| human | Q5 |
| P269 | IdRef ID | 262625598 |
| P496 | ORCID iD | 0000-0002-6807-9136 |
| P214 | VIAF ID | 9163208050407230624 |
| P108 | employer | University of Massachusetts Amherst | Q15142 |
| P735 | given name | Laura | Q429948 |
| Laura | Q429948 | ||
| P106 | occupation | researcher | Q1650915 |
| P21 | sex or gender | female | Q6581072 |
| Q34981379 | A path forward in the debate over health impacts of endocrine disrupting chemicals |
| Q28384100 | A proposed framework for the systematic review and integrated assessment (SYRINA) of endocrine disrupting chemicals |
| Q33789200 | A round robin approach to the analysis of bisphenol A (BPA) in human blood samples. |
| Q27008303 | A unified model for left-right asymmetry? Comparison and synthesis of molecular models of embryonic laterality |
| Q126893981 | A vision for safer food contact materials: Public health concerns as drivers for improved testing |
| Q35632704 | Assessing dose-response relationships for endocrine disrupting chemicals (EDCs): a focus on non-monotonicity |
| Q104797085 | Assessing the Public Health Implications of the Food Preservative Propylparaben: Has This Chemical Been Safely Used for Decades |
| Q57479027 | Asymmetric development of the male mouse mammary gland and its response to a prenatal or postnatal estrogen challenge |
| Q34060021 | Biomonitoring studies should be used by regulatory agencies to assess human exposure levels and safety of bisphenol A. |
| Q47911003 | Bisphenol S (BPS) Alters Maternal Behavior and Brain in Mice Exposed During Pregnancy/Lactation and Their Daughters |
| Q47620151 | Bisphenol S Alters the Lactating Mammary Gland and Nursing Behaviors in Mice Exposed During Pregnancy and Lactation |
| Q92765176 | Bisphenol S alters development of the male mouse mammary gland and sensitizes it to a peripubertal estrogen challenge |
| Q24648358 | Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption |
| Q91397826 | CLARITY-BPA academic laboratory studies identify consistent low-dose Bisphenol A effects on multiple organ systems |
| Q52651241 | Casting a wide net for endocrine disruptors. |
| Q24619643 | Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure |
| Q33569410 | Chlorinated persistent organic pollutants, obesity, and type 2 diabetes |
| Q36588156 | Concerns over use of glyphosate-based herbicides and risks associated with exposures: a consensus statement |
| Q126598349 | Conflicts of Interest in the Assessment of Chemicals, Waste, and Pollution |
| Q91260639 | Consensus on the key characteristics of endocrine-disrupting chemicals as a basis for hazard identification |
| Q39513045 | Consistent left-right asymmetry cannot be established by late organizers in Xenopus unless the late organizer is a conjoined twin |
| Q93066320 | Data describing effects of perinatal exposure to bisphenol S on a peripubertal estrogen challenge in intact female CD-1 mice |
| Q38631226 | Data describing lack of effects of 17α-ethinyl estradiol on mammary gland morphology in female mice exposed during pregnancy and lactation |
| Q47309263 | Developmental estrogen exposures and disruptions to maternal behavior and brain: Effects of ethinyl estradiol, a common positive control |
| Q64917373 | Developmental exposures to bisphenol S, a BPA replacement, alter estrogen-responsiveness of the female reproductive tract: a pilot study. |
| Q28385743 | Developmental origins of health and disease: a paradigm for understanding disease cause and prevention |
| Q91604995 | Distract, delay, disrupt: examples of manufactured doubt from five industries |
| Q24652548 | Does breast cancer start in the womb? |
| Q92655078 | Effects of Benzophenone-3 and Propylparaben on Estrogen Receptor-Dependent R-Loops and DNA Damage in Breast Epithelial Cells and Mice |
| Q46320908 | Endocrine Disruptors and Health Effects in Africa: A Call for Action. |
| Q35913778 | Endocrine disruptors alter social behaviors and indirectly influence social hierarchies via changes in body weight. |
| Q92179827 | Endocrine disruptors and the future of toxicology testing - lessons from CLARITY-BPA |
| Q97686578 | Endocrine-disrupting chemicals: economic, regulatory, and policy implications |
| Q42664482 | Erratum to: A round robin approach to the analysis of bisphenol a (BPA) in human blood samples. |
| Q42685050 | Evidence of altered brain sexual differentiation in mice exposed perinatally to low, environmentally relevant levels of bisphenol A. |
| Q34166195 | Exposure to bisphenol A in Canada: invoking the precautionary principle |
| Q24650732 | Exposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary gland |
| Q92594856 | Exposure to low doses of oxybenzone during perinatal development alters mammary gland morphology in male and female mice |
| Q37804719 | Far from solved: A perspective on what we know about early mechanisms of left–right asymmetry |
| Q43158781 | Flawed experimental design reveals the need for guidelines requiring appropriate positive controls in endocrine disruption research. |
| Q55521896 | Genetic variation in sensitivity to estrogens and breast cancer risk. |
| Q33751821 | Handling of thermal paper: Implications for dermal exposure to bisphenol A and its alternatives |
| Q36845997 | High-throughput Xenopus laevis immunohistochemistry using agarose sections |
| Q24613799 | Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses |
| Q29547406 | Human exposure to bisphenol A (BPA) |
| Q34683215 | Human exposures to bisphenol A: mismatches between data and assumptions |
| Q89980688 | Impacts of food contact chemicals on human health: a consensus statement |
| Q30235180 | Is it time to reassess current safety standards for glyphosate-based herbicides? |
| Q37548748 | It's never too early to get it Right: A conserved role for the cytoskeleton in left-right asymmetry. |
| Q39330507 | Laterality defects are influenced by timing of treatments and animal model |
| Q41036743 | Left-right patterning in Xenopus conjoined twin embryos requires serotonin signaling and gap junctions |
| Q37540864 | Light-activated serotonin for exploring its action in biological systems |
| Q51734796 | Low dose bisphenol S or ethinyl estradiol exposures during the perinatal period alter female mouse mammary gland development. |
| Q47330026 | Low doses of 17α-ethinyl estradiol alter the maternal brain and induce stereotypies in CD-1 mice exposed during pregnancy and lactation. |
| Q30474571 | Low frequency vibrations disrupt left-right patterning in the Xenopus embryo |
| Q27330527 | Low frequency vibrations induce malformations in two aquatic species in a frequency-, waveform-, and direction-specific manner |
| Q48416133 | Low-dose effects of endocrine disruptors, with Laura Vandenberg. Interview by Ashley Ahearn |
| Q38175567 | Low-dose effects of hormones and endocrine disruptors |
| Q40663640 | Manufacturing doubt about endocrine disrupter science--A rebuttal of industry-sponsored critical comments on the UNEP/WHO report "State of the Science of Endocrine Disrupting Chemicals 2012". |
| Q29248099 | Metabolism Disrupting Chemicals and Metabolic Disorders |
| Q42062186 | Neurally Derived Tissues in Xenopus laevis Embryos Exhibit a Consistent Bioelectrical Left-Right Asymmetry |
| Q33676877 | Non-monotonic dose responses in studies of endocrine disrupting chemicals: bisphenol a as a case study |
| Q58743559 | Nonmonotonic Dose-Response Curves Occur in Dose Ranges That Are Relevant to Regulatory Decision-Making |
| Q41821452 | Normalized shape and location of perturbed craniofacial structures in the Xenopus tadpole reveal an innate ability to achieve correct morphology |
| Q59315750 | Normalized shape and location of perturbed craniofacial structures in theXenopustadpole reveal an innate ability to achieve correct morphology |
| Q91885531 | Obesogenic Effect of Sulfamethoxazole on Drosophila melanogaster with Simultaneous Disturbances on Eclosion Rhythm, Glucolipid Metabolism, and Microbiota |
| Q64983609 | Oxybenzone Alters Mammary Gland Morphology in Mice Exposed During Pregnancy and Lactation. |
| Q28559107 | Peer-reviewed and unbiased research, rather than 'sound science', should be used to evaluate endocrine-disrupting chemicals |
| Q35752320 | Perinatal bisphenol A exposure increases estrogen sensitivity of the mammary gland in diverse mouse strains. |
| Q37580672 | Perinatal exposure to the xenoestrogen bisphenol-A induces mammary intraductal hyperplasias in adult CD-1 mice |
| Q37351407 | Perspectives and open problems in the early phases of left-right patterning |
| Q42497393 | Polarity proteins are required for left-right axis orientation and twin-twin instruction |
| Q50098972 | Prenatal exposure to unconventional oil and gas operation chemical mixtures altered mammary gland development in adult female mice. |
| Q50762920 | Rab GTPases are required for early orientation of the left-right axis in Xenopus. |
| Q52676956 | Reform of the Toxic Substances Control Act (TSCA): An Endocrine Society Policy Perspective. |
| Q37515730 | Regulatory decisions on endocrine disrupting chemicals should be based on the principles of endocrinology |
| Q21245825 | Science and policy on endocrine disrupters must not be mixed: a reply to a "common sense" intervention by toxicology journal editors |
| Q36486059 | Serotonin has early, cilia-independent roles in Xenopus left-right patterning. |
| Q33801522 | Should oral gavage be abandoned in toxicity testing of endocrine disruptors? |
| Q92950281 | The Mouse Mammary Gland: a Tool to Inform Adolescents About Environmental Causes of Breast Cancer |
| Q52667524 | The Path Forward on Endocrine Disruptors Requires Focus on the Basics. |
| Q91130926 | The Use and Misuse of Historical Controls in Regulatory Toxicology: Lessons from the CLARITY-BPA Study |
| Q37721584 | The male mammary gland: a target for the xenoestrogen bisphenol A. |
| Q63521160 | The mammary gland response to estradiol: Monotonic at the cellular level, non-monotonic at the tissue-level of organization? |
| Q37692507 | The mouse mammary gland as a sentinel organ: distinguishing 'control' populations with diverse environmental histories |
| Q92189764 | The parental brain and behavior: A target for endocrine disruption |
| Q111323240 | The science of spin: targeted strategies to manufacture doubt with detrimental effects on environmental and public health |
| Q28258110 | Theodora (Theo) Colborn: 1927-2014 |
| Q92580205 | There are good clinical, scientific, and social reasons to strengthen links between biomedical and environmental research |
| Q37057471 | To Cull or Not To Cull? Considerations for Studies of Endocrine-Disrupting Chemicals |
| Q33756682 | Two cleavage products of the Drosophila accessory gland protein ovulin can independently induce ovulation |
| Q99610136 | Update on the health effects of bisphenol A: Overwhelming evidence of harm |
| Q28833444 | Uppsala Consensus Statement on Environmental Contaminants and the Global Obesity Epidemic |
| Q28382768 | Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A |
| Q37977848 | Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A. |
| Q28079859 | Using systematic reviews for hazard and risk assessment of endocrine disrupting chemicals |
| Q59315751 | V-ATPase-dependent ectodermal voltage and pH regionalization are required for Xenopus craniofacial morphogenesis |
| Q34200733 | V-ATPase-dependent ectodermal voltage and pH regionalization are required for craniofacial morphogenesis |
| Q24651547 | Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A |
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