Metabolic phenotypes of obesity influence triglyceride and inflammation homoeostasis. (scientific article published in November 2014)

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Obesity is associated with macrophage accumulation in adipose tissue

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Triglyceride-rich lipoproteins and high-density lipoprotein cholesterol in patients at high risk of cardiovascular disease: evidence and guidance for management

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Metabolically healthy obesity and risk of all-cause and cardiovascular disease mortality

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Challenging homeostasis to define biomarkers for nutrition related health

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Weight reduction, but not a moderate intake of fish oil, lowers concentrations of inflammatory markers and PAI-1 antigen in obese men during the fasting and postprandial state

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The obese without cardiometabolic risk factor clustering and the normal weight with cardiometabolic risk factor clustering: prevalence and correlates of 2 phenotypes among the US population (NHANES 1999-2004).

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Q47251710

Dietary fat modifies the postprandial inflammatory state in subjects with metabolic syndrome: the LIPGENE study.

Q51355423

Assessment and clinical relevance of non-fasting and postprandial triglycerides: an expert panel statement.

Q51379451

Diagnostic value of postprandial triglyceride testing in healthy subjects: a meta-analysis.

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Clinical relevance of non-fasting and postprandial hypertriglyceridemia and remnant cholesterol.

Q51379466

Activation of leukocytes by postprandial lipemia in healthy volunteers

Q57790428

P433

issue

P921

main subject

triglyceride

Q186319

P304

page(s)

1053-1064

P577

publication date

2014-11-01

P1433

published in

European Journal of Clinical Investigation

Q15745208

P1476

title

Metabolic phenotypes of obesity influence triglyceride and inflammation homoeostasis.

P478

volume

Reverse relations

cites work (P2860)

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