scholarly article | Q13442814 |
review article | Q7318358 |
P6179 | Dimensions Publication ID | 1112158825 |
P356 | DOI | 10.1007/S12272-019-01127-Y |
P932 | PMC publication ID | 6399179 |
P698 | PubMed publication ID | 30771209 |
P2093 | author name string | Jae-Ho Cheong | |
Min-Kyue Shin | |||
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Integrated Pharmacodynamic Analysis Identifies Two Metabolic Adaption Pathways to Metformin in Breast Cancer | Q58696780 | ||
The Involvement of PPARs in the Peculiar Energetic Metabolism of Tumor Cells | Q59529106 | ||
Selective Cytotoxicity of the NAMPT Inhibitor FK866 Toward Gastric Cancer Cells With Markers of the Epithelial-Mesenchymal Transition, Due to Loss of NAPRT | Q60683128 | ||
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An inhibitor of oxidative phosphorylation exploits cancer vulnerability | Q89070252 | ||
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The Mitochondrial-Encoded Peptide MOTS-c Translocates to the Nucleus to Regulate Nuclear Gene Expression in Response to Metabolic Stress | Q89460819 | ||
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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function | Q35223050 | ||
Dual inhibition of tumor energy pathway by 2-deoxyglucose and metformin is effective against a broad spectrum of preclinical cancer models | Q35613319 | ||
Antibiotics that target mitochondria effectively eradicate cancer stem cells, across multiple tumor types: treating cancer like an infectious disease | Q35741510 | ||
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The Emerging Hallmarks of Cancer Metabolism | Q36468964 | ||
Metabolic Heterogeneity in Human Lung Tumors. | Q36578111 | ||
Oncogenic BRAF regulates oxidative metabolism via PGC1α and MITF. | Q36793719 | ||
MYC inhibition induces metabolic changes leading to accumulation of lipid droplets in tumor cells | Q36950291 | ||
Inhibition of fatty acid oxidation as a therapy for MYC-overexpressing triple-negative breast cancer | Q36967938 | ||
Mitochondria-Targeted Analogues of Metformin Exhibit Enhanced Antiproliferative and Radiosensitizing Effects in Pancreatic Cancer Cells | Q37059785 | ||
Cancer metabolism: fatty acid oxidation in the limelight | Q37155867 | ||
Aldehyde dehydrogenase is used by cancer cells for energy metabolism | Q37463894 | ||
Integrated omics-analysis reveals Wnt-mediated NAD+ metabolic reprogramming in cancer stem-like cells | Q37565536 | ||
Phospholipase D1-regulated autophagy supplies free fatty acids to counter nutrient stress in cancer cells | Q37603284 | ||
Changing appetites: the adaptive advantages of fuel choice | Q37624803 | ||
In vivo Reprogramming of Cancer Metabolism by MYC. | Q37746816 | ||
Oncocytic mania: a review of oncocytic lesions throughout the body | Q37837353 | ||
Profiling metabolic networks to study cancer metabolism | Q38064557 | ||
Peroxisome proliferator-activated receptor-α activation and excess energy burning in hepatocarcinogenesis | Q38167545 | ||
Emerging concepts in bioenergetics and cancer research: metabolic flexibility, coupling, symbiosis, switch, oxidative tumors, metabolic remodeling, signaling and bioenergetic therapy | Q38301212 | ||
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P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | drug discovery | Q1418791 |
neoplastic stem cells | Q1638475 | ||
tumor microenvironment | Q1786433 | ||
energy metabolism | Q30225378 | ||
P304 | page(s) | 113-127 | |
P577 | publication date | 2019-02-01 | |
P1433 | published in | Archives of Pharmacal Research | Q13557312 |
P1476 | title | Mitochondria-centric bioenergetic characteristics in cancer stem-like cells | |
P478 | volume | 42 |