Ponatinib circumvents all types of imatinib resistance in chronic myelogenous leukemia cell lines.

scientific article published on 10 May 2013

Ponatinib circumvents all types of imatinib resistance in chronic myelogenous leukemia cell lines. is …
instance of (P31):
editorialQ871232
scholarly articleQ13442814

External links are
P356DOI10.4161/CC.24982
P698PubMed publication ID23673326
P5875ResearchGate publication ID236836431

P50authorArnaud JacquelQ39868748
Patrick AubergerQ56997684
P2093author name stringGuillaume Robert
Maeva Dufies
Ophelie Cassuto
P2860cites workAP24534, a pan-BCR-ABL inhibitor for chronic myeloid leukemia, potently inhibits the T315I mutant and overcomes mutation-based resistanceQ24648017
Ponatinib in refractory Philadelphia chromosome-positive leukemiasQ27851967
Nilotinib versus imatinib for newly diagnosed chronic myeloid leukemiaQ34620246
Mechanisms of BCR-ABL in the pathogenesis of chronic myelogenous leukaemiaQ36047171
Quantitative phosphoproteomics revealed interplay between Syk and Lyn in the resistance to nilotinib in chronic myeloid leukemia cellsQ39512409
Persistent activation of the Fyn/ERK kinase signaling axis mediates imatinib resistance in chronic myelogenous leukemia cells through upregulation of intracellular SPARC.Q39628692
P433issue11
P921main subjectleukemiaQ29496
imatinibQ177094
P1104number of pages2
P304page(s)1645-1646
P577publication date2013-05-10
P1433published inCell CycleQ1254166
P1476titlePonatinib circumvents all types of imatinib resistance in chronic myelogenous leukemia cell lines
P478volume12

Reverse relations

cites work (P2860)
Q39106999Combined effects of PI3K and SRC kinase inhibitors with imatinib on intracellular calcium levels, autophagy, and apoptosis in CML-PBL cells.
Q39062384Dovitinib (CHIR258, TKI258): structure, development and preclinical and clinical activity
Q34781322Genomic quantitative real-time PCR proves residual disease positivity in more than 30% samples with negative mRNA-based qRT-PCR in Chronic Myeloid Leukemia
Q33576849Latest progress in tyrosine kinase inhibitors

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