |
| | (99m)Tc-labeled rituximab for imaging B lymphocyte infiltration in inflammatory autoimmune disease patients. | Q36236880 |
| | Replication history of B lymphocytes reveals homeostatic proliferation and extensive antigen-induced B cell expansion | Q36267196 |
| | Efficient generation of monoclonal antibodies from single human B cells by single cell RT-PCR and expression vector cloning | Q36459907 |
| | Alterations in peripheral blood memory B cells in patients with active rheumatoid arthritis are dependent on the action of tumour necrosis factor | Q37270827 |
| | Human immunoglobulin classes and subclasses show variability in VDJ gene mutation levels. | Q40199265 |
| | Human memory B cells originate from three distinct germinal center-dependent and -independent maturation pathways | Q42115840 |
| | Histopathology of central nervous system lesions in Behçet's disease | Q42521064 |
| | Adalimumab: a new modality for Behçet's disease? | Q42844975 |
| | Rituximab in intractable ocular lesions of Behcet's disease; randomized single-blind control study (pilot study). | Q42939553 |
| | Perigranuloma localization and abnormal maturation of B cells: emerging key players in sarcoidosis? | Q46384064 |
| | The role of TLR2 and 4 in Behçet's disease pathogenesis. | Q47792238 |
| | Deficiency of somatic hypermutation of the antibody light chain is associated with increased frequency of severe respiratory tract infection in common variable immunodeficiency | Q47926953 |
| | Strong intrinsic biases towards mutation and conservation of bases in human IgVH genes during somatic hypermutation prevent statistical analysis of antigen selection | Q48006794 |
| | Anti-Tumor Necrosis Factor Therapy Restores Peripheral Blood B-cell Subsets and CD40 Expression in Inflammatory Bowel Diseases. | Q50708114 |
| | Depletion of immunoglobulin M memory B cells is associated with splenic hypofunction in inflammatory bowel disease. | Q50758825 |
| | IgA subclass switch recombination in human mucosal and systemic immune compartments. | Q50880804 |
| P433 | issue | 1 | |
| P921 | main subject | TNF | Q18032037 |
| | Behçet's disease | Q911427 |
| | memory B cell | Q1195394 |
| P304 | page(s) | 134-144 | |
| P577 | publication date | 2016-10-15 | |
| P1433 | published in | Rheumatology | Q7320492 |
| P1476 | title | Chronic signs of memory B cell activation in patients with Behçet's disease are partially restored by anti-tumour necrosis factor treatment. | |
| P478 | volume | 56 | |