| review article | Q7318358 |
| scholarly article | Q13442814 |
| P50 | author | Menno de Winther | Q59688356 |
| K Prange | Q85197904 | ||
| Tatyana Kuznetsova | Q90442060 | ||
| P2093 | author name string | Christopher K Glass | |
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| Clonal Hematopoiesis and Risk of Atherosclerotic Cardiovascular Disease. | Q34558126 | ||
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| A dual cis-regulatory code links IRF8 to constitutive and inducible gene expression in macrophages | Q35104157 | ||
| Cholesterol loading reprograms the microRNA-143/145-myocardin axis to convert aortic smooth muscle cells to a dysfunctional macrophage-like phenotype | Q35130704 | ||
| Tissue-resident macrophage enhancer landscapes are shaped by the local microenvironment. | Q35623089 | ||
| Histone deacetylase 3 is an epigenomic brake in macrophage alternative activation. | Q35624730 | ||
| Transcription factors STAT6 and KLF4 implement macrophage polarization via the dual catalytic powers of MCPIP | Q35691107 | ||
| A Genome-wide CRISPR Screen in Primary Immune Cells to Dissect Regulatory Networks | Q35696798 | ||
| K Domain CR9 of Low Density Lipoprotein (LDL) Receptor-related Protein 1 (LRP1) Is Critical for Aggregated LDL-induced Foam Cell Formation from Human Vascular Smooth Muscle Cells | Q35721623 | ||
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| The transcriptional regulator network of human inflammatory macrophages is defined by open chromatin. | Q38299850 | ||
| Function and information content of DNA methylation | Q38319920 | ||
| Epigenetic pathways in macrophages emerge as novel targets in atherosclerosis | Q38500261 | ||
| Macrophage activation in human diseases | Q38572817 | ||
| KLF4 Regulation of Ch25h and LXR Mitigates Atherosclerosis Susceptibility | Q38631767 | ||
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| Accumulation of serum lipids by vascular smooth muscle cells involves a macropinocytosis-like uptake pathway and is associated with the downregulation of the ATP-binding cassette transporter A1. | Q39103066 | ||
| Targeting macrophage Histone deacetylase 3 stabilizes atherosclerotic lesions. | Q39162364 | ||
| Methyltransferase Dnmt3a upregulates HDAC9 to deacetylate the kinase TBK1 for activation of antiviral innate immunity | Q39725913 | ||
| β-Glucan Reverses the Epigenetic State of LPS-Induced Immunological Tolerance | Q40445242 | ||
| New insights into the multidimensional concept of macrophage ontogeny, activation and function. | Q40866271 | ||
| The transcription factor ATF7 mediates lipopolysaccharide-induced epigenetic changes in macrophages involved in innate immunological memory. | Q41024271 | ||
| IFN-γ Induces Histone 3 Lysine 27 Trimethylation in a Small Subset of Promoters to Stably Silence Gene Expression in Human Macrophages. | Q41572756 | ||
| Global, regional, and national age-sex specific mortality for 264 causes of death, 1980-2016: a systematic analysis for the Global Burden of Disease Study 2016. | Q41689458 | ||
| Epigenetic programming of monocyte-to-macrophage differentiation and trained innate immunity. | Q41725528 | ||
| IRF5:RelA interaction targets inflammatory genes in macrophages | Q41850238 | ||
| DNA methylation dynamics during in vivo differentiation of blood and skin stem cells | Q41879063 | ||
| Oleic acid induces smooth muscle foam cell formation and enhances atherosclerotic lesion development via CD36. | Q41975459 | ||
| Molecular determinants of crosstalk between nuclear receptors and toll-like receptors. | Q42135532 | ||
| IL-4 orchestrates STAT6-mediated DNA demethylation leading to dendritic cell differentiation | Q42199311 | ||
| Interferon-γ Represses M2 Gene Expression in Human Macrophages by Disassembling Enhancers Bound by the Transcription Factor MAF. | Q42513727 | ||
| Identification and characterization of enhancers controlling the inflammatory gene expression program in macrophages. | Q43141233 | ||
| MafB promotes atherosclerosis by inhibiting foam-cell apoptosis | Q46082307 | ||
| Latent enhancers activated by stimulation in differentiated cells | Q46305342 | ||
| IL-13 induces disease-promoting type 2 cytokines, alternatively activated macrophages and allergic inflammation during pulmonary infection of mice with Cryptococcus neoformans | Q46970232 | ||
| BCG Vaccination Protects against Experimental Viral Infection in Humans through the Induction of Cytokines Associated with Trained Immunity | Q47555324 | ||
| Static and Dynamic DNA Loops form AP-1-Bound Activation Hubs during Macrophage Development | Q47692532 | ||
| Type I interferons and the cytokine TNF cooperatively reprogram the macrophage epigenome to promote inflammatory activation | Q47927601 | ||
| Modulation of Myelopoiesis Progenitors Is an Integral Component of Trained Immunity. | Q49226761 | ||
| Corticosterone Preexposure Increases NF-κB Translocation and Sensitizes IL-1β Responses in BV2 Microglia-Like Cells. | Q49299368 | ||
| The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages. | Q49393104 | ||
| AP-1 Transcription Factors and the BAF Complex Mediate Signal-Dependent Enhancer Selection. | Q49614247 | ||
| Western Diet Triggers NLRP3-Dependent Innate Immune Reprogramming. | Q50065321 | ||
| Contribution of intimal smooth muscle cells to cholesterol accumulation and macrophage-like cells in human atherosclerosis. | Q50479986 | ||
| The Potential Role of Trained Immunity in Autoimmune and Autoinflammatory Disorders. | Q50531937 | ||
| Transdifferentiation of vascular smooth muscle cells to macrophage-like cells during atherogenesis. | Q51065835 | ||
| The IL-4/STAT6/PPARγ signaling axis is driving the expansion of the RXR heterodimer cistrome, providing complex ligand responsiveness in macrophages. | Q52366978 | ||
| Atlas of the Immune Cell Repertoire in Mouse Atherosclerosis Defined by Single-Cell RNA-Sequencing and Mass Cytometry. | Q52653300 | ||
| P433 | issue | 4 | |
| P304 | page(s) | 216-228 | |
| P577 | publication date | 2019-10-02 | |
| P1433 | published in | Nature Reviews Cardiology | Q2108444 |
| P1476 | title | Transcriptional and epigenetic regulation of macrophages in atherosclerosis | |
| P478 | volume | 17 |
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| Q92151548 | The low-density lipoprotein cholesterol lowering is an ineffective surrogate marker of statin responsiveness to predict cardiovascular outcomes: The 10-year experience of matched population (a STROBE-compliant article) |
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