| scholarly article | Q13442814 |
| P50 | author | Carole Torsney | Q73193392 |
| P2860 | cites work | Analysis of fluorescently labeled substance P analogs: binding, imaging and receptor activation | Q24799998 |
| Can we conquer pain? | Q28211527 | ||
| Synaptic plasticity in spinal lamina I projection neurons that mediate hyperalgesia | Q28582696 | ||
| Cellular and Molecular Mechanisms of Pain | Q29011961 | ||
| Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons | Q33305201 | ||
| Injury-induced mechanical hypersensitivity requires C-low threshold mechanoreceptors | Q33605530 | ||
| Silent synapses and the emergence of a postsynaptic mechanism for LTP. | Q33641298 | ||
| Speaking out of turn: a role for silent synapses in pain | Q33847974 | ||
| Multiple mechanisms of secondary hyperalgesia | Q34094087 | ||
| Evidence for a central component of post-injury pain hypersensitivity | Q34265604 | ||
| Evidence for silent synapses: Implications for the expression of LTP | Q34303011 | ||
| Activation of postsynaptically silent synapses during pairing-induced LTP in CA1 region of hippocampal slice | Q34311517 | ||
| Synaptic amplifier of inflammatory pain in the spinal dorsal horn | Q34538726 | ||
| A plethora of painful molecules | Q35867589 | ||
| Transformation of the output of spinal lamina I neurons after nerve injury and microglia stimulation underlying neuropathic pain | Q36168166 | ||
| Spinal dis-inhibition in inflammatory pain | Q37136246 | ||
| Central sensitization: a generator of pain hypersensitivity by central neural plasticity | Q37361185 | ||
| Evidence against AMPA receptor-lacking glutamatergic synapses in the superficial dorsal horn of the rat spinal cord. | Q37424758 | ||
| Expansion of receptive fields of spinal lamina I projection neurons in rats with unilateral adjuvant-induced inflammation: the contribution of dorsal horn mechanisms | Q38662652 | ||
| The presence of ineffective synapses and the circumstances which unmask them | Q39162004 | ||
| A rapamycin-sensitive signaling pathway is essential for the full expression of persistent pain states. | Q39332259 | ||
| Nociceptor modulated central sensitization causes mechanical hyperalgesia in acute chemogenic and chronic neuropathic pain | Q39484232 | ||
| A quantitative study of spinothalamic neurons in laminae I, III, and IV in lumbar and cervical segments of the rat spinal cord | Q41814661 | ||
| Molecular depletion of descending serotonin unmasks its novel facilitatory role in the development of persistent pain | Q41856863 | ||
| Soma size distinguishes projection neurons from neurokinin 1 receptor-expressing interneurons in lamina I of the rat lumbar spinal dorsal horn | Q41957276 | ||
| Descending serotonergic controls regulate inflammation-induced mechanical sensitivity and methyl-CpG-binding protein 2 phosphorylation in the rat superficial dorsal horn | Q42953386 | ||
| Spinal neurons that possess the substance P receptor are required for the development of central sensitization. | Q44186001 | ||
| Superficial NK1-expressing neurons control spinal excitability through activation of descending pathways | Q44195579 | ||
| Localization and function of ATP and GABAA receptors expressed by nociceptors and other postnatal sensory neurons in rat. | Q44383098 | ||
| Neurokinin receptor 1-expressing spinal cord neurons in lamina I and III/IV of postnatal rats receive inputs from capsaicin sensitive fibers | Q44660691 | ||
| Neurogenic hyperalgesia: psychophysical studies of underlying mechanisms | Q45020805 | ||
| Spinal neurons that express NK-1 receptors modulate descending controls that project through the dorsolateral funiculus | Q45084899 | ||
| The role of heterosynaptic facilitation in long-term potentiation (LTP) of human pain sensation | Q45241530 | ||
| Morphological characterization of substance P receptor‐immunoreactive neurons in the rat spinal cord and trigeminal nucleus caudalis | Q46090686 | ||
| Glycine inhibitory dysfunction induces a selectively dynamic, morphine-resistant, and neurokinin 1 receptor- independent mechanical allodynia. | Q46108378 | ||
| Inflammation increases the distribution of dorsal horn neurons that internalize the neurokinin-1 receptor in response to noxious and non-noxious stimulation. | Q46304913 | ||
| Translocation of GluR1-containing AMPA receptors to a spinal nociceptive synapse during acute noxious stimulation. | Q46492778 | ||
| N-ethylmaleimide-sensitive fusion protein (NSF) is involved in central sensitization in the spinal cord through GluR2 subunit composition switch after inflammation | Q46504263 | ||
| Long-term potentiation of silent synapses in substantia gelatinosa neurons | Q46523574 | ||
| Silent synapses during development of thalamocortical inputs | Q46721431 | ||
| Switch to Ca2+-permeable AMPA and reduced NR2B NMDA receptor-mediated neurotransmission at dorsal horn nociceptive synapses during inflammatory pain in the rat. | Q46885932 | ||
| Disinhibition opens the gate to pathological pain signaling in superficial neurokinin 1 receptor-expressing neurons in rat spinal cord. | Q46935411 | ||
| NMDA EPSCs at glutamatergic synapses in the spinal cord dorsal horn of the postnatal rat. | Q47710193 | ||
| Silent glutamatergic synapses and nociception in mammalian spinal cord | Q47924223 | ||
| A quantitative study of neurons which express neurokinin-1 or somatostatin sst2a receptor in rat spinal dorsal horn | Q47987422 | ||
| Central sensitization versus synaptic long-term potentiation (LTP): a critical comment | Q48104498 | ||
| Distinctive membrane and discharge properties of rat spinal lamina I projection neurones in vitro | Q48107053 | ||
| A quantitative and morphological study of projection neurons in lamina I of the rat lumbar spinal cord. | Q48144059 | ||
| State-dependent mechanisms of LTP expression revealed by optical quantal analysis | Q48365227 | ||
| Silent NMDA receptor-mediated synapses are developmentally regulated in the dorsal horn of the rat spinal cord. | Q51419254 | ||
| Peripheral inflammation facilitates Abeta fiber-mediated synaptic input to the substantia gelatinosa of the adult rat spinal cord. | Q51482181 | ||
| A role for transcriptional repressor methyl-CpG-binding protein 2 and plasticity-related gene serum- and glucocorticoid-inducible kinase 1 in the induction of inflammatory pain states. | Q51983867 | ||
| Long-term potentiation and functional synapse induction in developing hippocampus. | Q52048082 | ||
| Age-dependent effects of peripheral inflammation on the electrophysiological properties of neonatal rat dorsal horn neurons. | Q52123070 | ||
| Alteration in synaptic inputs through C-afferent fibers to substantia gelatinosa neurons of the rat spinal dorsal horn during postnatal development. | Q52164424 | ||
| Selective ablation of dorsal horn NK1 expressing cells reveals a modulation of spinal alpha2-adrenergic inhibition of dorsal horn neurones. | Q52586782 | ||
| The Cell and Molecular Basis of Mechanical, Cold, and Inflammatory Pain | Q57771834 | ||
| AMPA receptor–PDZ interactions in facilitation of spinal sensory synapses | Q58066146 | ||
| Extrasynaptic Glutamate Spillover in the Hippocampus: Dependence on Temperature and the Role of Active Glutamate Uptake | Q60243628 | ||
| The correlation between the distribution of the NK1 receptor and the actions of tachykinin agonists in the dorsal horn of the rat indicates that substance P does not have a functional role on substantia gelatinosa (lamina II) neurons | Q71662662 | ||
| Transmission of chronic nociception by spinal neurons expressing the substance P receptor | Q73195551 | ||
| Neurokinin 1 receptor expression by neurons in laminae I, III and IV of the rat spinal dorsal horn that project to the brainstem | Q73537433 | ||
| Spinal lamina I neurons that express neurokinin 1 receptors: morphological analysis | Q73755576 | ||
| Inhibition of hyperalgesia by ablation of lamina I spinal neurons expressing the substance P receptor | Q73763175 | ||
| Activation of NMDA receptors drives action potentials in superficial dorsal horn from neonatal rats | Q73892213 | ||
| Relationship of substance P to afferent characteristics of dorsal root ganglion neurones in guinea-pig | Q73966403 | ||
| Primary afferent fibers that contribute to increased substance P receptor internalization in the spinal cord after injury | Q74620741 | ||
| Plastic changes in sensory inputs to rat substantia gelatinosa neurons following peripheral inflammation | Q78048328 | ||
| Spinal substance P receptor expression and internalization in acute, short-term, and long-term inflammatory pain states | Q78166434 | ||
| Inflammation reduces the contribution of N-type calcium channels to primary afferent synaptic transmission onto NK1 receptor-positive lamina I neurons in the rat dorsal horn | Q79782359 | ||
| Both Ca2+-permeable and -impermeable AMPA receptors contribute to primary synaptic drive onto rat dorsal horn neurons | Q83954341 | ||
| Synaptic plasticity and central sensitization: author reply | Q84625824 | ||
| Neurokinin-1 receptors on lumbar spinothalamic neurons in the rat | Q95825658 | ||
| P433 | issue | 13 | |
| P304 | page(s) | 5158-5168 | |
| P577 | publication date | 2011-03-01 | |
| P1433 | published in | Journal of Neuroscience | Q1709864 |
| P1476 | title | Inflammatory pain unmasks heterosynaptic facilitation in lamina I neurokinin 1 receptor-expressing neurons in rat spinal cord | |
| P478 | volume | 31 |
| Q36574960 | A combined electrophysiological and morphological study of neuropeptide Y-expressing inhibitory interneurons in the spinal dorsal horn of the mouse |
| Q38160667 | AMPA-silent synapses in brain development and pathology |
| Q36867757 | Adaptations in responsiveness of brainstem pain-modulating neurons in acute compared with chronic inflammation |
| Q33811673 | Delta opioid receptors presynaptically regulate cutaneous mechanosensory neuron input to the spinal cord dorsal horn |
| Q35994933 | Developmental regulation of membrane excitability in rat spinal lamina I projection neurons |
| Q34954885 | Diminished neurokinin-1 receptor availability in patients with two forms of chronic visceral pain |
| Q38250226 | Endogenous analgesia, dependence, and latent pain sensitization |
| Q37419055 | Gastrodin protects against chronic inflammatory pain by inhibiting spinal synaptic potentiation |
| Q37955352 | Hyperalgesia by synaptic long-term potentiation (LTP): an update |
| Q35121657 | Increased local concentration of complement C5a contributes to incisional pain in mice. |
| Q41024387 | Inflammatory Pain Reduces C Fiber Activity-Dependent Slowing in a Sex-Dependent Manner, Amplifying Nociceptive Input to the Spinal Cord. |
| Q35598747 | Inhibitory Interneurons That Express GFP in the PrP-GFP Mouse Spinal Cord Are Morphologically Heterogeneous, Innervated by Several Classes of Primary Afferent and Include Lamina I Projection Neurons among Their Postsynaptic Targets |
| Q38694326 | Melatonin limits paclitaxel-induced mitochondrial dysfunction in vitro and protects against paclitaxel-induced neuropathic pain in the rat. |
| Q57096939 | Neurochemical characterization of extrinsic nerves in myenteric ganglia of the guinea pig distal colon |
| Q36052397 | Nociception and inflammatory hyperalgesia evaluated in rodents using infrared laser stimulation after Trpv1 gene knockout or resiniferatoxin lesion |
| Q36391184 | Persistent changes in peripheral and spinal nociceptive processing after early tissue injury |
| Q57284897 | Presynaptic Inhibition of Primary Nociceptive Signals to Dorsal Horn Lamina I Neurons by Dopamine |
| Q43043396 | Selective innervation of NK1 receptor-lacking lamina I spinoparabrachial neurons by presumed nonpeptidergic Aδ nociceptors in the rat. |
| Q87028823 | Slit2/Robo1 Mediation of Synaptic Plasticity Contributes to Bone Cancer Pain |
| Q34037539 | Synaptic GluN2A and GluN2B containing NMDA receptors within the superficial dorsal horn activated following primary afferent stimulation |
| Q33622717 | The chemerin receptor 23 agonist, chemerin, attenuates monosynaptic C-fibre input to lamina I neurokinin 1 receptor expressing rat spinal cord neurons in inflammatory pain |
| Q41853439 | The control of alternative splicing by SRSF1 in myelinated afferents contributes to the development of neuropathic pain. |
| Q36160142 | Transient, activity dependent inhibition of transmitter release from low threshold afferents mediated by GABAA receptors in spinal cord lamina III/IV. |
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