scholarly article | Q13442814 |
P50 | author | Steven R. Reiken | Q114724674 |
P2093 | author name string | Alain Lacampagne | |
Andrew R Marks | |||
Marco Mongillo | |||
Stefan Matecki | |||
Xiaoping Liu | |||
Andrew M Bellinger | |||
Christian Carlson | |||
Lisa Rothman | |||
P2860 | cites work | Serum CK, calcium, magnesium, and oxidative phosphorylation in mdx mouse muscular dystrophy | Q67938640 |
Activation of the cardiac calcium release channel (ryanodine receptor) by poly-S-nitrosylation | Q74016180 | ||
Ca2+-independent phospholipase A2 enhances store-operated Ca2+ entry in dystrophic skeletal muscle fibers | Q80153311 | ||
Uncontrolled calcium sparks act as a dystrophic signal for mammalian skeletal muscle | Q81657096 | ||
PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts | Q22254160 | ||
Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy | Q24307833 | ||
Enhancing calstabin binding to ryanodine receptors improves cardiac and skeletal muscle function in heart failure | Q24530067 | ||
Remodeling of ryanodine receptor complex causes "leaky" channels: a molecular mechanism for decreased exercise capacity | Q24649499 | ||
Phosphorylation-dependent regulation of ryanodine receptors: a novel role for leucine/isoleucine zippers | Q24678432 | ||
Rescue of dystrophic muscle through U7 snRNA-mediated exon skipping | Q28291830 | ||
iNOS expression in dystrophinopathies can be reduced by somatic gene transfer of dystrophin or utrophin | Q28344971 | ||
Association of dystrophin-related protein with dystrophin-associated proteins in mdx mouse muscle | Q28510608 | ||
Dystrophin: the protein product of the Duchenne muscular dystrophy locus | Q29618077 | ||
rAAV6-microdystrophin preserves muscle function and extends lifespan in severely dystrophic mice. | Q30602105 | ||
Alteration in calcium handling at the subcellular level in mdx myotubes. | Q31456406 | ||
Cysteine-3635 is responsible for skeletal muscle ryanodine receptor modulation by NO | Q33944345 | ||
Function and genetics of dystrophin and dystrophin-related proteins in muscle | Q34120764 | ||
Pharmacological strategies for muscular dystrophy | Q35128906 | ||
A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice | Q36294012 | ||
PKA phosphorylation activates the calcium release channel (ryanodine receptor) in skeletal muscle: defective regulation in heart failure | Q36325042 | ||
Increased calcium influx in dystrophic muscle | Q36530501 | ||
Sarcolemma-localized nNOS is required to maintain activity after mild exercise | Q36987100 | ||
Neuronal nitric oxide synthase and dystrophin-deficient muscular dystrophy | Q37387484 | ||
Heregulin ameliorates the dystrophic phenotype in mdx mice | Q37533952 | ||
alpha1-syntrophin gene disruption results in the absence of neuronal-type nitric-oxide synthase at the sarcolemma but does not induce muscle degeneration | Q38329382 | ||
Intracellular calcium accumulation in Duchenne dystrophy and other myopathies: a study of 567,000 muscle fibers in 114 biopsies | Q39486051 | ||
Studies of sarcolemmal integrity in myopathic muscle | Q39498832 | ||
Coupled gating between individual skeletal muscle Ca2+ release channels (ryanodine receptors) | Q41018951 | ||
Stabilization of calcium release channel (ryanodine receptor) function by FK506-binding protein | Q41467443 | ||
Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibers | Q41882302 | ||
Overexpression of a calpastatin transgene in mdx muscle reduces dystrophic pathology | Q44159151 | ||
Increased activity of calcium leak channels in myotubes of Duchenne human and mdx mouse origin | Q44177784 | ||
Nitric oxide, NOC-12, and S-nitrosoglutathione modulate the skeletal muscle calcium release channel/ryanodine receptor by different mechanisms. An allosteric function for O2 in S-nitrosylation of the channel | Q44267719 | ||
Duchenne muscular dystrophy: deficiency of dystrophin at the muscle cell surface | Q44350613 | ||
S-glutathionylation decreases Mg2+ inhibition and S-nitrosylation enhances Ca2+ activation of RyR1 channels | Q44550513 | ||
Effects of stretch-activated channel blockers on [Ca2+]i and muscle damage in the mdx mouse. | Q45140549 | ||
Increased protein degradation results from elevated free calcium levels found in muscle from mdx mice | Q46266620 | ||
Effects of S-glutathionylation and S-nitrosylation on calmodulin binding to triads and FKBP12 binding to type 1 calcium release channels. | Q46584340 | ||
Calcium entry through stretch-inactivated ion channels in mdx myotubes | Q46603395 | ||
Calpains are activated in necrotic fibers from mdx dystrophic mice | Q49165175 | ||
Evidence ofmdx mouse skeletal muscle fragility in vivo by eccentric running exercise | Q58109283 | ||
p66ShcAand Oxidative Stress Modulate Myogenic Differentiation and Skeletal Muscle Regeneration after Hind Limb Ischemia | Q62236571 | ||
P433 | issue | 3 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Dystrophin | Q412285 |
Dystrophin, muscular dystrophy | Q14864296 | ||
negative regulation of peptidyl-cysteine S-nitrosylation | Q21115225 | ||
FK506 binding protein 1a | Q21498441 | ||
Ryanodine receptor 1, skeletal muscle | Q21980348 | ||
P304 | page(s) | 325-30 | |
P577 | publication date | 2009-03-01 | |
P1433 | published in | Nature Medicine | Q1633234 |
P1476 | title | Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle | |
P478 | volume | 15 |
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Q37111473 | Dystrophin restoration therapy improves both the reduced excitability and the force drop induced by lengthening contractions in dystrophic mdx skeletal muscle |
Q44882443 | Editorial for "Methods for analysis of nitric oxide signalling by S-nitrosylation". |
Q28535427 | Effect of Calstabin1 depletion on calcium transients and energy utilization in muscle fibers and treatment opportunities with RyR1 stabilizers |
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Q46060420 | Fixing the leak |
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