Loss of Nucleobindin-2 Causes Insulin Resistance in Obesity without Impacting Satiety or Adiposity

scientific article published on 01 July 2018

Loss of Nucleobindin-2 Causes Insulin Resistance in Obesity without Impacting Satiety or Adiposity is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.CELREP.2018.06.112
P932PMC publication ID6223120
P698PubMed publication ID30067966

P50authorJoachim SchultzeQ1690457
Gerald I. ShulmanQ52509909
P2093author name stringLuis Varela
Tamas L Horvath
Vishwa Deep Dixit
Yun-Hee Youm
Jil Sander
Kim Nguyen
Anthony Ravussin
Seungjin Ryu
Sviatoslav Sidorov
P2860cites workGhrelin inhibits leptin- and activation-induced proinflammatory cytokine expression by human monocytes and T cellsQ24298483
The mammalian calcium-binding protein, nucleobindin (CALNUC), is a Golgi resident proteinQ24682684
Ghrelin-mediated sympathoinhibition and suppression of inflammation in sepsisQ27021339
Neuro-endocrine networks controlling immune system in health and diseaseQ27026101
Inflammation and metabolic disordersQ27860923
Nesfatin-1: functions and physiology of a novel regulatory peptideQ28072589
Molecular cloning of nucleobindin, a novel DNA-binding protein that contains both a signal peptide and a leucine zipper structureQ28117984
Identification of nesfatin-1 as a satiety molecule in the hypothalamusQ28268139
The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistanceQ29616026
Leptin and the immune system: how nutritional status influences the immune response.Q33855613
HISAT: a fast spliced aligner with low memory requirementsQ34043398
Transcriptome-based network analysis reveals a spectrum model of human macrophage activation.Q34659689
Inflammatory links between obesity and metabolic diseaseQ35015802
G Protein binding sites on Calnuc (nucleobindin 1) and NUCB2 (nucleobindin 2) define a new class of G(alpha)i-regulatory motifsQ35150118
Tissue-resident macrophage enhancer landscapes are shaped by the local microenvironment.Q35623089
Banking on ATM as a new target in metabolic syndromeQ36644721
Identification and characterization of nesfatin-1 immunoreactivity in endocrine cell types of the rat gastric oxyntic mucosaQ37072774
Adipose-immune interactions during obesity and caloric restriction: reciprocal mechanisms regulating immunity and health spanQ37089841
A stratified myeloid system, the challenge of understanding macrophage diversity.Q37141486
Nesfatin-1: an overview and future clinical applicationQ37491970
Quantification of adipose tissue leukocytosis in obesityQ37678116
Paraventricular NUCB2/Nesfatin-1 Supports Oxytocin and Vasopressin Neurons to Control Feeding Behavior and Fluid Balance in Male MiceQ40786065
Immune-neuro-endocrine interactions: facts and hypothesesQ40993419
Standard operating procedures for describing and performing metabolic tests of glucose homeostasis in miceQ42427575
Inflammasome-driven catecholamine catabolism in macrophages blunts lipolysis during ageing.Q49720453
An Integrated View of Immunometabolism.Q50065309
P4510describes a project that usesImageJQ1659584
P433issue5
P921main subjectinsulin resistanceQ1053470
satietyQ1562684
P304page(s)1085-1092.e6
P577publication date2018-07-01
P1433published inCell ReportsQ5058165
P1476titleLoss of Nucleobindin-2 Causes Insulin Resistance in Obesity without Impacting Satiety or Adiposity
P478volume24

Reverse relations

cites work (P2860)
Q90409945Altered adipose tissue and adipocyte function in the pathogenesis of metabolic syndrome
Q57300499Current Understanding of the Role of Nesfatin-1
Q94475801NUCB2 polymorphisms are associated with an increased risk for type 2 diabetes in the Chinese population
Q64091863Nesfatin-1 suppresses peripheral arterial remodeling without elevating blood pressure in mice

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