scholarly article | Q13442814 |
P2093 | author name string | Helga Vitzthum | |
Heimo Ehmke | |||
Anika Seniuk | |||
Hannah Hetz | |||
Laura Helene Schulte | |||
Maxie Luise Müller | |||
P2860 | cites work | Angiotensin II signaling increases activity of the renal Na-Cl cotransporter through a WNK4-SPAK-dependent pathway | Q24648927 |
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Mechanisms of disease: WNK-ing at the mechanism of salt-sensitive hypertension | Q28254512 | ||
Angiotensin II induces phosphorylation of the thiazide-sensitive sodium chloride cotransporter independent of aldosterone | Q28290886 | ||
Effects of dietary K on cell-surface expression of renal ion channels and transporters | Q28567696 | ||
A SPAK isoform switch modulates renal salt transport and blood pressure | Q28586855 | ||
Aldosterone induces rapid apical translocation of ENaC in early portion of renal collecting system: possible role of SGK. | Q33938102 | ||
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SPAK-knockout mice manifest Gitelman syndrome and impaired vasoconstriction | Q34457030 | ||
Increased blood pressure during potassium depletion in normotensive men. | Q34488423 | ||
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Sodium and potassium in the pathogenesis of hypertension | Q34627313 | ||
WNK1 kinase isoform switch regulates renal potassium excretion. | Q34694518 | ||
Recent advances in distal tubular potassium handling | Q34784068 | ||
Control of aldosterone secretion: a model for convergence in cellular signaling pathways | Q35715226 | ||
Activation of the renal Na+:Cl- cotransporter by angiotensin II is a WNK4-dependent process | Q35974133 | ||
Role of the WNK-activated SPAK kinase in regulating blood pressure | Q36039159 | ||
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Expression and phosphorylation of the Na+-Cl- cotransporter NCC in vivo is regulated by dietary salt, potassium, and SGK1. | Q37337316 | ||
The kidney-specific WNK1 isoform is induced by aldosterone and stimulates epithelial sodium channel-mediated Na+ transport | Q37714173 | ||
The WNK kinase network regulating sodium, potassium, and blood pressure | Q37857562 | ||
Aldosterone paradox: differential regulation of ion transport in distal nephron. | Q37864472 | ||
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Aldosterone does not require angiotensin II to activate NCC through a WNK4-SPAK-dependent pathway | Q41966666 | ||
Potassium depletion and salt sensitivity in essential hypertension | Q43633930 | ||
Gene expression of prostanoid forming enzymes along the rat nephron | Q44173341 | ||
Targeted disruption of the Wnk4 gene decreases phosphorylation of Na-Cl cotransporter, increases Na excretion and lowers blood pressure | Q46146557 | ||
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A simplified method for isolation of large numbers of defined nephron segments | Q73856939 | ||
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P433 | issue | 5 | |
P407 | language of work or name | English | Q1860 |
P1104 | number of pages | 19 | |
P304 | page(s) | 1139-1157 | |
P577 | publication date | 2014-01-06 | |
P1433 | published in | Journal of Physiology | Q7743612 |
P1476 | title | Functional coupling of renal K+ and Na+ handling causes high blood pressure in Na+ replete mice. | |
P478 | volume | 592 |
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