review article | Q7318358 |
scholarly article | Q13442814 |
P50 | author | Ulrike C Lange | Q89669106 |
P2093 | author name string | Carine Van Lint | |
Roxane Verdikt | |||
Amina Ait-Ammar | |||
P2860 | cites work | DNA methylation patterns and epigenetic memory | Q22065780 |
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Longitudinal variation in human immunodeficiency virus long terminal repeat methylation in individuals on suppressive antiretroviral therapy | Q90098575 | ||
Resistance to the Tat Inhibitor Didehydro-Cortistatin A Is Mediated by Heightened Basal HIV-1 Transcription | Q91563978 | ||
Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing | Q24294385 | ||
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COUP-TF interacting protein 2 represses the initial phase of HIV-1 gene transcription in human microglial cells | Q24806175 | ||
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The site of HIV-1 integration in the human genome determines basal transcriptional activity and response to Tat transactivation | Q28361880 | ||
Epigenetic regulation of HIV-1 latency by cytosine methylation | Q28475636 | ||
Non-catalytic site HIV-1 integrase inhibitors disrupt core maturation and induce a reverse transcription block in target cells | Q28536951 | ||
Perceptions of epigenetics | Q29547310 | ||
HIV-1 integration in the human genome favors active genes and local hotspots | Q29618457 | ||
HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation | Q29619611 | ||
Stochastic gene expression in a lentiviral positive-feedback loop: HIV-1 Tat fluctuations drive phenotypic diversity | Q33220546 | ||
Interfering RNA and HIV: reciprocal interferences | Q33372109 | ||
CpG methylation controls reactivation of HIV from latency | Q33495565 | ||
Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. | Q33604848 | ||
An HIV-encoded antisense long noncoding RNA epigenetically regulates viral transcription | Q33724213 | ||
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Infection with Human Immunodeficiency Virus Type 1 Upregulates DNA Methyltransferase, Resulting in De Novo Methylation of the Gamma Interferon (IFN-γ) Promoter and Subsequent Downregulation of IFN-γ Production | Q33777676 | ||
Infection of lymphoid cells by integration-defective human immunodeficiency virus type 1 increases de novo methylation | Q33846679 | ||
Involvement of histone H3 lysine 9 (H3K9) methyltransferase G9a in the maintenance of HIV-1 latency and its reactivation by BIX01294. | Q33883423 | ||
The Human Immunodeficiency Virus 1 ASP RNA promotes viral latency by recruiting the Polycomb Repressor Complex 2 and promoting nucleosome assembly | Q33893725 | ||
Immunologic strategies for HIV-1 remission and eradication | Q33896523 | ||
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Chromatin disruption in the promoter of human immunodeficiency virus type 1 during transcriptional activation. | Q34057748 | ||
Repressive LTR nucleosome positioning by the BAF complex is required for HIV latency | Q34090863 | ||
An analog of the natural steroidal alkaloid cortistatin A potently suppresses Tat-dependent HIV transcription | Q34289422 | ||
Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy | Q34290545 | ||
A proteomic glimpse into the initial global epigenetic changes during HIV infection | Q34344844 | ||
Nuclear architecture dictates HIV-1 integration site selection. | Q34465264 | ||
HIV latency. Proliferation of cells with HIV integrated into cancer genes contributes to persistent infection | Q34502840 | ||
The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit | Q34544409 | ||
Recurrent HIV-1 integration at the BACH2 locus in resting CD4+ T cell populations during effective highly active antiretroviral therapy | Q34607544 | ||
Towards an HIV cure: a global scientific strategy | Q34640562 | ||
HIV latency. Specific HIV integration sites are linked to clonal expansion and persistence of infected cells | Q34672568 | ||
DNA methylation and gene silencing in cancer: which is the guilty party? | Q34770507 | ||
Epigenetic heterogeneity in HIV-1 latency establishment | Q34877762 | ||
HIV reproducibly establishes a latent infection after acute infection of T cells in vitro. | Q34985945 | ||
Non-coding RNAs as regulators of gene expression and epigenetics. | Q34986526 | ||
Recruitment of the de novo DNA methyltransferase Dnmt3a by Kaposi's sarcoma-associated herpesvirus LANA | Q35080754 | ||
Viral Genes and Methylation | Q35118506 | ||
Accumulation of heterochromatin components on the terminal repeat sequence of Kaposi's sarcoma-associated herpesvirus mediated by the latency-associated nuclear antigen | Q35123088 | ||
Epigenetic silencing of HIV-1 by the histone H3 lysine 27 methyltransferase enhancer of Zeste 2. | Q35192959 | ||
HIV-1 integration landscape during latent and active infection | Q35211469 | ||
Regulation of HIV-1 gene transcription: from lymphocytes to microglial cells | Q35213780 | ||
Non-coding RNAs and HIV: viral manipulation of host dark matter to shape the cellular environment. | Q35219072 | ||
RNA-mediated epigenetic regulation of gene expression | Q35223649 | ||
Polarized expression of the membrane ASP protein derived from HIV-1 antisense transcription in T cells | Q35241307 | ||
A hardwired HIV latency program | Q35400385 | ||
Epigenetic modulations in activated cells early after HIV-1 infection and their possible functional consequences. | Q35601948 | ||
LSD1 cooperates with CTIP2 to promote HIV-1 transcriptional silencing | Q35823707 | ||
The Tat Inhibitor Didehydro-Cortistatin A Prevents HIV-1 Reactivation from Latency | Q35829282 | ||
HIV integration site selection: analysis by massively parallel pyrosequencing reveals association with epigenetic modifications | Q35914779 | ||
Paucity of HIV DNA methylation in latently infected, resting CD4+ T cells from infected individuals receiving antiretroviral therapy | Q35943698 | ||
HIV-1-encoded antisense RNA suppresses viral replication for a prolonged period | Q36136773 | ||
Proviruses with identical sequences comprise a large fraction of the replication-competent HIV reservoir | Q36318184 | ||
HIV latency: experimental systems and molecular models. | Q36585019 | ||
In vivo, in vitro, and in silico analysis of methylation of the HIV-1 provirus. | Q36592182 | ||
Development of 5' LTR DNA methylation of latent HIV-1 provirus in cell line models and in long-term-infected individuals | Q36596027 | ||
Clonally expanded CD4+ T cells can produce infectious HIV-1 in vivo | Q36607484 | ||
A Minor Subset of Super Elongation Complexes Plays a Predominant Role in Reversing HIV-1 Latency | Q36709227 | ||
HIV-1 and T cell dynamics after interruption of highly active antiretroviral therapy (HAART) in patients with a history of sustained viral suppression | Q36781531 | ||
DNase I-hypersensitive sites are associated with both long terminal repeats and with the intragenic enhancer of integrated human immunodeficiency virus type 1 | Q36828450 | ||
Methylation as a modulator of expression of human immunodeficiency virus | Q36886830 | ||
What Integration Sites Tell Us about HIV Persistence | Q36987230 | ||
Long noncoding RNA NRON contributes to HIV-1 latency by specifically inducing tat protein degradation | Q37010047 | ||
LEDGIN-mediated Inhibition of Integrase-LEDGF/p75 Interaction Reduces Reactivation of Residual Latent HIV | Q37035177 | ||
MicroRNA profile changes in human immunodeficiency virus type 1 (HIV-1) seropositive individuals | Q37102282 | ||
Defective HIV-1 proviruses produce novel protein-coding RNA species in HIV-infected patients on combination antiretroviral therapy | Q37161557 | ||
Methylome-wide Analysis of Chronic HIV Infection Reveals Five-Year Increase in Biological Age and Epigenetic Targeting of HLA. | Q37198065 | ||
Defective proviruses rapidly accumulate during acute HIV-1 infection | Q37239064 | ||
Broad activation of latent HIV-1 in vivo | Q37260309 | ||
Transcriptional competence of the integrated HIV-1 provirus at the nuclear periphery | Q37304680 | ||
Molecular control of HIV-1 postintegration latency: implications for the development of new therapeutic strategies | Q37644561 | ||
International AIDS Society global scientific strategy: towards an HIV cure 2016. | Q37661770 | ||
Multiple Histone Lysine Methyltransferases Are Required for the Establishment and Maintenance of HIV-1 Latency | Q37695602 | ||
Epigenetic reprogramming of host genes in viral and microbial pathogenesis | Q37781197 | ||
The control of HIV transcription: keeping RNA polymerase II on track | Q37958430 | ||
LEDGINs, non-catalytic site inhibitors of HIV-1 integrase: a patent review (2006 - 2014). | Q38199193 | ||
Regulation of HIV-1 latency by chromatin structure and nuclear architecture. | Q38235100 | ||
Multiple Origins of Virus Persistence during Natural Control of HIV Infection | Q38379102 | ||
Reversal of Latency as Part of a Cure for HIV-1. | Q38382980 | ||
HIV persistence: clonal expansion of cells in the latent reservoir | Q38669409 | ||
5' long terminal repeat (LTR)-selective methylation of latently infected HIV-1 provirus that is demethylated by reactivation signals | Q38758956 | ||
Shocking HIV out of hiding: where are we with clinical trials of latency reversing agents? | Q38773858 | ||
The molecular hallmarks of epigenetic control | Q38877902 | ||
P275 | copyright license | Creative Commons Attribution | Q6905323 |
P577 | publication date | 2020-02-11 | |
2020-04-01 | |||
P1433 | published in | Seminars in Immunopathology | Q15724576 |
P1476 | title | Epigenetic crosstalk in chronic infection with HIV-1 |
Q92137391 | Infection-induced epigenetic changes and their impact on the pathogenesis of diseases | cites work | P2860 |
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