| review article | Q7318358 |
| scholarly article | Q13442814 |
| P2093 | author name string | Jean-Sébastien Hoffmann | |
| Camille Franchet | |||
| P2860 | cites work | Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair | Q24317107 |
| Human GEN1 and the SLX4-associated nucleases MUS81 and SLX1 are essential for the resolution of replication-induced Holliday junctions | Q24337537 | ||
| Chromosome fragile sites | Q28235126 | ||
| Rad52 prevents excessive replication fork reversal and protects from nascent strand degradation | Q64104226 | ||
| Overexpression of Claspin and Timeless protects cancer cells from replication stress in a checkpoint-independent manner | Q64113941 | ||
| Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Tumor Cells. | Q64978193 | ||
| TRAIP drives replisome disassembly and mitotic DNA repair synthesis at sites of incomplete DNA replication | Q83228113 | ||
| DNA replication stress triggers rapid DNA replication fork breakage by Artemis and XPF | Q90611794 | ||
| RAD52 as a Potential Target for Synthetic Lethality-Based Anticancer Therapies | Q90725291 | ||
| 53BP1 nuclear bodies enforce replication timing at under-replicated DNA to limit heritable DNA damage | Q91907550 | ||
| Replication stress activates DNA repair synthesis in mitosis. | Q38814576 | ||
| 53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress | Q39593027 | ||
| MUS81 nuclease activity is essential for replication stress tolerance and chromosome segregation in BRCA2-deficient cells | Q41077646 | ||
| APC/C Dysfunction Limits Excessive Cancer Chromosomal Instability | Q42322723 | ||
| Cooperative control of holliday junction resolution and DNA repair by the SLX1 and MUS81-EME1 nucleases. | Q42933602 | ||
| The endonuclease EEPD1 mediates synthetic lethality in RAD52-depleted BRCA1 mutant breast cancer cells. | Q45898863 | ||
| A 'DNA replication' signature of progression and negative outcome in colorectal cancer | Q46243690 | ||
| The structure-specific endonuclease Mus81 contributes to replication restart by generating double-strand DNA breaks | Q46952804 | ||
| RAD52 Facilitates Mitotic DNA Synthesis Following Replication Stress. | Q48897701 | ||
| CHK1 as a therapeutic target to bypass chemoresistance in AML. | Q51510125 | ||
| DNA polymerase ν gene expression influences fludarabine resistance in chronic lymphocytic leukemia independently from p53 status. | Q52641879 | ||
| Human cancer cells utilize mitotic DNA synthesis to resist replication stress at telomeres regardless of their telomere maintenance mechanism. | Q52718919 | ||
| Role of specialized DNA polymerases in the limitation of replicative stress and DNA damage transmission. | Q52757813 | ||
| The Protective Role of Dormant Origins in Response to Replicative Stress | Q59810213 | ||
| RAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombination | Q28274789 | ||
| Rad52 inactivation is synthetically lethal with BRCA2 deficiency | Q28300346 | ||
| Identification of a Small Molecule Inhibitor of RAD52 by Structure-Based Selection | Q28552598 | ||
| Mammalian polymerase θ promotes alternative NHEJ and suppresses recombination | Q28593605 | ||
| Clonal evolution in cancer | Q29547696 | ||
| Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions | Q29614216 | ||
| DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis | Q29614217 | ||
| Role of RAD52 epistasis group genes in homologous recombination and double-strand break repair | Q29618204 | ||
| Human Rad52 binds and wraps single-stranded DNA and mediates annealing via two hRad52-ssDNA complexes | Q33524756 | ||
| Proteomic Profiling Reveals a Specific Role for Translesion DNA Polymerase η in the Alternative Lengthening of Telomeres | Q33605965 | ||
| Targeted inactivation of mouse RAD52 reduces homologous recombination but not resistance to ionizing radiation | Q33781473 | ||
| Genetic instability and darwinian selection in tumours | Q33804007 | ||
| DNA polymerase theta up-regulation is associated with poor survival in breast cancer, perturbs DNA replication, and promotes genetic instability | Q34067829 | ||
| Alternative lengthening of telomeres: models, mechanisms and implications | Q34107386 | ||
| Eukaryotic DNA replication origins: many choices for appropriate answers | Q34139588 | ||
| New insights into the formation and resolution of ultra-fine anaphase bridges | Q34202945 | ||
| Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair. | Q34218130 | ||
| Molecular pathways: understanding the role of Rad52 in homologous recombination for therapeutic advancement | Q34306334 | ||
| Regulation of early events in chromosome replication | Q34349937 | ||
| Cell-type-specific replication initiation programs set fragility of the FRA3B fragile site | Q34626544 | ||
| DNA polymerases and human disease | Q34795963 | ||
| Replication stress induces 53BP1-containing OPT domains in G1 cells | Q34854577 | ||
| Small-Molecule Disruption of RAD52 Rings as a Mechanism for Precision Medicine in BRCA-Deficient Cancers. | Q35835383 | ||
| Targeting BRCA1- and BRCA2-deficient cells with RAD52 small molecule inhibitors | Q35923115 | ||
| Deregulation of cyclin E in human cells interferes with prereplication complex assembly | Q36322482 | ||
| DNA replication stress response involving PLK1, CDC6, POLQ, RAD51 and CLASPIN upregulation prognoses the outcome of early/mid-stage non-small cell lung cancer patients. | Q36415115 | ||
| Replication fork stalling at natural impediments | Q36755308 | ||
| DNA synthesis by Pol η promotes fragile site stability by preventing under-replicated DNA in mitosis | Q36804077 | ||
| DNA polymerases and cancer. | Q37084895 | ||
| Nucleotide deficiency promotes genomic instability in early stages of cancer development | Q37087191 | ||
| Excess Polθ functions in response to replicative stress in homologous recombination-proficient cancer cells | Q37381321 | ||
| Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks | Q37528453 | ||
| Maintaining genome stability at the replication fork | Q37696954 | ||
| The essential kinase ATR: ensuring faithful duplication of a challenging genome. | Q38622938 | ||
| Transcription as a Threat to Genome Integrity | Q38792613 | ||
| P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 1 | |
| P577 | publication date | 2019-12-19 | |
| P1433 | published in | Cancers | Q27722963 |
| P1476 | title | When RAD52 Allows Mitosis to Accept Unscheduled DNA Synthesis | |
| P478 | volume | 12 |
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