Hypoxic pre-conditioning suppresses experimental autoimmune encephalomyelitis by modifying multiple properties of blood vessels

Hypoxic pre-conditioning suppresses experimental autoimmune encephalomyelitis by modifying multiple properties of blood vessels is …
instance of (P31):
scholarly articleQ13442814

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P6179Dimensions Publication ID1106525590
P356DOI10.1186/S40478-018-0590-5
P932PMC publication ID6122733
P698PubMed publication ID30176931

P2093author name stringRavi Kant
Richard Milner
Sebok K Halder
P2860cites workTight junctions of the blood-brain barrier: development, composition and regulationQ24292730
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In the hypoxic central nervous system, endothelial cell proliferation is followed by astrocyte activation, proliferation, and increased expression of the alpha 6 beta 4 integrin and dystroglycanQ34039076
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Angiogenesis is present in experimental autoimmune encephalomyelitis and pro-angiogenic factors are increased in multiple sclerosis lesionsQ34499810
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Intermittent hypoxia conditioning protects mitochondrial cytochrome c oxidase of rat cerebellum from ethanol withdrawal stress.Q36001963
Endothelial cell laminin isoforms, laminins 8 and 10, play decisive roles in T cell recruitment across the blood-brain barrier in experimental autoimmune encephalomyelitisQ36326348
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Endogenous adaptation to low oxygen modulates T-cell regulatory pathways in EAE.Q36476035
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Repetitive hypoxia extends endogenous neurovascular protection for strokeQ41991619
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Novel mouse endothelial cell surface marker is suppressed during differentiation of the blood brain barrierQ42804242
VEGF and angiogenesis in acute and chronic MOG((35-55)) peptide induced EAE.Q46114183
Spinal synaptic enhancement with acute intermittent hypoxia improves respiratory function after chronic cervical spinal cord injury.Q46384467
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Chronic mild hypoxia promotes profound vascular remodeling in spinal cord blood vessels, preferentially in white matter, via an α5β1 integrin-mediated mechanismQ47233530
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Tight junctional abnormality in multiple sclerosis white matter affects all calibres of vessel and is associated with blood-brain barrier leakage and active demyelination.Q47596113
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Ischemic preconditioning in 18- to 20-month-old gerbils: long-term survival with functional outcome measuresQ48194290
Blood-brain barrier alterations in the cerebral cortex in experimental autoimmune encephalomyelitisQ48366273
Cerebral protection by hypoxic preconditioning in a murine model of focal ischemia-reperfusionQ48838664
Alterations of Alzheimer's disease in the cholesterol-fed rabbit, including vascular inflammation. Preliminary observations.Q50511973
Synaptic pathways to phrenic motoneurons are enhanced by chronic intermittent hypoxia after cervical spinal cord injury.Q51678562
Chronic intermittent hypoxic preconditioning suppresses pilocarpine-induced seizures and associated hippocampal neurodegeneration.Q51745104
Chronic mild hypoxia ameliorates chronic inflammatory activity in myelin oligodendrocyte glycoprotein (MOG) peptide induced experimental autoimmune encephalomyelitis (EAE).Q52609519
Induction of active and adoptive relapsing experimental autoimmune encephalomyelitis (EAE) using an encephalitogenic epitope of proteolipid proteinQ67537170
Blood-brain barrier damage in acute multiple sclerosis plaques. An immunocytological studyQ70118044
Pathogenesis of multiple sclerosisQ72665671
Neurological deficits caused by tissue hypoxia in neuroinflammatory diseaseQ85849156
Chronic mild hypoxia increases expression of laminins 111 and 411 and the laminin receptor α6β1 integrin at the blood-brain barrierQ90100833
P433issue1
P407language of work or nameEnglishQ1860
P921main subjecthypoxiaQ105688
experimental autoimmune encephalomyelitisQ494066
P304page(s)86
P577publication date2018-09-03
P1433published inActa Neuropathologica CommunicationsQ27724912
P1476titleHypoxic pre-conditioning suppresses experimental autoimmune encephalomyelitis by modifying multiple properties of blood vessels
P478volume6

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Q64965247Propofol improved hypoxia-impaired integrity of blood-brain barrier via modulating the expression and phosphorylation of zonula occludens-1.cites workP2860

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