scholarly article | Q13442814 |
P2093 | author name string | Thomas E Lloyd | |
Brian M Woolums | |||
James B Machamer | |||
Gregory G Fuller | |||
P2860 | cites work | The Presynaptic Microtubule Cytoskeleton in Physiological and Pathological Conditions: Lessons from Drosophila Fragile X Syndrome and Hereditary Spastic Paraplegias | Q26738451 |
Mechanisms of FUS mutations in familial amyotrophic lateral sclerosis | Q26749169 | ||
Pokes, sunburn, and hot sauce: Drosophila as an emerging model for the biology of nociception | Q26862727 | ||
FUS transgenic rats develop the phenotypes of amyotrophic lateral sclerosis and frontotemporal lobar degeneration | Q27342067 | ||
Optimization of a GCaMP Calcium Indicator for Neural Activity Imaging | Q27674109 | ||
Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis | Q28236796 | ||
Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6 | Q28236805 | ||
Role of axonal transport in neurodegenerative diseases | Q28284096 | ||
ALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the disease | Q28392297 | ||
Delocalization of the multifunctional RNA splicing factor TLS/FUS in hippocampal neurones: exclusion from the nucleus and accumulation in dendritic granules and spine heads | Q28582163 | ||
Dendritic organization in the neurons of the visual and motor cortices of the cat | Q29616515 | ||
Pickpocket is a DEG/ENaC protein required for mechanical nociception in Drosophila larvae. | Q30497522 | ||
Deficits in axonal transport precede ALS symptoms in vivo | Q30497554 | ||
Axonal transport deficits and degeneration can evolve independently in mouse models of amyotrophic lateral sclerosis. | Q30511133 | ||
The multifunctional FUS, EWS and TAF15 proto-oncoproteins show cell type-specific expression patterns and involvement in cell spreading and stress response | Q33351073 | ||
Motor neuron expression of the voltage-gated calcium channel cacophony restores locomotion defects in a Drosophila, TDP-43 loss of function model of ALS. | Q33651150 | ||
The ALS gene FUS regulates synaptic transmission at the Drosophila neuromuscular junction. | Q33784500 | ||
Branching out: mechanisms of dendritic arborization | Q34111001 | ||
Activity-dependent FUS dysregulation disrupts synaptic homeostasis | Q34480824 | ||
Male sterility and enhanced radiation sensitivity in TLS(-/-) mice | Q34662602 | ||
How does calcium interact with the cytoskeleton to regulate growth cone motility during axon pathfinding? | Q57938863 | ||
Axonal ion channel dysfunction in amyotrophic lateral sclerosis | Q71681845 | ||
Protein arginine methyltransferase 1 and 8 interact with FUS to modify its sub-cellular distribution and toxicity in vitro and in vivo | Q34689011 | ||
Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease | Q34689753 | ||
A Drosophila model of FUS-related neurodegeneration reveals genetic interaction between FUS and TDP-43. | Q34882752 | ||
Early changes of neuromuscular transmission in the SOD1(G93A) mice model of ALS start long before motor symptoms onset | Q34989403 | ||
ALS-associated FUS mutations result in compromised FUS alternative splicing and autoregulation | Q35034350 | ||
Pathogenic polyglutamine proteins cause dendrite defects associated with specific actin cytoskeletal alterations in Drosophila | Q35288983 | ||
The p150(Glued) CAP-Gly domain regulates initiation of retrograde transport at synaptic termini | Q35965342 | ||
Super-Resolution Microscopy Reveals Presynaptic Localization of the ALS/FTD Related Protein FUS in Hippocampal Neurons | Q36453024 | ||
Expression of Fused in sarcoma mutations in mice recapitulates the neuropathology of FUS proteinopathies and provides insight into disease pathogenesis | Q36459963 | ||
ALS-associated mutant FUS induces selective motor neuron degeneration through toxic gain of function | Q36548825 | ||
Expression of human FUS protein in Drosophila leads to progressive neurodegeneration | Q36584516 | ||
Stress granules as crucibles of ALS pathogenesis. | Q36804082 | ||
Establishment of In Vitro FUS-Associated Familial Amyotrophic Lateral Sclerosis Model Using Human Induced Pluripotent Stem Cells. | Q36805034 | ||
Synaptic regulation of microtubule dynamics in dendritic spines by calcium, F-actin, and drebrin | Q37231480 | ||
Gamma motor neurons survive and exacerbate alpha motor neuron degeneration in ALS. | Q37534101 | ||
ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defects | Q37609639 | ||
TDP-43 and FUS/TLS: emerging roles in RNA processing and neurodegeneration | Q37733168 | ||
The genetics and neuropathology of amyotrophic lateral sclerosis | Q38035744 | ||
Loss and gain of FUS function impair neuromuscular synaptic transmission in a genetic model of ALS. | Q38314794 | ||
Visualizing Axons in the Drosophila Central Nervous System Using Immunohistochemistry and Immunofluorescence: Figure 1 | Q38617419 | ||
The ALS-associated proteins FUS and TDP-43 function together to affect Drosophila locomotion and life span. | Q38732625 | ||
Overexpression of nuclear FUS induces neuronal cell death | Q38929777 | ||
PINK1 and Parkin are genetic modifiers for FUS-induced neurodegeneration. | Q39235670 | ||
Axonal transport defects are a common phenotype in Drosophila models of ALS. | Q39774347 | ||
Altered axonal excitability properties in amyotrophic lateral sclerosis: impaired potassium channel function related to disease stage | Q40351989 | ||
Interaction of FUS and HDAC1 regulates DNA damage response and repair in neurons. | Q41469956 | ||
Drosophila Futsch regulates synaptic microtubule organization and is necessary for synaptic growth | Q41739468 | ||
Nuclear import factor transportin and arginine methyltransferase 1 modify FUS neurotoxicity in Drosophila | Q41758682 | ||
FUS regulates AMPA receptor function and FTLD/ALS-associated behaviour via GluA1 mRNA stabilization | Q41950782 | ||
Drosophila larval NMJ immunohistochemistry | Q42058055 | ||
Genetic mutations in RNA-binding proteins and their roles in ALS. | Q42278719 | ||
Fused in sarcoma/translocated in liposarcoma: a multifunctional DNA/RNA binding protein. | Q43028561 | ||
Tiling of the Drosophila epidermis by multidendritic sensory neurons. | Q45960946 | ||
Ectopic and increased expression of Fasciclin II alters motoneuron growth cone guidance | Q46045692 | ||
FUS toxicity is rescued by the modulation of lncRNA hsrω expression in Drosophila melanogaster | Q46068056 | ||
Axonal excitability properties in amyotrophic lateral sclerosis | Q47202111 | ||
Mutations in the 3' untranslated region of FUS causing FUS overexpression are associated with amyotrophic lateral sclerosis. | Q48021519 | ||
Unraveling Synaptic GCaMP Signals: Differential Excitability and Clearance Mechanisms Underlying Distinct Ca2+ Dynamics in Tonic and Phasic Excitatory, and Aminergic Modulatory Motor Terminals in Drosophila | Q49724117 | ||
Motorneurons require cysteine string protein-α to maintain the readily releasable vesicular pool and synaptic vesicle recycling. | Q50499095 | ||
Fus deficiency in mice results in defective B-lymphocyte development and activation, high levels of chromosomal instability and perinatal death. | Q52537887 | ||
Nuclear transport impairment of amyotrophic lateral sclerosis-linked mutations in FUS/TLS. | Q52607544 | ||
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | Pt A | |
P1104 | number of pages | 12 | |
P304 | page(s) | 55-66 | |
P577 | publication date | 2018-04-03 | |
P1433 | published in | Brain Research | Q4955782 |
P1476 | title | FUS causes synaptic hyperexcitability in Drosophila dendritic arborization neurons | |
P478 | volume | 1693 |
Q58766511 | as a Model for Assessing the Function of RNA-Binding Proteins during Neurogenesis and Neurological Disease | cites work | P2860 |
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