Cre recombinase-mediated inactivation of H-2Dd transgene expression: evidence for partial missing self-recognition by Ly49A NK cells

scientific article published on 01 December 2001

Cre recombinase-mediated inactivation of H-2Dd transgene expression: evidence for partial missing self-recognition by Ly49A NK cells is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.4049/JIMMUNOL.167.11.6256
P698PubMed publication ID11714788

P2093author name stringJ Zimmer
F Beermann
W Held
V Ioannidis
P433issue11
P407language of work or nameEnglishQ1860
P304page(s)6256-6262
P577publication date2001-12-01
P1433published inJournal of ImmunologyQ3521441
P1476titleCre recombinase-mediated inactivation of H-2Dd transgene expression: evidence for partial missing self-recognition by Ly49A NK cells
P478volume167

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cites work (P2860)
Q35982359Activation by SLAM Family Receptors Contributes to NK Cell Mediated "Missing-Self" Recognition
Q40967373Cell-Extrinsic MHC Class I Molecule Engagement Augments Human NK Cell Education Programmed by Cell-Intrinsic MHC Class I.
Q40587952Cis association of Ly49A with MHC class I restricts natural killer cell inhibition
Q37259094Deciphering the function of canonical Wnt signals in development and disease: conditional loss- and gain-of-function mutations of beta-catenin in mice
Q36370106H-2D ligand expression by Ly49A+ natural killer (NK) cells precludes ligand uptake from environmental cells: implications for NK cell function
Q90594589Inducible down-regulation of MHC class I results in natural killer cell tolerance
Q36370699Ligand-dependent inhibition of CD1d-restricted NKT cell development in mice transgenic for the activating receptor Ly49D.
Q47265762Low number of H-2Dd-negative haematopoietic cells in mixed bone marrow chimeras convey in vivo tolerance to H-2Dd-negative cells but fail to prevent resistance to H-2Dd-negative leukaemia
Q36569520NLRC5 shields T lymphocytes from NK-cell-mediated elimination under inflammatory conditions
Q34087767Natural killer cell mediated missing-self recognition can protect mice from primary chronic myeloid leukemia in vivo
Q37397516Natural killer cell tolerance licensing and other mechanisms.
Q37616324Natural killer cells: tolerance to self and innate immunity to viral infection and malignancy
Q33474773Probing natural killer cell education by Ly49 receptor expression analysis and computational modelling in single MHC class I mice
Q36014390Regulation of natural killer cell function: a role for the NK cell's own MHC class I molecules
Q35676745Stable masking by H-2Dd cis ligand limits Ly49A relocalization to the site of NK cell/target cell contact
Q35084300The NK cell receptor repertoire: formation, adaptation and exploitation
Q37315659Tolerance and reactivity of NK cells: two sides of the same coin?

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