Nav1.7 via Promotion of ERK in the Trigeminal Ganglion Plays an Important Role in the Induction of Pulpitis Inflammatory Pain

scientific article published on 28 March 2019

Nav1.7 via Promotion of ERK in the Trigeminal Ganglion Plays an Important Role in the Induction of Pulpitis Inflammatory Pain is …
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scholarly articleQ13442814

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P356DOI10.1155/2019/6973932
P932PMC publication ID6458889
P698PubMed publication ID31032358

P50authorWenkai JiangQ91586836
Longxing NiQ91586840
P2093author name stringWei Wang
Shukai Sun
Jiangxing Sun
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Glial interleukin-1β upregulates neuronal sodium channel 1.7 in trigeminal ganglion contributing to temporomandibular joint inflammatory hypernociception in rats.Q54115074
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Increased peripheral nerve excitability and local NaV1.8 mRNA up-regulation in painful neuropathy.Q41899981
ERK-GluR1 phosphorylation in trigeminal spinal subnucleus caudalis neurons is involved in pain associated with dry tongueQ42408881
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P407language of work or nameEnglishQ1860
P304page(s)6973932
P577publication date2019-03-28
P1433published inBioMed Research InternationalQ17509958
P1476titleNav1.7 via Promotion of ERK in the Trigeminal Ganglion Plays an Important Role in the Induction of Pulpitis Inflammatory Pain
P478volume2019

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