| scholarly article | Q13442814 |
| P2093 | author name string | Jia Lu | |
| Charanjit Kaur | |||
| Viswanathan Sivakumar | |||
| Eng Ang Ling | |||
| YiYu Deng | |||
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| P433 | issue | 3 | |
| P921 | main subject | microglia | Q1622829 |
| hypoxia | Q105688 | ||
| oligodendrocyte | Q916698 | ||
| P304 | page(s) | 387-400 | |
| P577 | publication date | 2008-03-26 | |
| P1433 | published in | Brain Pathology | Q4955776 |
| P1476 | title | Amoeboid microglia in the periventricular white matter induce oligodendrocyte damage through expression of proinflammatory cytokines via MAP kinase signaling pathway in hypoxic neonatal rats. | |
| P478 | volume | 18 |
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| Q34067306 | Hypoxia induced amoeboid microglial cell activation in postnatal rat brain is mediated by ATP receptor P2X4. |
| Q48686569 | Hypoxia-ischemia upregulates TRAIL and TRAIL receptors in the immature rat brain |
| Q40867409 | IL-1β induces hypomyelination in the periventricular white matter through inhibition of oligodendrocyte progenitor cell maturation via FYN/MEK/ERK signaling pathway in septic neonatal rats |
| Q48134729 | Improvement of hypoxia-ischemia-induced white matter injury in immature rat brain by ethyl pyruvate |
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| Q28570133 | Iron and iron regulatory proteins in amoeboid microglial cells are linked to oligodendrocyte death in hypoxic neonatal rat periventricular white matter through production of proinflammatory cytokines and reactive oxygen/nitrogen species |
| Q36150305 | JNK signaling is the shared pathway linking neuroinflammation, blood-brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain |
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| Q28582181 | Microglia-derived proinflammatory cytokines tumor necrosis factor-alpha and interleukin-1beta induce Purkinje neuronal apoptosis via their receptors in hypoxic neonatal rat brain |
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| Q34990855 | TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain. |
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| Q39323255 | White matter damage after traumatic brain injury: A role for damage associated molecular patterns |
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