| P50 | author | Francisco Altamirano | Q43078274 |
| | Carlos Henríquez-Olguín | Q51317527 |
| P2093 | author name string | Mariana Casas | |
| | Enrique Jaimovich | |
| | Paul D Allen | |
| | Denisse Valladares | |
| | Jose R López | |
| P2860 | cites work | Enhanced expression of the P2X4 receptor in Duchenne muscular dystrophy correlates with macrophage invasion | Q34303848 |
| | Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA. | Q34560693 |
| | Ecto-nucleoside triphosphate diphosphohydrolase 2 modulates local ATP-induced calcium signaling in human HaCaT keratinocytes | Q34639001 |
| | Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy | Q35054988 |
| | Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes | Q36033118 |
| | Purinergic P2 receptors as targets for novel analgesics | Q36286874 |
| | The role of apoptosis in neuromuscular diseases and prospects for anti-apoptosis therapy | Q36464902 |
| | Nonspecific sarcolemmal cation channels are critical for the pathogenesis of malignant hyperthermia | Q36615166 |
| | Calcium and the damage pathways in muscular dystrophy | Q37710975 |
| | ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle. | Q39788355 |
| | ATP stimulates interleukin-6 production via P2Y receptors in human HaCaT keratinocytes | Q40276930 |
| | Increased susceptibility to ATP via alteration of P2X receptor function in dystrophic mdx mouse muscle cells. | Q40296980 |
| | Differences in both inositol 1,4,5-trisphosphate mass and inositol 1,4,5-trisphosphate receptors between normal and dystrophic skeletal muscle cell lines | Q41033786 |
| | Clinical investigation in duchenne dystrophy: 2. Determination of the ?power? of therapeutic trials based on the natural history | Q41449126 |
| | Anti-oxidant and anti-inflammatory mechanisms of amlodipine action to improve endothelial cell dysfunction induced by irreversibly glycated LDL. | Q44137767 |
| | The action potential-evoked sarcoplasmic reticulum calcium release is impaired in mdx mouse muscle fibres | Q44788698 |
| | Cav1.1 controls frequency-dependent events regulating adult skeletal muscle plasticity. | Q45822550 |
| | Increased protein degradation results from elevated free calcium levels found in muscle from mdx mice | Q46266620 |
| | CINRG randomized controlled trial of creatine and glutamine in Duchenne muscular dystrophy. | Q46571375 |
| | Regression of atherosclerosis by amlodipine via anti-inflammatory and anti-oxidative stress actions. | Q54585173 |
| | The mdx mouse diaphragm reproduces the degenerative changes of Duchenne muscular dystrophy | Q59051261 |
| | Evidence for the association of dystrophin with the transverse tubular system in skeletal muscle | Q68140089 |
| | The effects of dihydropyridine derivatives on force and Ca2+ current in frog skeletal muscle fibres | Q68480398 |
| | Excitation contraction coupling in normal and mdx mice | Q68503061 |
| | Comparative antioxidant activities of propranolol, nifedipine, verapamil, and diltiazem against sarcolemmal membrane lipid peroxidation | Q68774177 |
| | Myoplasmic (Ca2+) in Duchenne muscular dystrophy patients | Q68848597 |
| | Clinical investigation in Duchenne dystrophy. VI. Double-blind controlled trial of nifedipine | Q68945618 |
| | Regularity in the change of serum creatine kinase level in Duchenne muscular dystrophy. A study with long-term follow-up cases | Q69520597 |
| | DNA-fragmentation and expression of apoptosis-related proteins in muscular dystrophies | Q73687145 |
| | The muscular dystrophies | Q77731754 |
| | Severely impaired neuromuscular synaptic transmission causes muscle weakness in the Cacna1a-mutant mouse rolling Nagoya | Q80170373 |
| | Activation of caspase 3, 9, 12, and Bax in masseter muscle of mdx mice during necrosis | Q81478294 |
| | Measurement of calcium release due to inositol trisphosphate receptors in skeletal muscle | Q82750954 |
| | Calcium antagonists for Duchenne muscular dystrophy | Q24241080 |
| | Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle | Q24308714 |
| | International Union of Pharmacology LVIII: update on the P2Y G protein-coupled nucleotide receptors: from molecular mechanisms and pathophysiology to therapy | Q24629255 |
| | Calcium currents and transients in co-cultured contracting normal and Duchenne muscular dystrophy human myotubes | Q28344004 |
| | Caveolin-2-deficient mice show evidence of severe pulmonary dysfunction without disruption of caveolae | Q28584954 |
| | Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals | Q30050310 |
| | L-type Ca2+ channel function is linked to dystrophin expression in mammalian muscle | Q33339391 |
| | Clinical heterogeneity of duchenne muscular dystrophy (DMD): definition of sub-phenotypes and predictive criteria by long-term follow-up | Q33405753 |
| | Creatine kinase, cell membrane and Duchenne muscular dystrophy | Q33547058 |
| | Integrated study of 100 patients with Xp21 linked muscular dystrophy using clinical, genetic, immunochemical, and histopathological data. Part 3. Differential diagnosis and prognosis | Q33595768 |
| | Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse | Q33781098 |
| | Excitation-contraction coupling alterations in mdx and utrophin/dystrophin double knockout mice: a comparative study | Q33840407 |
| | Function and genetics of dystrophin and dystrophin-related proteins in muscle | Q34120764 |
| | Intramembrane charge movement and L-type calcium current in skeletal muscle fibers isolated from control and mdx mice | Q34180105 |
| P275 | copyright license | Creative Commons Attribution 3.0 Unported | Q14947546 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 12 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | apoptotic process | Q14599311 |
| | nifedipine | Q39111 |
| P304 | page(s) | e81222 | |
| P577 | publication date | 2013-12-09 | |
| P1433 | published in | PLOS One | Q564954 |
| P1476 | title | Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice | |
| P478 | volume | 8 | |