scholarly article | Q13442814 |
P953 | full work available at URL | http://cancerres.aacrjournals.org/cgi/content/abstract/42/4/1473 |
P698 | PubMed publication ID | 6174225 |
P2093 | author name string | Lehmann AR | |
Mayne LV | |||
P433 | issue | 4 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | xeroderma pigmentosum | Q612693 |
P304 | page(s) | 1473-1478 | |
P577 | publication date | 1982-04-01 | |
P1433 | published in | Cancer Research | Q326097 |
P1476 | title | Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum | |
P478 | volume | 42 |
Q41352478 | A Novel Mutation in ERCC8 Gene Causing Cockayne Syndrome |
Q36552711 | A UV-responsive G2 checkpoint in rodent cells |
Q27318209 | A high-fat diet and NAD(+) activate Sirt1 to rescue premature aging in cockayne syndrome |
Q42177676 | A rapid, comprehensive system for assaying DNA repair activity and cytotoxic effects of DNA-damaging reagents |
Q39694111 | A ubiquitin-binding domain in Cockayne syndrome B required for transcription-coupled nucleotide excision repair |
Q37021545 | A ubiquitylation site in Cockayne syndrome B required for repair of oxidative DNA damage, but not for transcription-coupled nucleotide excision repair |
Q37219471 | Accumulation of (5'S)-8,5'-cyclo-2'-deoxyadenosine in organs of Cockayne syndrome complementation group B gene knockout mice |
Q86630527 | Age-associated decreases in human DNA repair capacity: Implications for the skin |
Q38721258 | Amplification of unscheduled DNA synthesis signal enables fluorescence-based single cell quantification of transcription-coupled nucleotide excision repair. |
Q37488658 | BHK cell lines with increased rates of gene amplification are hypersensitive to ultraviolet light |
Q24302667 | BRCA1/BARD1 inhibition of mRNA 3' processing involves targeted degradation of RNA polymerase II. |
Q34554610 | Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic disease |
Q37418002 | Blurring the line between the DNA damage response and transcription: the importance of chromatin dynamics |
Q30854865 | Chromatin restoration following nucleotide excision repair involves the incorporation of ubiquitinated H2A at damaged genomic sites |
Q38339920 | Cisplatin- and UV-damaged DNA lure the basal transcription factor TFIID/TBP. |
Q69109211 | Clinical and biochemical studies in three patients with severe early infantile Cockayne syndrome |
Q37638150 | Cockayne Syndrome group B protein stimulates NEIL2 DNA glycosylase activity. |
Q34590427 | Cockayne syndrome B protein regulates the transcriptional program after UV irradiation |
Q28306117 | Cockayne syndrome B protein stimulates apurinic endonuclease 1 activity and protects against agents that introduce base excision repair intermediates |
Q24317053 | Cockayne syndrome group B protein enhances elongation by RNA polymerase II |
Q24318851 | Cockayne syndrome protein B interacts with and is phosphorylated by c-Abl tyrosine kinase |
Q38923380 | Cockayne syndrome: Clinical features, model systems and pathways |
Q34232807 | Cockayne syndrome: Review of 140 cases |
Q36433210 | Cockayne syndrome: magnetic resonance images of the brain in a severe form with early onset |
Q28537642 | Cockayne syndrome: varied requirement of transcription-coupled nucleotide excision repair for the removal of three structurally different adducts from transcribed DNA |
Q41653440 | Cockayne's syndrome: correlation of clinical features with cellular sensitivity of RNA synthesis to UV irradiation |
Q37593459 | Complete absence of Cockayne syndrome group B gene product gives rise to UV-sensitive syndrome but not Cockayne syndrome |
Q89722956 | Continuous transcription initiation guarantees robust repair of all transcribed genes and regulatory regions |
Q28238854 | DNA repair diseases: What do they tell us about cancer and aging? |
Q36078695 | DNA transcription and repair: a confluence |
Q50106852 | Deep intronic variation in splicing regulatory element of the ERCC8 gene associated with severe but long-term survival Cockayne syndrome |
Q34348395 | Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells |
Q28594975 | Developmental defects and male sterility in mice lacking the ubiquitin-like DNA repair gene mHR23B |
Q28509765 | Different effects of CSA and CSB deficiency on sensitivity to oxidative DNA damage |
Q36850507 | Differential repair of DNA damage in the human metallothionein gene family |
Q40450525 | Differential repair of UV damage in Saccharomyces cerevisiae |
Q43183885 | Differential repair of UV damage in rad mutants of Saccharomyces cerevisiae: a possible function of G2 arrest upon UV irradiation |
Q33559326 | Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells |
Q28593341 | Differential role of transcription-coupled repair in UVB-induced G2 arrest and apoptosis in mouse epidermis |
Q35006896 | Disorders of nucleotide excision repair: the genetic and molecular basis of heterogeneity |
Q33603063 | Dissociation of CAK from core TFIIH reveals a functional link between XP-G/CS and the TFIIH disassembly state |
Q34407717 | Dysregulation of gene expression as a cause of Cockayne syndrome neurological disease |
Q33592846 | Early onset Cockayne's syndrome: case reports with neuropathological and fibroblast studies |
Q34503412 | Elements That Regulate the DNA Damage Response of Proteins Defective in Cockayne Syndrome |
Q36690666 | Evidence from mutation spectra that the UV hypermutability of xeroderma pigmentosum variant cells reflects abnormal, error-prone replication on a template containing photoproducts |
Q64230140 | Fluorescently-labelled CPD and 6-4PP photolyases: new tools for live-cell DNA damage quantification and laser-assisted repair |
Q38210260 | Gearing up chromatin: A role for chromatin remodeling during the transcriptional restart upon DNA damage |
Q43599752 | Gene-specific DNA repair in terminally differentiating rat myoblasts |
Q40819236 | Genomic damage and its repair in young and aging brain |
Q40019757 | HHR23B, a human Rad23 homolog, stimulates XPC protein in nucleotide excision repair in vitro |
Q33808192 | Hypomorphic PCNA mutation underlies a human DNA repair disorder. |
Q42512985 | Local UV-induced DNA damage in cell nuclei results in local transcription inhibition |
Q27022616 | Mammalian transcription-coupled excision repair |
Q40693785 | Mechanisms of transcription-repair coupling and mutation frequency decline |
Q89393146 | Mechanistic insights in transcription-coupled nucleotide excision repair of ribosomal DNA |
Q90387567 | Mechanistic insights into the regulation of transcription and transcription-coupled DNA repair by Cockayne syndrome protein B |
Q35943160 | Mfd is required for rapid recovery of transcription following UV-induced DNA damage but not oxidative DNA damage in Escherichia coli |
Q36874609 | Mitochondrial deficiency in Cockayne syndrome |
Q24538671 | Molecular analysis of mutations in the CSB (ERCC6) gene in patients with Cockayne syndrome |
Q33758049 | Molecular characterization of an acidic region deletion mutant of Cockayne syndrome group B protein |
Q39762223 | Multiomic Analysis of the UV-Induced DNA Damage Response |
Q24310583 | Mutations in UVSSA cause UV-sensitive syndrome and impair RNA polymerase IIo processing in transcription-coupled nucleotide-excision repair |
Q38880991 | NELF-E is recruited to DNA double-strand break sites to promote transcriptional repression and repair. |
Q49689803 | Non-coding RNA networks in cancer. |
Q40557149 | Nuclear matrix associated DNA is preferentially repaired in normal human fibroblasts, exposed to a low dose of ultraviolet light but not in Cockayne's syndrome fibroblasts |
Q37308518 | Nucleic acid binding activity of human Cockayne syndrome B protein and identification of Ca(2+) as a novel metal cofactor |
Q72236445 | Nucleotide excision repair in yeast |
Q28238547 | Physiological consequences of defects in ERCC1-XPF DNA repair endonuclease |
Q91859408 | Poly(ADP-ribose) polymerase 1 (PARP1) promotes oxidative stress-induced association of Cockayne syndrome group B protein with chromatin |
Q38125728 | Post-transcriptional regulation of DNA damage-responsive gene expression |
Q34660120 | Preferential DNA repair in expressed genes |
Q37408662 | Preferential DNA repair of an active gene in human cells |
Q34025252 | Premature aging and cancer in nucleotide excision repair-disorders |
Q37229309 | Progeroid syndromes: probing the molecular basis of aging? |
Q36458801 | Protein oxidative damage is associated with life expectancy of houseflies |
Q38741213 | RNA Processing and Genome Stability: Cause and Consequence |
Q39721529 | Recovery of RNA polymerase II synthesis following DNA damage in mutants of Saccharomyces cerevisiae defective in nucleotide excision repair |
Q24644162 | Recruitment of the putative transcription-repair coupling factor CSB/ERCC6 to RNA polymerase II elongation complexes |
Q36191704 | Regulation of active genome integrity and expression by Rad26p |
Q90110326 | Regulation of the RNAPII Pool Is Integral to the DNA Damage Response |
Q33695867 | Regulation of ultraviolet light-induced gene expression by gene size |
Q34636957 | Role of transcription-coupled DNA repair in susceptibility to environmental carcinogenesis |
Q54959914 | Similarity in the effect of caffeine on DNA synthesis after UV irradiation between xeroderma pigmentosum variant cells and mouse cells. |
Q36545639 | Sirt1 suppresses RNA synthesis after UV irradiation in combined xeroderma pigmentosum group D/Cockayne syndrome (XP-D/CS) cells |
Q41331200 | Somatic cell mutation frequency at the HPRT, T-cell antigen receptor and glycophorin A loci in Cockayne syndrome |
Q35953606 | Special issue on the segmental progeria Cockayne syndrome |
Q41992139 | Stabilization of Ultraviolet (UV)-stimulated Scaffold Protein A by Interaction with Ubiquitin-specific Peptidase 7 Is Essential for Transcription-coupled Nucleotide Excision Repair |
Q35804859 | Strand specificity for UV-induced DNA repair and mutations in the Chinese hamster HPRT gene |
Q22010799 | The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair |
Q38815648 | The C-terminal Region and SUMOylation of Cockayne Syndrome Group B Protein Play Critical Roles in Transcription-coupled Nucleotide Excision Repair |
Q52560605 | The Cellular Response to Transcription-Blocking DNA Damage. |
Q36871119 | The Cockayne Syndrome Natural History (CoSyNH) study: clinical findings in 102 individuals and recommendations for care. |
Q33887586 | The Cockayne syndrome B protein, involved in transcription-coupled DNA repair, resides in an RNA polymerase II-containing complex |
Q28240933 | The cockayne syndrome B protein is essential for neuronal differentiation and neuritogenesis |
Q33640104 | The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA. |
Q34609713 | The human CSB (ERCC6) gene corrects the transcription-coupled repair defect in the CHO cell mutant UV61 |
Q37593710 | The many faces of Cockayne syndrome |
Q35907237 | The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNA |
Q34649133 | The role of genetics in the establishment and maintenance of the epigenome. |
Q40019597 | The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes |
Q39718264 | The sensitivity of human fibroblasts to N-acetoxy-2-acetylaminofluorene is determined by the extent of transcription-coupled repair, and/or their capability to counteract RNA synthesis inhibition |
Q40605912 | The ultraviolet sensitivity of Cockayne syndrome cells is not a consequence of reduced cellular NAD content |
Q34640431 | Transcription and DNA damage: a link to a kink |
Q53801893 | Transcription-associated events affecting genomic integrity. |
Q35195489 | Transcription-coupled repair in yeast is independent from ubiquitylation of RNA pol II: implications for Cockayne's syndrome |
Q39607390 | Transcription-coupled repair is inducible in hamster cells |
Q33594900 | Transcription-coupled repair of DNA damage: unanticipated players, unexpected complexities |
Q42951445 | Transcription-related human disorders. |
Q38690465 | Transcriptional Signatures of Aging. |
Q38715789 | UV Irradiation Induces a Non-coding RNA that Functionally Opposes the Protein Encoded by the Same Gene |
Q37249463 | UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II. |
Q36458963 | Ultraviolet-induced mutations in Cockayne syndrome cells are primarily caused by cyclobutane dimer photoproducts while repair of other photoproducts is normal. |
Q39393194 | Uncommon nucleotide excision repair phenotypes revealed by targeted high-throughput sequencing |
Q35195587 | Xeroderma pigmentosum and Cockayne syndrome: overlapping clinical and biochemical phenotypes |
Q36728551 | Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes |
Q35194613 | Xeroderma pigmentosum complementation group G associated with Cockayne syndrome |
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