p16 INK4a can initiate an autonomous senescence program

scientific article published on 01 March 2000

p16 INK4a can initiate an autonomous senescence program is …
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scholarly articleQ13442814

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P6179Dimensions Publication ID1001521369
P356DOI10.1038/SJ.ONC.1203438
P698PubMed publication ID10763818

P2093author name stringDai CY
Enders GH
P2860cites workGrowth suppression by p16ink4 requires functional retinoblastoma proteinQ24317567
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Tetracycline-regulatable factors with distinct dimerization domains allow reversible growth inhibition by p16.Q42823937
Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells.Q44816904
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p16INK4a, but not constitutively active pRb, can impose a sustained G1 arrest: molecular mechanisms and implications for oncogenesis.Q45861136
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Increased p16 expression with first senescence arrest in human mammary epithelial cells and extended growth capacity with p16 inactivationQ47801895
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p16 gene overexpression in mouse bladder carcinomas.Q53433802
Role of the INK4a locus in tumor suppression and cell mortality.Q53455197
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Alterations in DNA Methylation: A Fundamental Aspect of NeoplasiaQ56418625
Retinoblastoma-protein-dependent cell-cycle inhibition by the tumour suppressor p16Q59082348
Loss of RB and MTS1/CDKN2 (p16) expression in human sarcomasQ73622335
Inhibitors of cyclin-dependent kinases induce features of replicative senescence in early passage human diploid fibroblastsQ74344707
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P433issue13
P407language of work or nameEnglishQ1860
P304page(s)1613-1622
P577publication date2000-03-01
P1433published inOncogeneQ1568657
P1476titlep16 INK4a can initiate an autonomous senescence program
P478volume19