Adenosine signaling mediates hypoxic responses in the chronic lymphocytic leukemia microenvironment

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Adenosine signaling mediates hypoxic responses in the chronic lymphocytic leukemia microenvironment is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1182/BLOODADVANCES.2016000984
P932PMC publication ID5744057
P698PubMed publication ID29296695

P50authorGianluca GaidanoQ30303486
Davide RossiQ89878155
P2093author name stringSimon C Robson
Richard R Furman
Giorgio Inghirami
Sara Serra
Shih-Shih Chen
Nicholas Chiorazzi
Marta Coscia
Cinzia Bologna
Davide Brusa
Silvia Deaglio
Tiziana Vaisitti
Valentina Audrito
Francesca Arruga
Ozren Jaksic
Roberta Buonincontri
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Protein kinase A-dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor 1.Q39721664
GLUT-1 and CAIX as intrinsic markers of hypoxia in carcinoma of the cervix: relationship to pimonidazole binding.Q40610106
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Adenosine affects expression of membrane molecules, cytokine and chemokine release, and the T-cell stimulatory capacity of human dendritic cellsQ44225915
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Mitochondrial metabolism contributes to oxidative stress and reveals therapeutic targets in chronic lymphocytic leukemiaQ46874661
Extracellular nicotinamide phosphoribosyltransferase (NAMPT) promotes M2 macrophage polarization in chronic lymphocytic leukemia.Q51686376
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P433issue1
P407language of work or nameEnglishQ1860
P921main subjecthypoxiaQ105688
leukemiaQ29496
chronic lymphocytic leukemiaQ1088156
P304page(s)47-61
P577publication date2016-11-29
P1476titleAdenosine signaling mediates hypoxic responses in the chronic lymphocytic leukemia microenvironment
P478volume1

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cites work (P2860)
Q90704170Adenosinergic signaling as a target for natural killer cell immunotherapy
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Q91753700HIF-1α is over-expressed in leukemic cells from TP53-disrupted patients and is a promising therapeutic target in chronic lymphocytic leukemia
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Q60921609Targeting Adenosine Receptor Signaling in Cancer Immunotherapy
Q52323624Targeting the Adenosinergic Axis in Chronic Lymphocytic Leukemia: A Way to Disrupt the Tumor Niche?

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