Exercise intolerance in patients with chronic heart failure and increased expression of inducible nitric oxide synthase in the skeletal muscle

scientific article published on 01 January 1999

Exercise intolerance in patients with chronic heart failure and increased expression of inducible nitric oxide synthase in the skeletal muscle is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/S0735-1097(98)00531-2
P698PubMed publication ID9935026

P2093author name stringH Jiang
J Yu
V Adams
G Schuler
R Hambrecht
A Linke
J Niebauer
S Gielen
S Möbius-Winkler
E Fiehn
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Impaired skeletal muscle function in patients with congestive heart failure. Relationship to systemic exercise performanceQ34216442
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Nitric oxide inhibits creatine kinase and regulates rat heart contractile reserveQ37593617
Nitric oxide: a physiologic messenger moleculeQ40394921
Nitric oxide: pathophysiological mechanismsQ40401521
Further characterization of contact sites from mitochondria of different tissues: topology of peripheral kinasesQ44764742
Retinoic acid accelerates embryonic stem cell-derived cardiac differentiation and enhances development of ventricular cardiomyocytesQ48691448
Skeletal muscle metabolism in patients with congestive heart failure: relation to clinical severity and blood flow.Q54397479
Exercise intolerance in patients with chronic heart failure: role of impaired nutritive flow to skeletal muscle.Q54473374
Lack of correlation between exercise capacity and indexes of resting left ventricular performance in heart failure.Q54540114
Alterations of skeletal muscle in chronic heart failureQ67487930
Contribution of skeletal muscle atrophy to exercise intolerance and altered muscle metabolism in heart failureQ68084268
Skeletal muscle metabolism during exercise under ischemic conditions in congestive heart failure. Evidence for abnormalities unrelated to blood flowQ68448350
Skeletal muscle biochemistry and histology in ambulatory patients with long-term heart failureQ68698026
Abnormal skeletal muscle bioenergetics during exercise in patients with heart failure: role of reduced muscle blood flowQ69485923
Anaerobic threshold and respiratory gas exchange during exerciseQ69558761
Contribution of intrinsic skeletal muscle changes to 31P NMR skeletal muscle metabolic abnormalities in patients with chronic heart failureQ69778142
Exertional fatigue due to skeletal muscle dysfunction in patients with heart failureQ70541290
Inhibition of creatine kinase by S-nitrosoglutathioneQ71448124
Stability of hexokinase II in vitro and in ascites tumor cellsQ71859372
Physical training in patients with stable chronic heart failure: effects on cardiorespiratory fitness and ultrastructural abnormalities of leg musclesQ72168544
Reversible inhibition of cytochrome c oxidase, the terminal enzyme of the mitochondrial respiratory chain, by nitric oxide. Implications for neurodegenerative diseasesQ72462338
Effects of endurance training on mitochondrial ultrastructure and fiber type distribution in skeletal muscle of patients with stable chronic heart failureQ73270987
Increased inducible nitric oxide synthase in skeletal muscle biopsies from patients with chronic heart failureQ73593462
P433issue1
P407language of work or nameEnglishQ1860
P921main subjectheart failureQ181754
chronic heart failureQ11829287
P304page(s)174-179
P577publication date1999-01-01
P1433published inJournal of the American College of CardiologyQ2984355
P1476titleExercise intolerance in patients with chronic heart failure and increased expression of inducible nitric oxide synthase in the skeletal muscle
P478volume33

Reverse relations

cites work (P2860)
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Q37425912Chronic heart failure and exercise intolerance: the hemodynamic paradox
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Q61776485Differential effects of exercise training in men and women with chronic heart failure
Q84353987Effects of chronic heart failure on neuronal nitric oxide synthase-mediated control of microvascular O2 pressure in contracting rat skeletal muscle
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