| scholarly article | Q13442814 |
| P50 | author | Maria Teresa Fiorillo | Q88640605 |
| Maria Maddalena Angioni | Q90835689 | ||
| Matteo Piga | Q37829183 | ||
| Alberto Floris | Q57569408 | ||
| P2093 | author name string | Fabiana Paladini | |
| Rosa Sorrentino | |||
| Alberto Cauli | |||
| Alessandro Mathieu | |||
| Valentina Tedeschi | |||
| Silvia Pinna | |||
| Mattia Congia | |||
| Enrico Mascia | |||
| Grazia Dessole | |||
| P2860 | cites work | Activating KIR genes are associated with ankylosing spondylitis in Asian populations | Q61021670 |
| CD8+ T-cell mediated self-reactivity in HLA-B27 context as a consequence of dual peptide conformation | Q62593802 | ||
| Cell-surface expression and immune receptor recognition of HLA-B27 homodimers | Q78505485 | ||
| Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide | Q80094575 | ||
| HLA-E surface expression is independent of the availability of HLA class I signal sequence-derived peptides in human tumor cell lines | Q81201456 | ||
| HLA-G and HLA-E in patients with juvenile idiopathic arthritis | Q83033960 | ||
| The Rapid Induction of HLA-E Is Essential for the Survival of Antigen-Activated Naive CD4 T Cells from Attack by NK Cells | Q85207178 | ||
| Significance of association of HLA-C and HLA-E with psoriatic arthritis | Q86123939 | ||
| Interactions between HLA-G and HLA-E in Physiological and Pathological Conditions | Q21131161 | ||
| HLA-E allelic variants. Correlating differential expression, peptide affinities, crystal structures, and thermal stabilities | Q24314932 | ||
| Structural basis for NKG2A/CD94 recognition of HLA-E. | Q24336854 | ||
| Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants | Q24646663 | ||
| HLA-B27 misfolding and ankylosing spondylitis | Q26860818 | ||
| TAP- and tapasin-dependent HLA-E surface expression correlates with the binding of an MHC class I leader peptide | Q28258860 | ||
| HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C | Q28263511 | ||
| Interaction pattern of Arg 62 in the A-pocket of differentially disease-associated HLA-B27 subtypes suggests distinct TCR binding modes | Q34189720 | ||
| HLA-B27. | Q34471169 | ||
| Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis | Q35534758 | ||
| Analysis of IL-17(+) cells in facet joints of patients with spondyloarthritis suggests that the innate immune pathway might be of greater relevance than the Th17-mediated adaptive immune response | Q35557923 | ||
| HLA-E is a major ligand for the natural killer inhibitory receptor CD94/NKG2A | Q36065870 | ||
| Lifestyle factors may modify the effect of disease activity on radiographic progression in patients with ankylosing spondylitis: a longitudinal analysis | Q36215822 | ||
| Increased HLA-E expression in white matter lesions in multiple sclerosis. | Q36488406 | ||
| Regulation of CD8+ regulatory T cells: Interruption of the NKG2A-Qa-1 interaction allows robust suppressive activity and resolution of autoimmune disease | Q37039579 | ||
| HLA-B27 and ankylosing spondylitis geographic distribution as the result of a genetic selection induced by malaria endemic? A review supporting the hypothesis. | Q37166851 | ||
| The arthritis-associated HLA-B*27:05 allele forms more cell surface B27 dimer and free heavy chain ligands for KIR3DL2 than HLA-B*27:09. | Q37234677 | ||
| Causes and consequences of endoplasmic reticulum stress in rheumatic disease | Q39025066 | ||
| The Ankylosing Spondylitis-associated HLA-B*2705 presents a B*0702-restricted EBV epitope and sustains the clonal amplification of cytotoxic T cells in patients | Q39715318 | ||
| HLA-E: strong association with beta2-microglobulin and surface expression in the absence of HLA class I signal sequence-derived peptides | Q39932959 | ||
| CD94/NKG2A inhibits NK cell activation by disrupting the actin network at the immunological synapse | Q40236182 | ||
| HLA-E surface expression depends on binding of TAP-dependent peptides derived from certain HLA class I signal sequences | Q41042473 | ||
| HLA-E gene polymorphism associates with ankylosing spondylitis in Sardinia. | Q41201765 | ||
| Contribution of KIR3DL1/3DS1 to ankylosing spondylitis in human leukocyte antigen-B27 Caucasian populations | Q42136558 | ||
| Relevance of residue 116 of HLA-B27 in determining susceptibility to ankylosing spondylitis. | Q43814358 | ||
| High Association of an HL-A Antigen, W27, with Ankylosing Spondylitis | Q48012715 | ||
| HLA-B*2709 and lack of susceptibility to sacroiliitis: further support from the clinic. | Q51826796 | ||
| HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response. | Q53663069 | ||
| Analysis of killer immunoglobulin-like receptor genes in ankylosing spondylitis | Q57667583 | ||
| Identification of previously unrecognized predisposing factors for ankylosing spondylitis from analysis of HLA–B27 extended haplotypes in sardinia | Q58852357 | ||
| P433 | issue | 2 | |
| P304 | page(s) | e000597 | |
| P577 | publication date | 2018-07-13 | |
| P1433 | published in | RMD open | Q27726913 |
| P1476 | title | Expression analysis of HLA-E and NKG2A and NKG2C receptors points at a role for natural killer function in ankylosing spondylitis. | |
| P478 | volume | 4 |
| Q90397104 | An Exploratory Gene Expression Study of the Intestinal Mucosa of Patients with Non-Celiac Wheat Sensitivity | cites work | P2860 |
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