| scholarly article | Q13442814 |
| P356 | DOI | 10.1002/HON.2287 |
| P698 | PubMed publication ID | 26947932 |
| P50 | author | María Teresa Vargas | Q58888783 |
| Rosario Mª Morales Camacho | Q80141340 | ||
| Sergio Burillo | Q82442393 | ||
| Concepción Prats-Martín | Q86312175 | ||
| P2093 | author name string | J Sánchez | |
| J A Pérez-Simón | |||
| R Bernal | |||
| J R García-Lozano | |||
| I Pérez de Soto | |||
| T Caballero-Velázquez | |||
| P2860 | cites work | RUNX1 amplification in lineage conversion of childhood B-cell acute lymphoblastic leukemia to acute myelogenous leukemia | Q38430224 |
| NUP98-HOXA9 bearing therapy-related myeloid neoplasm involves myeloid-committed cell and induces HOXA5, EVI1, FLT3, and MEIS1 expression. | Q41291574 | ||
| The combined expression of HOXA4 and MEIS1 is an independent prognostic factor in patients with AML. | Q44703694 | ||
| The transcriptome of the leukemogenic homeoprotein HOXA9 in human hematopoietic cells | Q47398312 | ||
| Ring chromosome 21 in healthy persons: different consequencies in females and in males | Q58194230 | ||
| Amplification of the AML1(CBFA2) gene on ring chromosomes in a patient with acute myeloid leukemia and a constitutional ring chromosome 21 | Q73411197 | ||
| Ring syndrome: still true? | Q79772754 | ||
| Duplications in addition to terminal deletions are present in a proportion of ring chromosomes: clues to the mechanisms of formation | Q79843317 | ||
| Gain of chromosome 21 or amplification of chromosome arm 21q is one mechanism for increased ERG expression in acute myeloid leukemia | Q86636781 | ||
| Blood Spotlight on iAMP21 acute lymphoblastic leukemia (ALL), a high-risk pediatric disease | Q28080812 | ||
| High EVI1 expression predicts poor survival in acute myeloid leukemia: a study of 319 de novo AML patients | Q28209409 | ||
| Downregulation of RUNX1/CBFβ by MLL fusion proteins enhances hematopoietic stem cell self-renewal | Q33794291 | ||
| Mechanisms of ring chromosome formation, ring instability and clinical consequences | Q34107702 | ||
| Genomic characterization implicates iAMP21 as a likely primary genetic event in childhood B-cell precursor acute lymphoblastic leukemia | Q34180968 | ||
| SAMSN1 is a tumor suppressor gene in multiple myeloma | Q34347229 | ||
| Complex genomic alterations and gene expression in acute lymphoblastic leukemia with intrachromosomal amplification of chromosome 21. | Q34650243 | ||
| An international study of intrachromosomal amplification of chromosome 21 (iAMP21): cytogenetic characterization and outcome | Q34820210 | ||
| New mechanisms of AML1 gene alteration in hematological malignancies | Q35046095 | ||
| Complex human chromosomal and genomic rearrangements | Q35731269 | ||
| Transcription factor RUNX1 promotes survival of acute myeloid leukemia cells | Q37123913 | ||
| P433 | issue | 4 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | AML | Q295358 |
| P304 | page(s) | 894-899 | |
| P577 | publication date | 2016-03-07 | |
| P1433 | published in | Hematological Oncology | Q15762617 |
| P1476 | title | RUNX1 amplification in AML with myelodysplasia-related changes and ring 21 chromosomes | |
| P478 | volume | 35 |
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