NO may prompt calcium leakage in dystrophic muscle.

scientific article published in March 2009

NO may prompt calcium leakage in dystrophic muscle. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1038/NM0309-243
P698PubMed publication ID19265820

P2093author name stringJames G Tidball
S Armando Villalta
P2860cites workNuclear factor kappa-B blockade reduces skeletal muscle degeneration and enhances muscle function in Mdx miceQ46892558
Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscleQ24308714
Duchenne dystrophy: Electron microscopic findings pointing to a basic or early abnormality in the plasma membrane of the muscle fiberQ30951467
Genomic and nongenomic effects of glucocorticoidsQ34819002
Dystrophin protects the sarcolemma from stresses developed during muscle contractionQ36259954
A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx miceQ36294012
Increased calcium influx in dystrophic muscleQ36530501
Shifts in macrophage phenotypes and macrophage competition for arginine metabolism affect the severity of muscle pathology in muscular dystrophyQ37090043
Complex regulation of human inducible nitric oxide synthase gene transcription by Stat 1 and NF-kappa B.Q37108918
Effects of stretch-activated channel blockers on [Ca2+]i and muscle damage in the mdx mouse.Q45140549
Reduced muscle necrosis and long-term benefits in dystrophic mdx mice after cV1q (blockade of TNF) treatmentQ46799394
P433issue3
P407language of work or nameEnglishQ1860
P304page(s)243-244
P577publication date2009-03-01
P1433published inNature MedicineQ1633234
P1476titleNO may prompt calcium leakage in dystrophic muscle.
P478volume15