Nuclear Rad51 foci induced by DNA damage are distinct from Rad51 foci associated with B cell activation and recombination

scientific article published on 01 November 1997

Nuclear Rad51 foci induced by DNA damage are distinct from Rad51 foci associated with B cell activation and recombination is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1006/EXCR.1997.3761
P698PubMed publication ID9417871

P2093author name stringM J Li
N Maizels
P433issue1
P407language of work or nameEnglishQ1860
P304page(s)93-100
P577publication date1997-11-01
P1433published inExperimental Cell ResearchQ1524289
P1476titleNuclear Rad51 foci induced by DNA damage are distinct from Rad51 foci associated with B cell activation and recombination
P478volume237

Reverse relations

cites work (P2860)
Q35625730A molecular portrait of Arabidopsis meiosis
Q33893554ASCIZ regulates lesion-specific Rad51 focus formation and apoptosis after methylating DNA damage
Q54532004Analysis of mouse Rad54 expression and its implications for homologous recombination.
Q40820236Coordinated response of mammalian Rad51 and Rad52 to DNA damage
Q34367487DNA-PK-dependent binding of DNA ends to plasmids containing nuclear matrix attachment region DNA sequences: evidence for assembly of a repair complex
Q42611090Localization and dynamic relocalization of mammalian Rad52 during the cell cycle and in response to DNA damage
Q34542246Molecular mechanism of class switch recombination: linkage with somatic hypermutation
Q40959743Mouse Rad54 affects DNA conformation and DNA-damage-induced Rad51 foci formation
Q34581235RAD51 supports spontaneous non-homologous recombination in mammalian cells, but not the corresponding process induced by topoisomerase inhibitors
Q64389096Recombination-based mechanisms for somatic hypermutation
Q24551864The Rad51 paralog Rad51B promotes homologous recombinational repair
Q33601052The contribution of homologous recombination in preserving genome integrity in mammalian cells