scholarly article | Q13442814 |
P356 | DOI | 10.1002/GCC.1178 |
P698 | PubMed publication ID | 11550284 |
P2093 | author name string | C Hoffmann | |
T Ozaki | |||
K L Schaefer | |||
W Winkelmann | |||
B Dockhorn-Dworniczak | |||
C Poremba | |||
H Juergens | |||
S Ahrens | |||
M Paulussen | |||
W Boecker | |||
A Hillmann | |||
C Brinkschmidt | |||
D Wai | |||
J Rerin | |||
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Gene fusion with an ETS DNA-binding domain caused by chromosome translocation in human tumours | Q24298221 | ||
A new member of the ETS family fused to EWS in Ewing tumors | Q24317176 | ||
The Ewing's sarcoma EWS/FLI-1 fusion gene encodes a more potent transcriptional activator and is a more powerful transforming gene than FLI-1 | Q24629399 | ||
EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma | Q27851507 | ||
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Comparative genomic hybridization analysis of myoepithelial carcinoma of the breast | Q30883731 | ||
Assessment of genetic changes in hepatocellular carcinoma by comparative genomic hybridization analysis: relationship to disease stage, tumor size, and cirrhosis | Q35754634 | ||
Association of EWS-FLI1 type 1 fusion with lower proliferative rate in Ewing's sarcoma | Q35809865 | ||
Recurrent gains of 1q, 8 and 12 in the Ewing family of tumours by comparative genomic hybridization | Q36421638 | ||
Overexpression of p53 protein in primary Ewing's sarcoma of bone: relationship to tumour stage, response and prognosis | Q36617437 | ||
Demonstration of the translocation der(16)t(1;16)(q12;q11.2) in interphase nuclei of Ewing tumors | Q38506119 | ||
Optimizing comparative genomic hybridization for analysis of DNA sequence copy number changes in solid tumors | Q39370472 | ||
Comparative genomic hybridization and telomerase activity analysis identify two biologically different groups of 4s neuroblastomas | Q42859308 | ||
Three independently deleted regions at chromosome arm 16q in human prostate cancer: allelic loss at 16q24.1-q24.2 is associated with aggressive behaviour of the disease, recurrent growth, poor differentiation of the tumour and poor prognosis for the | Q43201271 | ||
Loss of chromosome 16q in lobular carcinoma in situ | Q46668779 | ||
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Does expression of different EWS chimeric transcripts define clinically distinct risk groups of Ewing tumor patients? | Q50752435 | ||
Prognostic significance of tumor volume in localized Ewing's sarcoma of bone in children and adolescents | Q52870795 | ||
EWS-FLI1 and EWS-ERG gene fusions are associated with similar clinical phenotypes in Ewing's sarcoma | Q54158232 | ||
Comparative genomic hybridization reveals recurrent enhancements on chromosome 20 and in one case combined amplification sites on 15q24q26 and 20p11p12 in glioblastomas. | Q55474980 | ||
Characterization of regions of chromosomes 12 and 16 involved in nephroblastoma tumorigenesis | Q57252309 | ||
Clinical significance of mutator phenotype and chromosome 17p and 18q allelic loss in gastric cancer | Q57426147 | ||
The Ewing Family of Tumors -- A Subgroup of Small-Round-Cell Tumors Defined by Specific Chimeric Transcripts | Q57756648 | ||
Fusion of an ETS-family gene, EIAF, to EWS by t(17;22)(q12;q12) chromosome translocation in an undifferentiated sarcoma of infancy | Q64382940 | ||
Chromosomes in Ewing's sarcoma. II. Nonrandom additional changes, trisomy 8 and der(16)t(1;16) | Q67920396 | ||
A second nonrandom translocation, der(16)t(1;16)(q21;q13), in Ewing sarcoma and peripheral neuroectodermal tumor | Q68467794 | ||
Morphological grades of regression in osteosarcoma after polychemotherapy - study COSS 80 | Q70911486 | ||
Gains and losses of DNA sequences in liposarcomas evaluated by comparative genomic hybridization | Q71598960 | ||
Clinical correlations of genetic changes by comparative genomic hybridization in Ewing sarcoma and related tumors | Q73094960 | ||
Clinical significance of genetic imbalances revealed by comparative genomic hybridization in chondrosarcomas | Q73114191 | ||
Frequent gain of copy number on the long arm of chromosome 20 in human pancreatic adenocarcinoma | Q73501765 | ||
16q loss of heterozygosity and microsatellite instability in Wilms' tumor | Q73937460 | ||
Recurrent chromosome alterations in hepatocellular carcinoma detected by comparative genomic hybridization | Q74223875 | ||
P433 | issue | 2 | |
P304 | page(s) | 164-171 | |
P577 | publication date | 2001-10-01 | |
P1433 | published in | Genes, Chromosomes and Cancer | Q5532697 |
P1476 | title | Genetic imbalances revealed by comparative genomic hybridization in Ewing tumors | |
P478 | volume | 32 |
Q39493814 | 1q gain and CDT2 overexpression underlie an aggressive and highly proliferative form of Ewing sarcoma |
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Q53520709 | Array CGH and gene-expression profiling reveals distinct genomic instability patterns associated with DNA repair and cell-cycle checkpoint pathways in Ewing's sarcoma |
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Q38003799 | Chromosomal anomalies and prognostic markers for intracranial and spinal ependymomas |
Q37078506 | Combined use of expression and CGH arrays pinpoints novel candidate genes in Ewing sarcoma family of tumors |
Q34700651 | Copy Number Alterations and Methylation in Ewing's Sarcoma |
Q36585123 | Current concepts in the molecular genetics of pediatric brain tumors: implications for emerging therapies |
Q28085071 | Diagnosis and treatment of Ewing sarcoma of the bone: a review article |
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Q37980482 | Ewing Sarcoma: Clinical State-of-the-Art |
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Q35748208 | Genomic imbalances in pediatric intracranial ependymomas define clinically relevant groups |
Q34691553 | Genomic landscape of Ewing sarcoma defines an aggressive subtype with co-association of STAG2 and TP53 mutations |
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Q38209599 | Tumor protein D52 (TPD52) and cancer-oncogene understudy or understudied oncogene? |
Q26782649 | Twenty Years on: What Do We Really Know about Ewing Sarcoma and What Is the Path Forward? |
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