RAD51 and XRCC3 polymorphisms: impact on the risk and treatment outcomes of de novo inv(16) or t(16;16)/CBFβ-MYH11(+) acute myeloid leukemia

scientific article published on 05 February 2011

RAD51 and XRCC3 polymorphisms: impact on the risk and treatment outcomes of de novo inv(16) or t(16;16)/CBFβ-MYH11(+) acute myeloid leukemia is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.LEUKRES.2011.01.014
P698PubMed publication ID21296419

P2093author name stringLin Yang
Yue Zhang
Jianyong Li
Liang Liu
Jianxiang Wang
Xiaotang Ma
Yingchang Mi
Zefeng Xu
Zhijian Xiao
Tiejun Qin
P2860cites workGenomic acute myeloid leukemia-associated inv(16)(p13q22) breakpoints are tightly clusteredQ47991148
P433issue8
P921main subjectacute myeloid leukemiaQ264118
P304page(s)1020-1026
P577publication date2011-02-05
P1433published inLeukemia ResearchQ15716445
P1476titleRAD51 and XRCC3 polymorphisms: impact on the risk and treatment outcomes of de novo inv(16) or t(16;16)/CBFβ-MYH11(+) acute myeloid leukemia
P478volume35

Reverse relations

cites work (P2860)
Q34990550Association between the RAD51 135 G>C polymorphism and risk of cancer: a meta-analysis of 19,068 cases and 22,630 controls
Q38403345Comprehensive assessment of the association between DNA repair gene XRCC3 Thr241Met polymorphism and leukemia risk
Q38238954DNA damage accumulation and repair defects in acute myeloid leukemia: implications for pathogenesis, disease progression, and chemotherapy resistance
Q43749889Genetic 135G/C polymorphism of RAD51 gene and risk of cancer: a meta-analysis of 28,956 cases and 28,372 controls
Q37562135RAD51 Gene 135G/C polymorphism and the risk of four types of common cancers: a meta-analysis
Q37521105Relationship between Rad51 G135C and G172T variants and the susceptibility to cancer: a meta-analysis involving 54 case-control studies
Q39320029The Thr241Met polymorphism in the XRCC3 gene is associated with increased risk of cancer in Chinese mainland populations
Q38268221Therapeutic targeting the cell division cycle 25 (CDC25) phosphatases in human acute myeloid leukemia--the possibility to target several kinases through inhibition of the various CDC25 isoforms.

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