scholarly article | Q13442814 |
P50 | author | Richard J Di Paolo | Q98222229 |
P2093 | author name string | E. R. Unanue | |
P2860 | cites work | Class I–restricted Cross-Presentation of Exogenous Self-Antigens Leads to Deletion of Autoreactive CD8+ T Cells | Q24653129 |
Crystal Structure of I-Ak in Complex with a Dominant Epitope of Lysozyme | Q27749020 | ||
Mice expressing both B7-1 and viral glycoprotein on pancreatic beta cells along with glycoprotein-specific transgenic T cells develop diabetes due to a breakdown of T-lymphocyte unresponsiveness | Q35157542 | ||
Hindering auxiliary anchors are potent modulators of peptide binding and selection by I-Ak class II molecules | Q35300270 | ||
Resting and anergic B cells are defective in CD28-dependent costimulation of naive CD4+ T cells. | Q36363106 | ||
Constitutive class I-restricted exogenous presentation of self antigens in vivo | Q36367249 | ||
Initiation of autoimmune diabetes by developmentally regulated presentation of islet cell antigens in the pancreatic lymph nodes | Q36367942 | ||
Cross-tolerance: A Pathway for Inducing Tolerance to Peripheral Tissue Antigens | Q36400803 | ||
CD4+ T Cell Tolerance to Parenchymal Self-Antigens Requires Presentation by Bone Marrow–derived Antigen-presenting Cells | Q36400881 | ||
Ontogeny of T cell tolerance to peripherally expressed antigens | Q36453129 | ||
CD8 T cell ignorance or tolerance to islet antigens depends on antigen dose | Q36552144 | ||
Identification of the naturally processed form of hen egg white lysozyme bound to the murine major histocompatibility complex class II molecule I-Ak | Q37147375 | ||
Focal expression of interleukin-2 does not break unresponsiveness to "self" (viral) antigen expressed in beta cells but enhances development of autoimmune disease (diabetes) after initiation of an anti-self immune response | Q40341897 | ||
Limited capacity for tolerization of CD4+ T cells specific for a pancreatic beta cell neo-antigen. | Q41337228 | ||
Heritable formation of pancreatic beta-cell tumours in transgenic mice expressing recombinant insulin/simian virus 40 oncogenes | Q43470250 | ||
Virus infection triggers insulin-dependent diabetes mellitus in a transgenic model: role of anti-self (virus) immune response | Q43986613 | ||
How virus induces a rapid or slow onset insulin-dependent diabetes mellitus in a transgenic model | Q45783598 | ||
Ablation of "tolerance" and induction of diabetes by virus infection in viral antigen transgenic mice | Q45854348 | ||
Coexpression of B7-1 and viral ("self") transgenes in pancreatic beta cells can break peripheral ignorance and lead to spontaneous autoimmune diabetes | Q46495737 | ||
A range of CD4 T cell tolerance: partial inactivation to organ-specific antigen allows nondestructive thyroiditis or insulitis. | Q46650203 | ||
Characterization and quantitation of peptide-MHC complexes produced from hen egg lysozyme using a monoclonal antibody | Q52827100 | ||
The threshold for autoimmune T cell killing is influenced by B7-1 | Q57228048 | ||
Peripheral tolerance of CD4 T cells following local activation in adolescent mice | Q57258533 | ||
Peripheral tolerance to an islet cell-specific hemagglutinin transgene affects both CD4+ and CD8+ T cells | Q61140647 | ||
On the various manifestations of spontaneous autoimmune diabetes in rodent models | Q71665322 | ||
Very-low-dose streptozotocin induces diabetes in insulin promoter-mB7-1 transgenic mice | Q71698039 | ||
A role for non-MHC genetic polymorphism in susceptibility to spontaneous autoimmunity | Q72628270 | ||
Autoimmune insulitis and diabetes in the absence of antigen-specific contact between T cells and islet beta-cells | Q73129283 | ||
Quantitative analysis of the T cell repertoire that escapes negative selection | Q73152040 | ||
Quantitation of lysozyme peptides bound to class II MHC molecules indicates very large differences in levels of presentation | Q73785346 | ||
Inhibition of thymocyte positive selection by natural MHC: peptide ligands | Q74578783 | ||
Local expression of TNFalpha in neonatal NOD mice promotes diabetes by enhancing presentation of islet antigens | Q77658430 | ||
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | antigen presentation | Q575296 |
P304 | page(s) | 3453-3459 | |
P577 | publication date | 2001-12-01 | |
P1433 | published in | European Journal of Immunology | Q5412727 |
P1476 | title | The level of peptide-MHC complex determines the susceptibility to autoimmune diabetes: studies in HEL transgenic mice | |
P478 | volume | 31 |
Q57244914 | Aire mediates thymic expression and tolerance of pancreatic antigens via an unconventional transcriptional mechanism |
Q37969489 | Antigen presentation events in autoimmune diabetes. |
Q36232748 | Antigen presentation: lysoyme, autoimmune diabetes, and Listeria--what do they have in common? |
Q37267230 | Availability of autoantigenic epitopes controls phenotype, severity, and penetrance in TCR Tg autoimmune gastritis |
Q34792432 | Perspective on antigen processing and presentation. |
Q33986306 | The central role of antigen presentation in islets of Langerhans in autoimmune diabetes |
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