| scholarly article | Q13442814 |
| P2093 | author name string | Jin Li | |
| William A Catterall | |||
| Joshua S Kaplan | |||
| Rachael E Stein | |||
| P2860 | cites work | De novo mutations in the sodium-channel gene SCN1A cause severe myoclonic epilepsy of infancy | Q24533495 |
| Dissecting the phenotypes of Dravet syndrome by gene deletion | Q27303721 | ||
| Genetic background modulates impaired excitability of inhibitory neurons in a mouse model of Dravet syndrome | Q27308208 | ||
| Reduced sodium current in Purkinje neurons from Nav1.1 mutant mice: implications for ataxia in severe myoclonic epilepsy in infancy | Q28513583 | ||
| Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy | Q28585126 | ||
| Nav1.1 localizes to axons of parvalbumin-positive inhibitory interneurons: a circuit basis for epileptic seizures in mice carrying an Scn1a gene mutation | Q28587835 | ||
| Place navigation impaired in rats with hippocampal lesions | Q29547869 | ||
| The dentate gyrus as a control point for seizures in the hippocampus and beyond | Q30311316 | ||
| Temperature- and age-dependent seizures in a mouse model of severe myoclonic epilepsy in infancy | Q30486496 | ||
| Sudden unexpected death in a mouse model of Dravet syndrome | Q30538288 | ||
| Correlations in timing of sodium channel expression, epilepsy, and sudden death in Dravet syndrome | Q33702545 | ||
| Behavioral phenotyping of transgenic and knockout mice: experimental design and evaluation of general health, sensory functions, motor abilities, and specific behavioral tests | Q33711537 | ||
| Impaired excitability of somatostatin- and parvalbumin-expressing cortical interneurons in a mouse model of Dravet syndrome | Q34002328 | ||
| Impaired action potential initiation in GABAergic interneurons causes hyperexcitable networks in an epileptic mouse model carrying a human Na(V)1.1 mutation | Q34451662 | ||
| Hippocampal representation in place learning. | Q34635657 | ||
| Cerebellar Directed Optogenetic Intervention Inhibits Spontaneous Hippocampal Seizures in a Mouse Model of Temporal Lobe Epilepsy | Q34948808 | ||
| Sleep impairment and reduced interneuron excitability in a mouse model of Dravet Syndrome | Q35472627 | ||
| Na(V)1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms | Q35750926 | ||
| Specific deletion of NaV1.1 sodium channels in inhibitory interneurons causes seizures and premature death in a mouse model of Dravet syndrome | Q36221618 | ||
| Autistic-like behaviour in Scn1a+/- mice and rescue by enhanced GABA-mediated neurotransmission. | Q36252621 | ||
| Nav1.1 haploinsufficiency in excitatory neurons ameliorates seizure-associated sudden death in a mouse model of Dravet syndrome. | Q37287002 | ||
| Cre-dependent selection yields AAV variants for widespread gene transfer to the adult brain | Q37382030 | ||
| Strain- and age-dependent hippocampal neuron sodium currents correlate with epilepsy severity in Dravet syndrome mice | Q37670832 | ||
| NaV1.1 channels and epilepsy | Q37701079 | ||
| Hippocampus and neocortex: recognition and spatial memory | Q37847373 | ||
| The core Dravet syndrome phenotype | Q37861252 | ||
| The genetics of Dravet syndrome. | Q37861258 | ||
| Dravet syndrome history | Q37866828 | ||
| Memory deficits associated with senescence: A neurophysiological and behavioral study in the rat | Q40232008 | ||
| De novo SCN1A mutations are a major cause of severe myoclonic epilepsy of infancy | Q40558873 | ||
| In vivo evaluation of the dentate gate theory in epilepsy. | Q41266313 | ||
| Manipulating gene expression in projection-specific neuronal populations using combinatorial viral approaches | Q41810942 | ||
| Mouse with Nav1.1 haploinsufficiency, a model for Dravet syndrome, exhibits lowered sociability and learning impairment | Q44066884 | ||
| Double dissociation between the effects of peri-postrhinal cortex and hippocampal lesions on tests of object recognition and spatial memory: heterogeneity of function within the temporal lobe. | Q44960342 | ||
| Impairments in social novelty recognition and spatial memory in mice with conditional deletion of Scn1a in parvalbumin-expressing cells | Q47558970 | ||
| Cannabidiol attenuates seizures and social deficits in a mouse model of Dravet syndrome | Q47668118 | ||
| SCN1A duplications and deletions detected in Dravet syndrome: implications for molecular diagnosis. | Q47923406 | ||
| Modification of seizure activity by electrical stimulation. 3. Mechanisms. | Q48793593 | ||
| Fetal gene therapy for neurodegenerative disease of infants | Q59356806 | ||
| A Transient Developmental Window of Fast-Spiking Interneuron Dysfunction in a Mouse Model of Dravet Syndrome. | Q64967262 | ||
| Dravet Syndrome: A Sodium Channel Interneuronopathy | Q91032526 | ||
| P433 | issue | 33 | |
| P921 | main subject | Dravet syndrome | Q1255956 |
| cognitive deficit | Q5141203 | ||
| P304 | page(s) | 16571-16576 | |
| P577 | publication date | 2019-07-25 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | Hippocampal deletion of NaV1.1 channels in mice causes thermal seizures and cognitive deficit characteristic of Dravet Syndrome | |
| P478 | volume | 116 |
| Q90848333 | Deficiency of NONO is associated with impaired cardiac function and fibrosis in mice |
| Q90453059 | Focal and generalized seizure activity after local hippocampal or cortical ablation of NaV 1.1 channels in mice |
| Q94671569 | Turning strains into strengths for understanding psychiatric disorders |
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