Keith A Youker

researcher

Keith A Youker is …
instance of (P31):
humanQ5

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P496ORCID iD0000-0003-2535-7973

P69educated atBaylor College of MedicineQ2892284
Oakland UniversityQ782116
P108employerBaylor College of MedicineQ2892284
P735given nameKeithQ1159033
KeithQ1159033
P106occupationresearcherQ1650915

Reverse relations

author (P50)
Q34204473Adherence of neutrophils to canine cardiac myocytes in vitro is dependent on intercellular adhesion molecule-1
Q28348531Angiotensin II blockade reverses myocardial fibrosis in a transgenic mouse model of human hypertrophic cardiomyopathy
Q51717471Apicoaortic conduit in a patient with severe hemolysis after three aortic valve replacements.
Q77932873Cardiac myocytes produce interleukin-6 in culture and in viable border zone of reperfused infarctions
Q72133140Cardiolipin-protein complexes and initiation of complement activation after coronary artery occlusion
Q84187536Cellular evidence of reverse cardiac remodeling induced by cardiac resynchronization therapy
Q73047639Complement C5a, TGF-beta 1, and MCP-1, in sequence, induce migration of monocytes into ischemic canine myocardium within the first one to five hours after reperfusion
Q77548701Coronary microembolization: the role of TNF-alpha in contractile dysfunction
Q78174120Cytokines and the microcirculation in ischemia and reperfusion
Q63984849Decreased expression of tumor necrosis factor-alpha in failing human myocardium after mechanical circulatory support : A potential mechanism for cardiac recovery
Q73580355Decreased left ventricular ejection fraction in transgenic mice expressing mutant cardiac troponin T-Q(92), responsible for human hypertrophic cardiomyopathy
Q51720898Degree of cardiac fibrosis and hypertrophy at time of implantation predicts myocardial improvement during left ventricular assist device support.
Q51517191Fluorescence imaging microscopy of cellular markers in ischemic vs non-ischemic cardiomyopathy after left ventricular unloading.
Q80011865Free fatty acids inhibit insulin signaling-stimulated endothelial nitric oxide synthase activation through upregulating PTEN or inhibiting Akt kinase
Q37716157Freshly isolated mitochondria from failing human hearts exhibit preserved respiratory function
Q83823177Heart failure research: translating basic science into therapies
Q73532277Hydrogen peroxide induces LFA-1-dependent neutrophil adherence to cardiac myocytes
Q44081028Impaired long-chain fatty acid oxidation and contractile dysfunction in the obese Zucker rat heart.
Q36845491Induction of monocyte chemoattractant protein-1 in the small veins of the ischemic and reperfused canine myocardium.
Q73696394Inhibitory cardiac transcription factor, SRF-N, is generated by caspase 3 cleavage in human heart failure and attenuated by ventricular unloading
Q47744332Interaction between isolated human myocardial mast cells and cultured fibroblasts
Q34193068Interleukin-8 gene induction in the myocardium after ischemia and reperfusion in vivo.
Q59192157Is inflammation good for the ischemic heart perspectives beyond the ordinary
Q80554391Localizing and quantifying ablation lesions in the left ventricle by myocardial contrast echocardiography
Q47789573Mast cell burden and reticulin fibrosis in the myeloproliferative neoplasms: a computer-assisted image analysis study
Q51028605Mast cell-derived cathepsin g: a possible role in the adverse remodeling of the failing human heart.
Q51824360Mechanical unloading of the failing human heart fails to activate the protein kinase B/Akt/glycogen synthase kinase-3beta survival pathway.
Q51122579Mechanical unloading of the heart activates the calpain system.
Q72685498Molecular evidence for a border zone vulnerable to inflammatory reperfusion injury
Q72680145Molecular evidence for induction of intracellular adhesion molecule-1 in the viable border zone associated with ischemia-reperfusion injury of the dog heart
Q84147537Molecular, cellular, and functional characterization of myocardial regions in hypertrophic cardiomyopathy
Q81497332Myocardial contrast echocardiography of radiofrequency ablation lesions
Q34250655Neutrophil adherence to isolated adult canine myocytes. Evidence for a CD18-dependent mechanism.
Q35598054Neutrophil adherence to isolated adult cardiac myocytes. Induction by cardiac lymph collected during ischemia and reperfusion
Q35606652Neutrophil induced oxidative injury of cardiac myocytes. A compartmented system requiring CD11b/CD18-ICAM-1 adherence
Q47687955Phagocytes in ischemia injury.
Q51428172Quantitative changes in mast cell populations after left ventricular assist device implantation.
Q37358790Reciprocal regulation of myocardial microRNAs and messenger RNA in human cardiomyopathy and reversal of the microRNA signature by biomechanical support.
Q63984831Regression of fibrosis and hypertrophy in failing myocardium following mechanical circulatory support
Q77142325Resident cardiac mast cells degranulate and release preformed TNF-alpha, initiating the cytokine cascade in experimental canine myocardial ischemia/reperfusion
Q63984794Reversal of secondary pulmonary hypertension by axial and pulsatile mechanical circulatory support
Q71574771Role of early reperfusion in the induction of adhesion molecules and cytokines in previously ischemic myocardium
Q51616293Role of mast cells and their mediators in failing myocardium under mechanical ventricular support.
Q24298646Stanniocalcin-1 is a naturally occurring L-channel inhibitor in cardiomyocytes: relevance to human heart failure
Q34983035Telomere attrition and Chk2 activation in human heart failure
Q63984837The implications for cardiac recovery of left ventricular assist device support on myocardial collagen content
Q38091933The role of B-cells in heart failure
Q37170728The role of mast cells after solid organ transplantation
Q46223821The use of continuous milrinone therapy as bridge to transplant is safe in patients with short waiting times.
Q85097540Therapeutic plasma exchange a potential strategy for patients with advanced heart failure
Q73458506Time-dependent loss of Mac-1 from infiltrating neutrophils in the reperfused myocardium
Q67902942Unsaturated aminophospholipids are preferentially retained by the fast skeletal muscle CaATPase during detergent solubilization. Evidence for a specific association between aminophospholipids and the calcium pump protein

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