Contribution of PAR-1, PAR-4 and GPIbalpha in intracellular signaling leading to the cleavage of the beta3 cytoplasmic domain during thrombin-induced platelet aggregation.

scientific article published in April 2004

Contribution of PAR-1, PAR-4 and GPIbalpha in intracellular signaling leading to the cleavage of the beta3 cytoplasmic domain during thrombin-induced platelet aggregation. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1160/TH03-06-0391
P698PubMed publication ID15045135

P2093author name stringBeat Steiner
Christophe Dubois
Sylvie C Meyer Reigner
P433issue4
P921main subjectplatelet aggregationQ14913634
P304page(s)733-742
P577publication date2004-04-01
P1433published inThrombosis and HaemostasisQ15724413
P1476titleContribution of PAR-1, PAR-4 and GPIbalpha in intracellular signaling leading to the cleavage of the beta3 cytoplasmic domain during thrombin-induced platelet aggregation.
P478volume91

Reverse relations

cites work (P2860)
Q36783478Calpain-mediated regulation of platelet signaling pathways
Q37331535Cancer cell-derived microparticles bearing P-selectin glycoprotein ligand 1 accelerate thrombus formation in vivo.
Q27342751Disabled-2 is required for efficient hemostasis and platelet activation by thrombin in mice
Q37788205Thrombin receptor antagonists for the treatment of atherothrombosis: therapeutic potential of vorapaxar and E-5555.

Search more.