scholarly article | Q13442814 |
P2093 | author name string | Richard A Hodin | |
Laurence G Rahme | |||
Mohammad Hadi Gharedaghi | |||
Marianna Almpani | |||
Mehran Najibi | |||
Fatemeh Adiliaghdam | |||
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The contribution of MvfR to Pseudomonas aeruginosa pathogenesis and quorum sensing circuitry regulation: multiple quorum sensing-regulated genes are modulated without affecting lasRI, rhlRI or the production of N-acyl-L-homoserine lactones | Q28493075 | ||
Identification of anti-virulence compounds that disrupt quorum-sensing regulated acute and persistent pathogenicity | Q28542505 | ||
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Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice | Q29615055 | ||
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Burns in low- and middle-income countries: a review of available literature on descriptive epidemiology, risk factors, treatment, and prevention | Q34538554 | ||
Claudins and the modulation of tight junction permeability | Q34650517 | ||
Prediction of multiple infections after severe burn trauma: a prospective cohort study | Q34825134 | ||
Antibacterial-resistant Pseudomonas aeruginosa: clinical impact and complex regulation of chromosomally encoded resistance mechanisms | Q35007624 | ||
Burn Injury Alters the Intestinal Microbiome and Increases Gut Permeability and Bacterial Translocation | Q35685953 | ||
Burn-induced gut barrier injury is attenuated by phosphodiesterase inhibition: effects on tight junction structural proteins | Q36241916 | ||
A mouse model of pathological small intestinal epithelial cell apoptosis and shedding induced by systemic administration of lipopolysaccharide | Q37287436 | ||
Evidence for Direct Control of Virulence and Defense Gene Circuits by the Pseudomonas aeruginosa Quorum Sensing Regulator, MvfR | Q37291373 | ||
Cytokine regulation of tight junctions | Q37308566 | ||
Postnatal regulation of MAMDC4 in the porcine intestinal epithelium is influenced by bacterial colonization | Q37419012 | ||
Potential of Lactobacillus plantarum CCFM639 in Protecting against Aluminum Toxicity Mediated by Intestinal Barrier Function and Oxidative Stress. | Q37535417 | ||
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Gut to Brain Dysbiosis: Mechanisms Linking Western Diet Consumption, the Microbiome, and Cognitive Impairment. | Q37611785 | ||
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Epithelial cell shedding and barrier function: a matter of life and death at the small intestinal villus tip | Q38271846 | ||
Polypharmacology Approaches against the Pseudomonas aeruginosa MvfR Regulon and Their Application in Blocking Virulence and Antibiotic Tolerance | Q38852941 | ||
Regulation of intestinal permeability: The role of proteases | Q39239228 | ||
In-depth Profiling of MvfR-Regulated Small Molecules in Pseudomonas aeruginosa after Quorum Sensing Inhibitor Treatment | Q40179538 | ||
Assessment of the Intestinal Barrier with Five Different Permeability Tests in Healthy C57BL/6J and BALB/cJ Mice | Q40371820 | ||
Proinflammatory cytokines disrupt epithelial barrier function by apoptosis-independent mechanisms | Q40614064 | ||
Effects of Mesalamine Treatment on Gut Barrier Integrity After Burn Injury | Q41036330 | ||
Increased intestinal permeability associated with infection in burn patients | Q41390988 | ||
Burn-injury affects gut-associated lymphoid tissues derived CD4+ T cells | Q41822917 | ||
Increased gut permeability early after burns correlates with the extent of burn injury | Q43462274 | ||
Differential pathophysiology of bacterial translocation after thermal injury and sepsis | Q44014128 | ||
TNF-alpha-induced increase in intestinal epithelial tight junction permeability requires NF-kappa B activation | Q44758263 | ||
PSEUDOMONAS BURN WOUND SEPSIS. II HEMATOGENOUS INFECTION AT THE JUNCTION OF THE BURN WOUND AND THE UNBURNED HYPODERMIS. | Q44774788 | ||
Exacerbation of intestinal permeability in rats after a two-hit injury: burn and Enterococcus faecalis infection | Q45217244 | ||
Pharmacological Inhibition of the Pseudomonas aeruginosa MvfR Quorum-Sensing System Interferes with Biofilm Formation and Potentiates Antibiotic-Mediated Biofilm Disruption | Q48657210 | ||
Systemic inflammatory responses and multiple organ dysfunction syndrome following skin burn wound and Pseudomonas aeruginosa infection in mice. | Q50960262 | ||
Risk factors for nosocomial infection and mortality in burn patients: 10 years of experience at a university hospital. | Q50994040 | ||
Segmental expression of claudin proteins correlates with tight junction barrier properties in rat intestine | Q56972159 | ||
A standard animal burn | Q72220242 | ||
Additive effects of thermal injury and infection on gut permeability | Q72296273 | ||
PSEUDOMONAS BURN WOUND SEPSIS. I PATHOGENESIS OF EXPERIMENTAL PSEUDOMONAS BURN WOUND SEPSIS | Q76846032 | ||
Cutaneous burn increases apoptosis in the gut epithelium of mice | Q77895098 | ||
P433 | issue | 5 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | quorum sensing | Q905754 |
P304 | page(s) | 4057-4066 | |
P577 | publication date | 2019-05-01 | |
P1433 | published in | Molecular Medicine Reports | Q26842180 |
P1476 | title | Targeting bacterial quorum sensing shows promise in improving intestinal barrier function following burn‑site infection | |
P478 | volume | 19 |
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