scholarly article | Q13442814 |
P50 | author | Ernst Niggli | Q52357375 |
P2093 | author name string | Isabelle Vögeli | |
Charlotte Lorin | |||
P2860 | cites work | A capsaicin-receptor homologue with a high threshold for noxious heat | Q22009379 |
Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy | Q24295360 | ||
Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathy | Q24306846 | ||
Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle | Q24308714 | ||
International Union of Basic and Clinical Pharmacology. LXXVI. Current progress in the mammalian TRP ion channel family | Q24623493 | ||
Stretch-activated ion channels: what are they? | Q24630314 | ||
X-linked dilated cardiomyopathy and the dystrophin gene | Q33688402 | ||
TRPV2 is critical for the maintenance of cardiac structure and function in mice | Q33729367 | ||
The anti-influenza drug oseltamivir exhibits low potential to induce pharmacokinetic drug interactions via renal secretion-correlation of in vivo and in vitro studies. | Q33956427 | ||
Ultrastructural and functional alterations of EC coupling elements in mdx cardiomyocytes: an analysis from membrane surface to depth. | Q52638107 | ||
Genetic inhibition of PKA phosphorylation of RyR2 prevents dystrophic cardiomyopathy | Q34059173 | ||
Isolation of candidate cDNAs for portions of the Duchenne muscular dystrophy gene | Q34190227 | ||
Role of nitric oxide in the pathogenesis of muscular dystrophies: a "two hit" hypothesis of the cause of muscle necrosis | Q34465820 | ||
Pharmacology of vanilloid transient receptor potential cation channels | Q34965016 | ||
Regulation of cytosolic calcium in skeletal muscle cells of the mdx mouse under conditions of stress | Q35873884 | ||
Probenecid: novel use as a non-injurious positive inotrope acting via cardiac TRPV2 stimulation | Q36024463 | ||
A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin. | Q36232550 | ||
The control of sarcoplasmic reticulum Ca content in cardiac muscle. | Q36247686 | ||
Dystrophin protects the sarcolemma from stresses developed during muscle contraction | Q36259954 | ||
A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel | Q36323554 | ||
Permeation and selectivity of TRP channels | Q36387952 | ||
Distribution of the Na(+)-Ca2+ exchange protein in mammalian cardiac myocytes: an immunofluorescence and immunocolloidal gold-labeling study | Q36531271 | ||
Hypersensitivity of excitation-contraction coupling in dystrophic cardiomyocytes | Q37174186 | ||
What do we know about the transient receptor potential vanilloid 2 (TRPV2) ion channel? | Q37198480 | ||
Pathways of abnormal stress-induced Ca2+ influx into dystrophic mdx cardiomyocytes | Q37349387 | ||
Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism | Q37419160 | ||
Tranilast administration reduces fibrosis and improves fatigue resistance in muscles of mdx dystrophic mice | Q37539643 | ||
Novel role of transient receptor potential vanilloid 2 in the regulation of cardiac performance. | Q37576154 | ||
Calcium and the damage pathways in muscular dystrophy | Q37710975 | ||
Duchenne muscular dystrophy--what causes the increased membrane permeability in skeletal muscle? | Q37810351 | ||
Dystrophinopathies. | Q37865690 | ||
Oxidative and nitrosative stress in the maintenance of myocardial function. | Q38028230 | ||
Insulin/phosphoinositide 3-kinase pathway accelerates the glucose-induced first-phase insulin secretion through TrpV2 recruitment in pancreatic β-cells | Q39653915 | ||
Dominant-negative inhibition of Ca2+ influx via TRPV2 ameliorates muscular dystrophy in animal models | Q39909880 | ||
Pharmacological characterization and molecular determinants of the activation of transient receptor potential V2 channel orthologs by 2-aminoethoxydiphenyl borate. | Q40098586 | ||
Regulation of capacitative calcium entries by alpha1-syntrophin: association of TRPC1 with dystrophin complex and the PDZ domain of alpha1-syntrophin. | Q40187377 | ||
Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibers | Q41882302 | ||
Involvement of TRPV2 and SOCE in calcium influx disorder in DMD primary human myotubes with a specific contribution of α1-syntrophin and PLC/PKC in SOCE regulation. | Q42520954 | ||
Essential role of TRPV2 ion channel in the sensitivity of dystrophic muscle to eccentric contractions | Q43258728 | ||
Functional and morphological preservation of adult ventricular myocytes in culture by sub-micromolar cytochalasin D supplement | Q45113218 | ||
Regulation of phosphatidylinositol 3-kinase (PI3K)/Akt and nuclear factor-kappa B signaling pathways in dystrophin-deficient skeletal muscle in response to mechanical stretch. | Q46452445 | ||
Mechanical deformation of ventricular myocytes modulates both TRPC6 and Kir2.3 channels | Q46477997 | ||
Dystrophic heart failure blocked by membrane sealant poloxamer | Q46604988 | ||
Immunohistochemical localization of growth factor-regulated channel (GRC) in human tissues | Q48498099 | ||
Stretch-activated calcium channel protein TRPC1 is correlated with the different degrees of the dystrophic phenotype in mdx mice. | Q50272734 | ||
Blockade of sarcolemmal TRPV2 accumulation inhibits progression of dilated cardiomyopathy. | Q50890996 | ||
TRPV2 Is a Component of Osmotically Sensitive Cation Channels in Murine Aortic Myocytes | Q51833369 | ||
P433 | issue | 1 | |
P921 | main subject | cardiomyopathy | Q847583 |
P304 | page(s) | 153-162 | |
P577 | publication date | 2015-01-23 | |
P1433 | published in | Cardiovascular Research | Q4642329 |
P1476 | title | Dystrophic cardiomyopathy: role of TRPV2 channels in stretch-induced cell damage. | |
P478 | volume | 106 |
Q53688949 | A TRPV2 interactome-based signature for prognosis in glioblastoma patients. |
Q36834928 | Axial stretch-dependent cation entry in dystrophic cardiomyopathy: Involvement of several TRPs channels |
Q92502773 | Blockade of TRPV2 is a Novel Therapy for Cardiomyopathy in Muscular Dystrophy |
Q48598460 | Blocking transient receptor potential vanilloid 2 channel in astrocytes enhances astrocyte-mediated neuroprotection after oxygen-glucose deprivation and reoxygenation |
Q38830295 | Calcium Entry Through Thermosensory Channels |
Q39010745 | Can Human Pluripotent Stem Cell-Derived Cardiomyocytes Advance Understanding of Muscular Dystrophies? |
Q28601309 | Cardiac Mechano-Gated Ion Channels and Arrhythmias. |
Q92825511 | Cardiac Pathophysiology and the Future of Cardiac Therapies in Duchenne Muscular Dystrophy |
Q35738899 | Chronic Dosing with Membrane Sealant Poloxamer 188 NF Improves Respiratory Dysfunction in Dystrophic Mdx and Mdx/Utrophin-/- Mice |
Q28082625 | Current understanding of molecular pathology and treatment of cardiomyopathy in duchenne muscular dystrophy |
Q33608478 | Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel Therapies |
Q90734610 | Effects of non-euphoric plant cannabinoids on muscle quality and performance of dystrophic mdx mice |
Q54115486 | Hyperactive ryanodine receptors in human heart failure and ischemic cardiomyopathy reside outside of couplons. |
Q39093425 | Mechano-chemo-transduction in cardiac myocytes. |
Q46359838 | Mitochondrial Mechanosensor Microdomains in Cardiovascular Disorders. |
Q38563300 | Pathoproteomic profiling of the skeletal muscle matrisome in dystrophinopathy associated myofibrosis |
Q38646692 | Piezo channels and GsMTx4: Two milestones in our understanding of excitatory mechanosensitive channels and their role in pathology |
Q50053955 | Probenecid Improves Cardiac Function in Patients With Heart Failure With Reduced Ejection Fraction In Vivo and Cardiomyocyte Calcium Sensitivity In Vitro. |
Q92381168 | Production of TRPV2-targeting functional antibody ameliorating dilated cardiomyopathy and muscular dystrophy in animal models |
Q64076937 | TRP Channels: Current Perspectives in the Adverse Cardiac Remodeling |
Q92133012 | TRPV2 channel as a possible drug target for the treatment of heart failure |
Q89183196 | TRPV4 increases cardiomyocyte calcium cycling and contractility yet contributes to damage in the aged heart following hypoosmotic stress |
Q48126236 | Ups and downs of calcium in the heart |
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