Dystrophic cardiomyopathy: role of TRPV2 channels in stretch-induced cell damage.

scientific article

Dystrophic cardiomyopathy: role of TRPV2 channels in stretch-induced cell damage. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1093/CVR/CVV021
P698PubMed publication ID25616416
P5875ResearchGate publication ID271329751

P50authorErnst NiggliQ52357375
P2093author name stringIsabelle Vögeli
Charlotte Lorin
P2860cites workA capsaicin-receptor homologue with a high threshold for noxious heatQ22009379
Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophyQ24295360
Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathyQ24306846
Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscleQ24308714
International Union of Basic and Clinical Pharmacology. LXXVI. Current progress in the mammalian TRP ion channel familyQ24623493
Stretch-activated ion channels: what are they?Q24630314
X-linked dilated cardiomyopathy and the dystrophin geneQ33688402
TRPV2 is critical for the maintenance of cardiac structure and function in miceQ33729367
The anti-influenza drug oseltamivir exhibits low potential to induce pharmacokinetic drug interactions via renal secretion-correlation of in vivo and in vitro studies.Q33956427
Ultrastructural and functional alterations of EC coupling elements in mdx cardiomyocytes: an analysis from membrane surface to depth.Q52638107
Genetic inhibition of PKA phosphorylation of RyR2 prevents dystrophic cardiomyopathyQ34059173
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Role of nitric oxide in the pathogenesis of muscular dystrophies: a "two hit" hypothesis of the cause of muscle necrosisQ34465820
Pharmacology of vanilloid transient receptor potential cation channelsQ34965016
Regulation of cytosolic calcium in skeletal muscle cells of the mdx mouse under conditions of stressQ35873884
Probenecid: novel use as a non-injurious positive inotrope acting via cardiac TRPV2 stimulationQ36024463
A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin.Q36232550
The control of sarcoplasmic reticulum Ca content in cardiac muscle.Q36247686
Dystrophin protects the sarcolemma from stresses developed during muscle contractionQ36259954
A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channelQ36323554
Permeation and selectivity of TRP channelsQ36387952
Distribution of the Na(+)-Ca2+ exchange protein in mammalian cardiac myocytes: an immunofluorescence and immunocolloidal gold-labeling studyQ36531271
Hypersensitivity of excitation-contraction coupling in dystrophic cardiomyocytesQ37174186
What do we know about the transient receptor potential vanilloid 2 (TRPV2) ion channel?Q37198480
Pathways of abnormal stress-induced Ca2+ influx into dystrophic mdx cardiomyocytesQ37349387
Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanismQ37419160
Tranilast administration reduces fibrosis and improves fatigue resistance in muscles of mdx dystrophic miceQ37539643
Novel role of transient receptor potential vanilloid 2 in the regulation of cardiac performance.Q37576154
Calcium and the damage pathways in muscular dystrophyQ37710975
Duchenne muscular dystrophy--what causes the increased membrane permeability in skeletal muscle?Q37810351
Dystrophinopathies.Q37865690
Oxidative and nitrosative stress in the maintenance of myocardial function.Q38028230
Insulin/phosphoinositide 3-kinase pathway accelerates the glucose-induced first-phase insulin secretion through TrpV2 recruitment in pancreatic β-cellsQ39653915
Dominant-negative inhibition of Ca2+ influx via TRPV2 ameliorates muscular dystrophy in animal modelsQ39909880
Pharmacological characterization and molecular determinants of the activation of transient receptor potential V2 channel orthologs by 2-aminoethoxydiphenyl borate.Q40098586
Regulation of capacitative calcium entries by alpha1-syntrophin: association of TRPC1 with dystrophin complex and the PDZ domain of alpha1-syntrophin.Q40187377
Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibersQ41882302
Involvement of TRPV2 and SOCE in calcium influx disorder in DMD primary human myotubes with a specific contribution of α1-syntrophin and PLC/PKC in SOCE regulation.Q42520954
Essential role of TRPV2 ion channel in the sensitivity of dystrophic muscle to eccentric contractionsQ43258728
Functional and morphological preservation of adult ventricular myocytes in culture by sub-micromolar cytochalasin D supplementQ45113218
Regulation of phosphatidylinositol 3-kinase (PI3K)/Akt and nuclear factor-kappa B signaling pathways in dystrophin-deficient skeletal muscle in response to mechanical stretch.Q46452445
Mechanical deformation of ventricular myocytes modulates both TRPC6 and Kir2.3 channelsQ46477997
Dystrophic heart failure blocked by membrane sealant poloxamerQ46604988
Immunohistochemical localization of growth factor-regulated channel (GRC) in human tissuesQ48498099
Stretch-activated calcium channel protein TRPC1 is correlated with the different degrees of the dystrophic phenotype in mdx mice.Q50272734
Blockade of sarcolemmal TRPV2 accumulation inhibits progression of dilated cardiomyopathy.Q50890996
TRPV2 Is a Component of Osmotically Sensitive Cation Channels in Murine Aortic MyocytesQ51833369
P433issue1
P921main subjectcardiomyopathyQ847583
P304page(s)153-162
P577publication date2015-01-23
P1433published inCardiovascular ResearchQ4642329
P1476titleDystrophic cardiomyopathy: role of TRPV2 channels in stretch-induced cell damage.
P478volume106

Reverse relations

cites work (P2860)
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Q48598460Blocking transient receptor potential vanilloid 2 channel in astrocytes enhances astrocyte-mediated neuroprotection after oxygen-glucose deprivation and reoxygenation
Q38830295Calcium Entry Through Thermosensory Channels
Q39010745Can Human Pluripotent Stem Cell-Derived Cardiomyocytes Advance Understanding of Muscular Dystrophies?
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Q35738899Chronic Dosing with Membrane Sealant Poloxamer 188 NF Improves Respiratory Dysfunction in Dystrophic Mdx and Mdx/Utrophin-/- Mice
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Q64076937TRP Channels: Current Perspectives in the Adverse Cardiac Remodeling
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