scholarly article | Q13442814 |
P50 | author | Alan Sher | Q88390533 |
Ruslan Medzhitov | Q517459 | ||
P2093 | author name string | Dragana Jankovic | |
Julio Aliberti | |||
Eric Y Denkers | |||
Soumaya Bennouna | |||
Charles A Scanga | |||
Florence Tilloy | |||
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Toxoplasma gondii | Q131003 |
dendritic cell | Q506253 | ||
P304 | page(s) | 5997-6001 | |
P577 | publication date | 2002-06-01 | |
P1433 | published in | Journal of Immunology | Q3521441 |
P1476 | title | Cutting edge: MyD88 is required for resistance to Toxoplasma gondii infection and regulates parasite-induced IL-12 production by dendritic cells. | |
P478 | volume | 168 |
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Q37719443 | Bacterial infections in Myd88-deficient mice |
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Q35209897 | CD8α(+) dendritic cells are the critical source of interleukin-12 that controls acute infection by Toxoplasma gondii tachyzoites |
Q35018100 | CXCR3-dependent CD4⁺ T cells are required to activate inflammatory monocytes for defense against intestinal infection |
Q44908648 | Candidate gene analysis of ocular toxoplasmosis in Brazil: evidence for a role for toll-like receptor 9 (TLR9). |
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Q38293610 | Containment of aerogenic Mycobacterium tuberculosis infection in mice does not require MyD88 adaptor function for TLR2, -4 and -9. |
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Q33187036 | Critical role of the Toll-like receptor signal adaptor protein MyD88 in acute allograft rejection |
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Q36709991 | Cytokine regulation of immunopathology in toxoplasmosis |
Q35178634 | Dendritic cells and host resistance to infection. |
Q38944438 | Dendritic cells and parasites: from recognition and activation to immune response instruction |
Q35219298 | Dendritic cells as inducers of antimicrobial immunity in vivo |
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Q36710716 | Distinct roles for MyD88 and Toll-like receptors 2, 5, and 9 in phagocytosis of Borrelia burgdorferi and cytokine induction |
Q38266457 | Do you see what I see: Recognition of protozoan parasites by Toll-like receptors |
Q37621648 | Dual role for inflammasome sensors NLRP1 and NLRP3 in murine resistance to Toxoplasma gondii |
Q35220267 | Dual role of MyD88 in rapid clearance of relapsing fever Borrelia spp |
Q36029085 | Early response of mucosal epithelial cells during Toxoplasma gondii infection |
Q34983845 | Evasion of innate immunity by parasitic protozoa |
Q24673216 | Evidence for licensing of IFN-gamma-induced IFN regulatory factor 1 transcription factor by MyD88 in Toll-like receptor-dependent gene induction program |
Q37119134 | Experimental and natural infections in MyD88- and IRAK-4-deficient mice and humans |
Q37700302 | Fatal Mycobacterium tuberculosis infection despite adaptive immune response in the absence of MyD88. |
Q40991779 | Flt3 Ligand Is Essential for Survival and Protective Immune Responses during Toxoplasmosis |
Q35594836 | From cells to signaling cascades: manipulation of innate immunity by Toxoplasma gondii |
Q42623337 | Full-length sequence and expression analysis of a myeloid differentiation factor 88 (MyD88) in half-smooth tongue sole Cynoglossus semilaevis |
Q40053470 | Functional analysis of a zebrafish myd88 mutant identifies key transcriptional components of the innate immune system. |
Q37374974 | Functional aspects of Toll-like receptor/MyD88 signalling during protozoan infection: focus on Toxoplasma gondii |
Q36090933 | Functional diversity and plasticity of human dendritic cell subsets |
Q39500713 | Fusion of foreign T-cell epitopes and addition of TLR agonists enhance immunity against Neospora caninum profilin in cattle |
Q36921649 | Genome-wide comparative analysis revealed significant transcriptome changes in mice after Toxoplasma gondii infection |
Q50332623 | Graft-versus-host disease is independent of innate signaling pathways triggered by pathogens in host hematopoietic cells |
Q37353312 | Gut commensal bacteria direct a protective immune response against Toxoplasma gondii. |
Q37521754 | Helicobacter pylori infection targets adherens junction regulatory proteins and results in increased rates of migration in human gastric epithelial cells. |
Q34941305 | High levels of susceptibility and T helper 2 response in MyD88-deficient mice infected with Leishmania major are interleukin-4 dependent |
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Q36015869 | Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii |
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Q36685549 | Impact of lipoxin-mediated regulation on immune response to infectious disease |
Q36396820 | Impaired innate immunity in mice deficient in interleukin-1 receptor-associated kinase 4 leads to defective type 1 T cell responses, B cell expansion, and enhanced susceptibility to infection with Toxoplasma gondii |
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Q35176429 | Induction and regulation of IL-12-dependent host resistance to Toxoplasma gondii |
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Q50714975 | Induction of dendritic cell migration upon Toxoplasma gondii infection potentiates parasite dissemination. |
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Q34043775 | Inflammasomes in host response to protozoan parasites. |
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Q56880717 | Inflammatory mediators are insufficient for full dendritic cell activation and promote expansion of CD4+ T cell populations lacking helper function |
Q36835115 | Inhibition of TLR3 and TLR4 function and expression in human dendritic cells by helminth parasites |
Q38181331 | Innate immunity to Toxoplasma gondii infection |
Q37735128 | Innate resistance against Toxoplasma gondii: an evolutionary tale of mice, cats, and men. |
Q35175437 | Innate responses to Toxoplasma gondii in mice and humans |
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Q37953824 | Insights into inflammatory bowel disease using Toxoplasma gondii as an infectious trigger |
Q35832497 | Interactions between NK cells and B lymphocytes |
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Q34576329 | Internalization and TLR-dependent type I interferon production by monocytes in response to Toxoplasma gondii |
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Q34992629 | Kaposi's sarcoma-associated herpesvirus-encoded replication and transcription activator impairs innate immunity via ubiquitin-mediated degradation of myeloid differentiation factor 88 |
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Q36879444 | Macrophage migration inhibitory factor (MIF) is critical for the host resistance against Toxoplasma gondii |
Q35884234 | Manipulation of mitogen-activated protein kinase/nuclear factor-kappaB-signaling cascades during intracellular Toxoplasma gondii infection |
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Q39124518 | Modulation of host immune responses to Toxoplasma gondii by microRNAs. |
Q34033981 | Modulation of innate immunity by Toxoplasma gondii virulence effectors |
Q37577380 | Molecular markers of susceptibility to ocular toxoplasmosis, host and guest behaving badly |
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Q37764125 | Monocytes mediate mucosal immunity to Toxoplasma gondii |
Q36426058 | Mucosal defences against orally acquired protozoan parasites, emphasis on Toxoplasma gondii infections |
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Q34491817 | MyD88 deficiency results in tissue-specific changes in cytokine induction and inflammation in interleukin-18-independent mice infected with Borrelia burgdorferi |
Q33835188 | MyD88 expression is required for efficient cross-presentation of viral antigens from infected cells. |
Q36594433 | MyD88 is crucial for the development of a protective CNS immune response to Toxoplasma gondii infection |
Q40630648 | MyD88 is essential for clearance of Leishmania major: possible role for lipophosphoglycan and Toll‐like receptor 2 signaling |
Q37062172 | MyD88 is necessary for neutrophil recruitment in hypersensitivity pneumonitis |
Q33392688 | MyD88 is required for protection from lethal infection with a mouse-adapted SARS-CoV. |
Q36539704 | MyD88 negatively controls hypergammaglobulinemia with autoantibody production during bacterial infection. |
Q33714626 | MyD88 plays a critical T cell-intrinsic role in supporting CD8 T cell expansion during acute lymphocytic choriomeningitis virus infection |
Q36371756 | MyD88 primes macrophages for full-scale activation by interferon-gamma yet mediates few responses to Mycobacterium tuberculosis |
Q34416382 | MyD88 signaling is indispensable for primary influenza A virus infection but dispensable for secondary infection |
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Q37117674 | MyD88-deficient mice display a profound loss in resistance to Mycobacterium tuberculosis associated with partially impaired Th1 cytokine and nitric oxide synthase 2 expression |
Q37451102 | MyD88-deficient mice exhibit decreased parasite-induced immune responses but reduced disease severity in a murine model of neurocysticercosis |
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